Longitudinal survey of insecticide resistance in a village of central region of Burkina Faso reveals co-occurrence of 1014F, 1014S and 402L mutations in Anopheles coluzzii and Anopheles arabiensis.


Journal

Malaria journal
ISSN: 1475-2875
Titre abrégé: Malar J
Pays: England
ID NLM: 101139802

Informations de publication

Date de publication:
20 Aug 2024
Historique:
received: 28 03 2024
accepted: 07 08 2024
medline: 21 8 2024
pubmed: 21 8 2024
entrez: 20 8 2024
Statut: epublish

Résumé

Pyrethroid resistance is one of the major threats for effectiveness of insecticide-treated bed nets (ITNs) in malaria vector control. Genotyping of mutations in the voltage gated sodium channel (VGSC) gene is widely used to easily assess the evolution and spread of pyrethroid target-site resistance among malaria vectors. L1014F and L1014S substitutions are the most common and best characterized VGSC mutations in major African malaria vector species of the Anopheles gambiae complex. Recently, an additional substitution involved in pyrethroid resistance, i.e. V402L, has been detected in Anopheles coluzzii from West Africa lacking any other resistance alleles at locus 1014. The evolution of target-site resistance mutations L1014F/S and V402L was monitored in An. coluzzii and Anopheles arabiensis specimens from a Burkina Faso village over a 10-year range after the massive ITN scale-up started in 2010. Anopheles coluzzii (N = 300) and An. arabiensis (N = 362) specimens collected both indoors and outdoors by different methods (pyrethrum spray catch, sticky resting box and human landing collections) in 2011, 2015 and 2020 at Goden village were genotyped by TaqMan assays and sequencing for the three target site resistance mutations; allele frequencies were statistically investigated over the years. A divergent trend in resistant allele frequencies was observed in the two species: 1014F decreased in An. coluzzii (from 0.76 to 0.52) but increased in An. arabiensis (from 0.18 to 0.70); 1014S occurred only in An. arabiensis and slightly decreased over time (from 0.33 to 0.23); 402L increased in An. coluzzii (from 0.15 to 0.48) and was found for the first time in one An. arabiensis specimen. In 2020 the co-occurrence of different resistance alleles reached 43% in An. coluzzii (alleles 410L and 1014F) and 32% in An. arabiensis (alleles 1014F and 1014S). Overall, an increasing level of target-site resistance was observed among the populations with only 1% of the two malaria vector species being wild type at both loci, 1014 and 402, in 2020. This, together with the co-occurrence of different mutations in the same specimens, calls for future investigations on the possible synergism between resistance alleles and their phenotype to implement local tailored intervention strategies.

Sections du résumé

BACKGROUND BACKGROUND
Pyrethroid resistance is one of the major threats for effectiveness of insecticide-treated bed nets (ITNs) in malaria vector control. Genotyping of mutations in the voltage gated sodium channel (VGSC) gene is widely used to easily assess the evolution and spread of pyrethroid target-site resistance among malaria vectors. L1014F and L1014S substitutions are the most common and best characterized VGSC mutations in major African malaria vector species of the Anopheles gambiae complex. Recently, an additional substitution involved in pyrethroid resistance, i.e. V402L, has been detected in Anopheles coluzzii from West Africa lacking any other resistance alleles at locus 1014. The evolution of target-site resistance mutations L1014F/S and V402L was monitored in An. coluzzii and Anopheles arabiensis specimens from a Burkina Faso village over a 10-year range after the massive ITN scale-up started in 2010.
METHODS METHODS
Anopheles coluzzii (N = 300) and An. arabiensis (N = 362) specimens collected both indoors and outdoors by different methods (pyrethrum spray catch, sticky resting box and human landing collections) in 2011, 2015 and 2020 at Goden village were genotyped by TaqMan assays and sequencing for the three target site resistance mutations; allele frequencies were statistically investigated over the years.
RESULTS RESULTS
A divergent trend in resistant allele frequencies was observed in the two species: 1014F decreased in An. coluzzii (from 0.76 to 0.52) but increased in An. arabiensis (from 0.18 to 0.70); 1014S occurred only in An. arabiensis and slightly decreased over time (from 0.33 to 0.23); 402L increased in An. coluzzii (from 0.15 to 0.48) and was found for the first time in one An. arabiensis specimen. In 2020 the co-occurrence of different resistance alleles reached 43% in An. coluzzii (alleles 410L and 1014F) and 32% in An. arabiensis (alleles 1014F and 1014S).
CONCLUSIONS CONCLUSIONS
Overall, an increasing level of target-site resistance was observed among the populations with only 1% of the two malaria vector species being wild type at both loci, 1014 and 402, in 2020. This, together with the co-occurrence of different mutations in the same specimens, calls for future investigations on the possible synergism between resistance alleles and their phenotype to implement local tailored intervention strategies.

Identifiants

pubmed: 39164725
doi: 10.1186/s12936-024-05069-9
pii: 10.1186/s12936-024-05069-9
doi:

Substances chimiques

Insecticides 0
Voltage-Gated Sodium Channels 0
Pyrethrins 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

250

Subventions

Organisme : Sapienza Università di Roma
ID : AR11916B4CC76BF1, AR120172B8125A08 and AR22117A76DC0132
Organisme : Sapienza Università di Roma
ID : RM11916B7AFEA99E
Organisme : Sapienza Università di Roma
ID : RM11916B7AFEA99E

Informations de copyright

© 2024. The Author(s).

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Auteurs

Eleonora Perugini (E)

Department of Public Health and Infectious Diseases, Sapienza University of Rome, Rome, Italy.

Verena Pichler (V)

Department of Public Health and Infectious Diseases, Sapienza University of Rome, Rome, Italy.

Wamdaogo M Guelbeogo (WM)

Centre National de Recherche et Formation Sur le Paludisme, Ouagadougou, Burkina Faso.

Martina Micocci (M)

Department of Public Health and Infectious Diseases, Sapienza University of Rome, Rome, Italy.

Cristiana Poggi (C)

Department of Public Health and Infectious Diseases, Sapienza University of Rome, Rome, Italy.

Sara Manzi (S)

Istituto Zooprofilattico Sperimentale delle Venezie, Legnaro, Padua, Italy.

Hilary Ranson (H)

Liverpool School of Tropical Medicine, Department of Vector Biology, Liverpool, UK.

Alessandra Della Torre (A)

Department of Public Health and Infectious Diseases, Sapienza University of Rome, Rome, Italy.

Emiliano Mancini (E)

Department of Biology and Biotechnology Charles Darwin, Sapienza University of Rome, Rome, Italy.

Marco Pombi (M)

Department of Public Health and Infectious Diseases, Sapienza University of Rome, Rome, Italy. marco.pombi@uniroma1.it.

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