Comprehensive behavioural assessment of TERT in bladder cancer.

Biomarker Bladder cancer Gene expression Mutation TERT promoter THOR Telomere length

Journal

Urologic oncology
ISSN: 1873-2496
Titre abrégé: Urol Oncol
Pays: United States
ID NLM: 9805460

Informations de publication

Date de publication:
14 Aug 2024
Historique:
received: 11 03 2024
revised: 20 06 2024
accepted: 27 06 2024
medline: 16 8 2024
pubmed: 16 8 2024
entrez: 15 8 2024
Statut: aheadofprint

Résumé

Telomerase activity plays a crucial role in cancer development and progression. Thus, telomerase activation through the interplay of mutations and epigenetic alterations in the telomerase reverse transcriptase (TERT) promoter may provide further insight into bladder cancer induction and progression. In this study 100 bladder tumour tissues were selected, and four molecular signatures were analysed: THOR methylation status, TERT promotor mutation, telomere length, and TERT expression. In our study, 88% of bladder cancer patients had an hypermethylation of the THOR region and 60% had mutations in the TERT promoter region. TERT promoter methylation was observed in all stages and grades of bladder cancer. While, TERT promoter mutations were detected in advanced stages and grades. In our cohort, high levels of TERT expression and long telomeres have been found in noninvasive cases of bladder cancer, with a significant association between TERT expression and Telomere length. Interestingly, patients with low TERT expression and cases with long telomeres had significantly longer Disease-free survival and overall survival. The methylation and mutations occurring in the TERT promoter are implicated in bladder carcinogenesis, offering added prognostic and supplying novel insight into telomere biology in cancer.

Sections du résumé

BACKGROUND BACKGROUND
Telomerase activity plays a crucial role in cancer development and progression. Thus, telomerase activation through the interplay of mutations and epigenetic alterations in the telomerase reverse transcriptase (TERT) promoter may provide further insight into bladder cancer induction and progression.
METHODS METHODS
In this study 100 bladder tumour tissues were selected, and four molecular signatures were analysed: THOR methylation status, TERT promotor mutation, telomere length, and TERT expression.
RESULTS RESULTS
In our study, 88% of bladder cancer patients had an hypermethylation of the THOR region and 60% had mutations in the TERT promoter region. TERT promoter methylation was observed in all stages and grades of bladder cancer. While, TERT promoter mutations were detected in advanced stages and grades. In our cohort, high levels of TERT expression and long telomeres have been found in noninvasive cases of bladder cancer, with a significant association between TERT expression and Telomere length. Interestingly, patients with low TERT expression and cases with long telomeres had significantly longer Disease-free survival and overall survival.
CONCLUSION CONCLUSIONS
The methylation and mutations occurring in the TERT promoter are implicated in bladder carcinogenesis, offering added prognostic and supplying novel insight into telomere biology in cancer.

Identifiants

pubmed: 39147693
pii: S1078-1439(24)00536-2
doi: 10.1016/j.urolonc.2024.06.024
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright © 2024 Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.

Auteurs

Meryem El Azzouzi (M)

Biology and Medical Research Unit, CNESTEN, Rabat, Morocco; Rabat Medical and Pharmacy School, Mohammed V University in Rabat, Rabat, Morocco.

Hajar El Ahanidi (H)

Biology and Medical Research Unit, CNESTEN, Rabat, Morocco; Department of Pathology and Immunology, University of Geneva, Geneva, Switzerland; Ludwig Institute for Cancer Research, Lausanne Branch, Lausanne, Switzerland.

Ilias Hassan (I)

Rabat Medical and Pharmacy School, Mohammed V University in Rabat, Rabat, Morocco; Department of Urology, Military Hospital Mohammed V, Rabat, Morocco.

Mohammed Tetou (M)

Rabat Medical and Pharmacy School, Mohammed V University in Rabat, Rabat, Morocco; Department of Urology, Military Hospital Mohammed V, Rabat, Morocco.

Ahmed Ameur (A)

Rabat Medical and Pharmacy School, Mohammed V University in Rabat, Rabat, Morocco; Department of Urology, Military Hospital Mohammed V, Rabat, Morocco.

Mounia Bensaid (M)

Laboratory of Pathological Anatomy, Military Hospital Mohamed V, Rabat, Morocco; Royal School of Military Health Service, Rabat, Morocco.

Abderrahmane Al Bouzidi (A)

Rabat Medical and Pharmacy School, Mohammed V University in Rabat, Rabat, Morocco.

Mohamed Oukabli (M)

Rabat Medical and Pharmacy School, Mohammed V University in Rabat, Rabat, Morocco; Laboratory of Pathological Anatomy, Military Hospital Mohamed V, Rabat, Morocco.

Chaimae Hafidi Alaoui (CH)

Biology and Medical Research Unit, CNESTEN, Rabat, Morocco; Faculty of Sciences, Mohammed V University in Rabat, Rabat, Morocco.

Boutaina Addoum (B)

Biology and Medical Research Unit, CNESTEN, Rabat, Morocco.

Imane Chaoui (I)

Biology and Medical Research Unit, CNESTEN, Rabat, Morocco.

Laila Benbacer (L)

Biology and Medical Research Unit, CNESTEN, Rabat, Morocco.

Mohammed El Mzibri (ME)

Biology and Medical Research Unit, CNESTEN, Rabat, Morocco.

Mohammed Attaleb (M)

Biology and Medical Research Unit, CNESTEN, Rabat, Morocco. Electronic address: attaleb_mohammed@yahoo.fr.

Classifications MeSH