Genomic analyses of intricate interaction of TE-lncRNA overlapping genes with miRNAs in human diseases.

Bioinformatics Human disease Transposable elements long non-coding RNA microRNA

Journal

Genes & genomics
ISSN: 2092-9293
Titre abrégé: Genes Genomics
Pays: Korea (South)
ID NLM: 101481027

Informations de publication

Date de publication:
31 Aug 2024
Historique:
received: 09 05 2024
accepted: 09 07 2024
medline: 1 9 2024
pubmed: 1 9 2024
entrez: 31 8 2024
Statut: aheadofprint

Résumé

Transposable elements (TEs) are known to be inserted into genome to create transcript isoforms or to generate long non-coding RNA (lncRNA) sequences. The insertion of TEs generates a gene protein sequence within the genome, but also provides a microRNA (miRNA) regulatory region. To determine the effect of gene sequence changes caused by TE insertion on miRNA binding and to investigate the formation of an overlapping lncRNA that represses it. The distribution of overlapping regions between exons and TE regions with lncRNA was examined using the Bedtools. miRNAs that can bind to those overlapping regions were identified through the miRDB web program. For TE-lncRNA overlapping genes, bioinformatic analysis was conducted using DAVID web database. Differential expression analysis was conducted using data from the GEO dataset and TCGA. Most TEs were distributed more frequently in untranslated regions than open reading frames. There were 30 annotated TE-lncRNA overlapping genes with same strand that could bind to the same miRNA. As a result of identifying the association between these 30 genes and diseases, TGFB2, FCGR2A, DCTN5, and IFI6 were associated with breast cancer, and HMGCS1, FRMD4A, EDNRB, and SNCA were associated with Alzheimer's disease. Analysis of the GEO and TCGA data showed that the relevant expression of miR-891a and miR-28, which bind to the TE overlapping region of DCTN5 and HMGCS1, decreased. This study indicates that the interaction between TE-lncRNA overlapping genes and miRNAs can affect disease progression.

Sections du résumé

BACKGROUND BACKGROUND
Transposable elements (TEs) are known to be inserted into genome to create transcript isoforms or to generate long non-coding RNA (lncRNA) sequences. The insertion of TEs generates a gene protein sequence within the genome, but also provides a microRNA (miRNA) regulatory region.
OBJECTIVE OBJECTIVE
To determine the effect of gene sequence changes caused by TE insertion on miRNA binding and to investigate the formation of an overlapping lncRNA that represses it.
METHODS METHODS
The distribution of overlapping regions between exons and TE regions with lncRNA was examined using the Bedtools. miRNAs that can bind to those overlapping regions were identified through the miRDB web program. For TE-lncRNA overlapping genes, bioinformatic analysis was conducted using DAVID web database. Differential expression analysis was conducted using data from the GEO dataset and TCGA.
RESULTS RESULTS
Most TEs were distributed more frequently in untranslated regions than open reading frames. There were 30 annotated TE-lncRNA overlapping genes with same strand that could bind to the same miRNA. As a result of identifying the association between these 30 genes and diseases, TGFB2, FCGR2A, DCTN5, and IFI6 were associated with breast cancer, and HMGCS1, FRMD4A, EDNRB, and SNCA were associated with Alzheimer's disease. Analysis of the GEO and TCGA data showed that the relevant expression of miR-891a and miR-28, which bind to the TE overlapping region of DCTN5 and HMGCS1, decreased.
CONCLUSION CONCLUSIONS
This study indicates that the interaction between TE-lncRNA overlapping genes and miRNAs can affect disease progression.

Identifiants

pubmed: 39215947
doi: 10.1007/s13258-024-01547-1
pii: 10.1007/s13258-024-01547-1
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© 2024. The Author(s) under exclusive licence to The Genetics Society of Korea.

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Auteurs

Du Hyeong Lee (DH)

Department of Integrated Biological Sciences, Pusan National University, Busan, 46241, Republic of Korea.
Institute of Systems Biology, Pusan National University, Busan, 46241, Republic of Korea.

Eun Gyung Park (EG)

Department of Integrated Biological Sciences, Pusan National University, Busan, 46241, Republic of Korea.
Institute of Systems Biology, Pusan National University, Busan, 46241, Republic of Korea.

Jung-Min Kim (JM)

Department of Integrated Biological Sciences, Pusan National University, Busan, 46241, Republic of Korea.
Institute of Systems Biology, Pusan National University, Busan, 46241, Republic of Korea.

Hae Jin Shin (HJ)

Department of Integrated Biological Sciences, Pusan National University, Busan, 46241, Republic of Korea.
Institute of Systems Biology, Pusan National University, Busan, 46241, Republic of Korea.

Yun Ju Lee (YJ)

Department of Integrated Biological Sciences, Pusan National University, Busan, 46241, Republic of Korea.
Institute of Systems Biology, Pusan National University, Busan, 46241, Republic of Korea.

Hyeon-Su Jeong (HS)

Department of Integrated Biological Sciences, Pusan National University, Busan, 46241, Republic of Korea.
Institute of Systems Biology, Pusan National University, Busan, 46241, Republic of Korea.

Hyun-Young Roh (HY)

Institute of Systems Biology, Pusan National University, Busan, 46241, Republic of Korea.
Department of Biological Sciences, College of Natural Sciences, Pusan National University, Busan, 46241, Republic of Korea.

Woo Ryung Kim (WR)

Department of Integrated Biological Sciences, Pusan National University, Busan, 46241, Republic of Korea.
Institute of Systems Biology, Pusan National University, Busan, 46241, Republic of Korea.

Hongseok Ha (H)

Institute of Endemic Disease, Medical Research Center, Seoul National University, Seoul, 03080, Republic of Korea.

Sang-Woo Kim (SW)

Department of Integrated Biological Sciences, Pusan National University, Busan, 46241, Republic of Korea.
Department of Biological Sciences, College of Natural Sciences, Pusan National University, Busan, 46241, Republic of Korea.

Yung Hyun Choi (YH)

Department of Biochemistry, College of Oriental Medicine, Dong-Eui University, Busan, 47227, Republic of Korea.

Heui-Soo Kim (HS)

Institute of Systems Biology, Pusan National University, Busan, 46241, Republic of Korea. khs307@pusan.ac.kr.
Department of Biological Sciences, College of Natural Sciences, Pusan National University, Busan, 46241, Republic of Korea. khs307@pusan.ac.kr.

Classifications MeSH