Severe Hepatic Steatosis Is Associated With Low-Level Viremia and Advanced Fibrosis in Patients With Chronic Hepatitis B in North America.

Controlled Attenuated Parameter (CAP) Fatty Liver Disease Hepatic Steatosis Hepatitis B Transient Elastography (TE)

Journal

Gastro hep advances
ISSN: 2772-5723
Titre abrégé: Gastro Hep Adv
Pays: Netherlands
ID NLM: 9918350485906676

Informations de publication

Date de publication:
2022
Historique:
received: 19 07 2021
accepted: 14 09 2021
medline: 3 2 2022
pubmed: 3 2 2022
entrez: 12 8 2024
Statut: epublish

Résumé

The obesity epidemic has increased the risk of nonalcoholic fatty liver disease (NAFLD) in both the general and chronic hepatitis B (CHB) populations. Our study aims to determine the prevalence of NAFLD in patients with CHB based on controlled attenuation parameter (CAP) and the epidemiological, clinical, and virological factors associated with severe hepatic steatosis. The Canadian Hepatitis B Network cohort was utilized to provide a cross-sectional description of demographics, comorbidities, antiviral treatment, and hepatits B virus (HBV) tests. Liver fibrosis and steatosis were measured by transient elastography and CAP, respectively. Any grade and severe steatosis were defined as CAP >248 and >280 dB/m, respectively. Advanced liver fibrosis was defined as transient elastography measurement >10.7 kPa. In 1178 patients with CHB (median age: 47.4%, 57.7% males, 75.7% Asian, 13% African, 6.5% White, 86% HBV e antigen negative, median HBV DNA of 2.44 log In this large multiethnic CHB population, hepatic steatosis is common. Severe steatosis is independently associated with higher fibrosis, but negatively with HBV DNA, regardless of antiviral therapy history.

Sections du résumé

Background and Aims UNASSIGNED
The obesity epidemic has increased the risk of nonalcoholic fatty liver disease (NAFLD) in both the general and chronic hepatitis B (CHB) populations. Our study aims to determine the prevalence of NAFLD in patients with CHB based on controlled attenuation parameter (CAP) and the epidemiological, clinical, and virological factors associated with severe hepatic steatosis.
Methods UNASSIGNED
The Canadian Hepatitis B Network cohort was utilized to provide a cross-sectional description of demographics, comorbidities, antiviral treatment, and hepatits B virus (HBV) tests. Liver fibrosis and steatosis were measured by transient elastography and CAP, respectively. Any grade and severe steatosis were defined as CAP >248 and >280 dB/m, respectively. Advanced liver fibrosis was defined as transient elastography measurement >10.7 kPa.
Results UNASSIGNED
In 1178 patients with CHB (median age: 47.4%, 57.7% males, 75.7% Asian, 13% African, 6.5% White, 86% HBV e antigen negative, median HBV DNA of 2.44 log
Conclusion UNASSIGNED
In this large multiethnic CHB population, hepatic steatosis is common. Severe steatosis is independently associated with higher fibrosis, but negatively with HBV DNA, regardless of antiviral therapy history.

Identifiants

pubmed: 39129930
doi: 10.1016/j.gastha.2021.09.005
pii: S2772-5723(21)00010-8
pmc: PMC11307651
doi:

Types de publication

Journal Article

Langues

eng

Pagination

106-116

Informations de copyright

© 2022 The Authors.

Auteurs

Hin Hin Ko (HH)

Department of Medicine, University of British Columbia, Vancouver, British Columbia, Canada.

Nishi H Patel (NH)

Cumming School of Medicine, University of Calgary, Calgary, Alberta, Canada.

Sarah Haylock-Jacobs (S)

Cumming School of Medicine, University of Calgary, Calgary, Alberta, Canada.

Karen Doucette (K)

Department of Medicine, University of Alberta, Edmonton, Alberta, Canada.

Mang M Ma (MM)

Department of Medicine, University of Alberta, Edmonton, Alberta, Canada.

Curtis Cooper (C)

Department of Medicine, University of Ottawa, Ottawa, Ontario, Canada.

Erin Kelly (E)

Department of Medicine, University of Ottawa, Ottawa, Ontario, Canada.

Magdy Elkhashab (M)

Depatment of Medicine, Toronto Liver Centre, Toronto, Ontario, Canada.

Edward Tam (E)

Depatment of Medicine, Pacific Gastroenterology Associates, Vancouver, British Columbia, Canada.

Robert Bailey (R)

Department of Medicine, University of Alberta, Edmonton, Alberta, Canada.

Alexander Wong (A)

Department of Medicine, University of Saskatchewan, Regina, Saskatchewan, Canada.

Gerald Minuk (G)

Department of Medicine, University of Manitoba, Winnipeg, Manitoba, Canada.

Philip Wong (P)

Department of Medicine, McGill University Health Centre, Montreal, Quebec, Canada.

Scott K Fung (SK)

Department of Medicine, University Health Network, Toronto, Ontario, Canada.

Giada Sebastiani (G)

Department of Medicine, McGill University Health Centre, Montreal, Quebec, Canada.

Alnoor Ramji (A)

Department of Medicine, University of British Columbia, Vancouver, British Columbia, Canada.

Carla S Coffin (CS)

Cumming School of Medicine, University of Calgary, Calgary, Alberta, Canada.

Classifications MeSH