Mega-analysis of the brain-age gap in substance use disorder: An ENIGMA Addiction working group study.

ENIGMA addiction brain age machine learning neuroimaging predicted brain age difference substance use disorder

Journal

Addiction (Abingdon, England)
ISSN: 1360-0443
Titre abrégé: Addiction
Pays: England
ID NLM: 9304118

Informations de publication

Date de publication:
21 Aug 2024
Historique:
received: 29 01 2024
accepted: 19 06 2024
medline: 21 8 2024
pubmed: 21 8 2024
entrez: 21 8 2024
Statut: aheadofprint

Résumé

The brain age gap (BAG), calculated as the difference between a machine learning model-based predicted brain age and chronological age, has been increasingly investigated in psychiatric disorders. Tobacco and alcohol use are associated with increased BAG; however, no studies have compared global and regional BAG across substances other than alcohol and tobacco. This study aimed to compare global and regional estimates of brain age in individuals with substance use disorders and healthy controls. This was a cross-sectional study. This is an Enhancing Neuro Imaging through Meta-Analysis Consortium (ENIGMA) Addiction Working Group study including data from 38 global sites. This study included 2606 participants, of whom 1725 were cases with a substance use disorder and 881 healthy controls. This study used the Kaufmann brain age prediction algorithms to generate global and regional brain age estimates using T1 weighted magnetic resonance imaging (MRI) scans. We used linear mixed effects models to compare global and regional (FreeSurfer lobestrict output) BAG (i.e. predicted minus chronological age) between individuals with one of five primary substance use disorders as well as healthy controls. Alcohol use disorder (β = -5.49, t = -5.51, p < 0.001) was associated with higher global BAG, whereas amphetamine-type stimulant use disorder (β = 3.44, t = 2.42, p = 0.02) was associated with lower global BAG in the separate substance-specific models. People with alcohol use disorder appear to have a higher brain-age gap than people without alcohol use disorder, which is consistent with other evidence of the negative impact of alcohol on the brain.

Sections du résumé

BACKGROUND AND AIMS OBJECTIVE
The brain age gap (BAG), calculated as the difference between a machine learning model-based predicted brain age and chronological age, has been increasingly investigated in psychiatric disorders. Tobacco and alcohol use are associated with increased BAG; however, no studies have compared global and regional BAG across substances other than alcohol and tobacco. This study aimed to compare global and regional estimates of brain age in individuals with substance use disorders and healthy controls.
DESIGN METHODS
This was a cross-sectional study.
SETTING METHODS
This is an Enhancing Neuro Imaging through Meta-Analysis Consortium (ENIGMA) Addiction Working Group study including data from 38 global sites.
PARTICIPANTS METHODS
This study included 2606 participants, of whom 1725 were cases with a substance use disorder and 881 healthy controls.
MEASUREMENTS METHODS
This study used the Kaufmann brain age prediction algorithms to generate global and regional brain age estimates using T1 weighted magnetic resonance imaging (MRI) scans. We used linear mixed effects models to compare global and regional (FreeSurfer lobestrict output) BAG (i.e. predicted minus chronological age) between individuals with one of five primary substance use disorders as well as healthy controls.
FINDINGS RESULTS
Alcohol use disorder (β = -5.49, t = -5.51, p < 0.001) was associated with higher global BAG, whereas amphetamine-type stimulant use disorder (β = 3.44, t = 2.42, p = 0.02) was associated with lower global BAG in the separate substance-specific models.
CONCLUSIONS CONCLUSIONS
People with alcohol use disorder appear to have a higher brain-age gap than people without alcohol use disorder, which is consistent with other evidence of the negative impact of alcohol on the brain.

Identifiants

pubmed: 39165145
doi: 10.1111/add.16621
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : CIHR
Pays : Canada
Organisme : NIDA NIH HHS
ID : R01DA014100
Pays : United States
Organisme : CONACYT-FOSISS
ID : 0201493
Organisme : CONACYT-Catedras
ID : 2358948
Organisme : Netherlands Organisation for Health Research and Development
Organisme : NIH HHS
ID : DA051922
Pays : United States
Organisme : AI and Val Rosenstrauss Senior Research Fellowship
Organisme : National Health and Medical Research (NHMRC) Investigator Grant
ID : 2016833
Organisme : Australian Catholic University competitive scheme
Organisme : NIAAA NIH HHS
ID : ZIAAA000125
Pays : United States
Organisme : Division of Intramural Clinical and Biological Research
Organisme : NHMRC Investigator Leadership
ID : 2017962
Organisme : University of Melbourne Dame Kate Campbell fellowship
Organisme : NIH HHS
ID : RO1MH129832
Pays : United States
Organisme : NHMRC Investigator Leadership Grant
ID : 2009464

Investigateurs

A Batalla (A)
K T Brady (KT)
J Cousijn (J)
A Dagher (A)
F M Filbey (FM)
J J Foxe (JJ)
E A Garza-Villarreal (EA)
A E Goudriaan (AE)
R H Hester (RH)
K E Hutchison (KE)
A M Kaag (AM)
E Kroon (E)
C R Li (CR)
E D London (ED)
V Lorenzetti (V)
M Luijten (M)
R Martin-Santos (R)
A L McRae (AL)
R Momenan (R)
M P Paulus (MP)
G D Pearlson (GD)
L Reneman (L)
R Salas (R)
L Schmaal (L)
M L J Schouw (MLJ)
R Sinha (R)
N Solowij (N)
E A Stein (EA)
R J Van Holst (RJ)
D J Veltman (DJ)
A Verdejo-García (A)
R W Wiers (RW)
M Yucel (M)

Informations de copyright

© 2024 The Author(s). Addiction published by John Wiley & Sons Ltd on behalf of Society for the Study of Addiction.

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Auteurs

Freda Scheffler (F)

Neuroscience Institute, University of Cape Town, Cape Town, South Africa.
Department of Psychiatry and Mental Health, University of Cape Town, Cape Town, South Africa.

Jonathan Ipser (J)

Neuroscience Institute, University of Cape Town, Cape Town, South Africa.
Department of Psychiatry and Mental Health, University of Cape Town, Cape Town, South Africa.

Devarshi Pancholi (D)

Department of Psychiatry, University of Vermont College of Medicine, Burlington, USA.

Alistair Murphy (A)

Department of Psychiatry, University of Vermont College of Medicine, Burlington, USA.

Zhipeng Cao (Z)

Department of Psychiatry, University of Vermont College of Medicine, Burlington, USA.

Jonatan Ottino-González (J)

Department of Pediatrics, Division of Endocrinology, Diabetes, and Metabolism, Children's Hospital Los Angeles, Los Angeles, USA.

Paul M Thompson (PM)

Imaging Genetics Center, Mark and Mary Stevens Institute for Neuroimaging and Informatics, Department of Neurology, Keck School of Medicine, University of Southern California, Marina del Rey, CA, USA.

Steve Shoptaw (S)

Department of Family Medicine, UCLA, Los Angeles, CA, USA.
University of Cape Town, Cape Town, South Africa.

Patricia Conrod (P)

Department of Psychiatry, Université de Montreal, CHU Ste Justine Hospital, Montreal, Canada.

Scott Mackey (S)

Department of Psychiatry, University of Vermont College of Medicine, Burlington, USA.

Hugh Garavan (H)

Department of Psychiatry, University of Vermont College of Medicine, Burlington, USA.

Dan J Stein (DJ)

Neuroscience Institute, University of Cape Town, Cape Town, South Africa.
Department of Psychiatry and Mental Health, University of Cape Town, Cape Town, South Africa.
South African Medical Research Council (SAMRC) Unit on Risk and Resilience in Mental Disorders, University of Cape Town, Cape Town, South Africa.

Classifications MeSH