Contrast-Induced Encephalopathy: A Case Series Analysis.


Journal

Annals of Indian Academy of Neurology
ISSN: 0972-2327
Titre abrégé: Ann Indian Acad Neurol
Pays: India
ID NLM: 101273955

Informations de publication

Date de publication:
01 Jul 2024
Historique:
received: 09 07 2024
accepted: 11 08 2024
medline: 29 8 2024
pubmed: 29 8 2024
entrez: 28 8 2024
Statut: ppublish

Résumé

Contrast-induced encephalopathy (CIE) is a rare adverse event linked to intravascular use of iodine-containing contrast media. The prevalence of CIE could increase in the future due to growing numbers of endovascular procedures. We provide insights from a case series of 7 patients. Cases from 3 centers were collected based on existing academic collaborations, and key factors were extracted to illustrate development and management of CIE. In our retrospective case-series analysis of 7 cases from 3 countries, affected patients had an equal distribution of sex (4 women, 3 men) and a median age of 75 (IQR 63-77). Common risk factors included hypertension (5/7), hyperlipidemia (5/7), previous stroke (3/7), and type 2 diabetes (3/7). CIE developed in 3 cases after endovascular thrombectomy (EVT) for stroke, in 2 cases after aneurysm treatment, in 1 case after cardiac catheterization, and in 1 case after diagnostic computed tomography (CT) angiography without an endovascular procedure. The median procedure time was 48 min (IQR 40-81). All patients received non-ionic, low-osmolar contrast agents with volumes ranging from 100-300 ml. Symptom onset was close to contrast administration, with stroke-like neurological deficits being most common (4/7). Prednisolone was the most frequently used medication to treat the symptoms (4/7). Symptom resolution occurred in 4 out of 7 patients within two to several days, and 1 patient died, but without clear connection to CIE. CIE is a rare and possibly underrecognized condition, but fortunately, with a favorable outcome in most cases.

Sections du résumé

BACKGROUND BACKGROUND
Contrast-induced encephalopathy (CIE) is a rare adverse event linked to intravascular use of iodine-containing contrast media. The prevalence of CIE could increase in the future due to growing numbers of endovascular procedures. We provide insights from a case series of 7 patients.
METHODS METHODS
Cases from 3 centers were collected based on existing academic collaborations, and key factors were extracted to illustrate development and management of CIE.
RESULTS RESULTS
In our retrospective case-series analysis of 7 cases from 3 countries, affected patients had an equal distribution of sex (4 women, 3 men) and a median age of 75 (IQR 63-77). Common risk factors included hypertension (5/7), hyperlipidemia (5/7), previous stroke (3/7), and type 2 diabetes (3/7). CIE developed in 3 cases after endovascular thrombectomy (EVT) for stroke, in 2 cases after aneurysm treatment, in 1 case after cardiac catheterization, and in 1 case after diagnostic computed tomography (CT) angiography without an endovascular procedure. The median procedure time was 48 min (IQR 40-81). All patients received non-ionic, low-osmolar contrast agents with volumes ranging from 100-300 ml. Symptom onset was close to contrast administration, with stroke-like neurological deficits being most common (4/7). Prednisolone was the most frequently used medication to treat the symptoms (4/7). Symptom resolution occurred in 4 out of 7 patients within two to several days, and 1 patient died, but without clear connection to CIE.
CONCLUSION CONCLUSIONS
CIE is a rare and possibly underrecognized condition, but fortunately, with a favorable outcome in most cases.

Identifiants

pubmed: 39196808
doi: 10.4103/aian.aian_548_24
pii: 02223306-202427040-00010
doi:

Types de publication

Journal Article

Langues

eng

Pagination

403-407

Informations de copyright

Copyright © 2024 Copyright: © 2024 Annals of Indian Academy of Neurology.

Références

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Auteurs

Alexander Stebner (A)

Department of Diagnostic Imaging, Foothills Medical Centre, University of Calgary, Canada.
Department of Clinical Neurosciences, Foothills Medical Centre, University of Calgary, Canada.
Institute of Radiology, Cantonal Hospital Münsterlingen, Münsterlingen, Switzerland.

Salome Bosshart (S)

Department of Diagnostic Imaging, Foothills Medical Centre, University of Calgary, Canada.
Department of Clinical Neurosciences, Foothills Medical Centre, University of Calgary, Canada.

Nicolin Hainc (N)

Department of Neuroradiology, Clinical Neuroscience Center, University Hospital Zurich, University of Zurich, Zurich, Switzerland.

Nathalie Nierobisch (N)

Department of Neuroradiology, Clinical Neuroscience Center, University Hospital Zurich, University of Zurich, Zurich, Switzerland.

Marialuisa Zedde (M)

Neurology Unit, Stroke Unit, Azienda USL-IRCCS di Reggio Emilia, Reggio Emilia, Italy.

Rosario Pascarella (R)

Neuroradiology Unit, Azienda USL-IRCCS di Reggio Emilia, Reggio Emilia, Italy.

Mayank Goyal (M)

Department of Diagnostic Imaging, Foothills Medical Centre, University of Calgary, Canada.
Department of Clinical Neurosciences, Foothills Medical Centre, University of Calgary, Canada.

Johanna Ospel (J)

Department of Diagnostic Imaging, Foothills Medical Centre, University of Calgary, Canada.
Department of Clinical Neurosciences, Foothills Medical Centre, University of Calgary, Canada.

Classifications MeSH