Low dose TamOxifen and LifestylE changes for bReast cANcer prevention (TOLERANT study): Study protocol of a randomized phase II biomarker trial in women at increased risk for breast cancer.


Journal

PloS one
ISSN: 1932-6203
Titre abrégé: PLoS One
Pays: United States
ID NLM: 101285081

Informations de publication

Date de publication:
2024
Historique:
received: 05 08 2024
accepted: 13 08 2024
medline: 3 9 2024
pubmed: 3 9 2024
entrez: 3 9 2024
Statut: epublish

Résumé

Breast Cancer (BC) prevention strategies range from lifestyle changes such as increasing physical activity and reducing body weight to preventive drugs like tamoxifen, known to reduce BC incidence in high-risk women. Sex Hormone Binding Globulin (SHBG) is related to BC risk due to its ability to bind circulating estradiol at high affinity and to regulate estradiol action. A study protocol is presented based on the assessment of the effect of different interventions such as tamoxifen at 10 mg every other day (LDT), intermittent caloric restriction (ICR) two days per week, lifestyle intervention (LI, step counter use) and their combination on the modulation of SHBG and several other biomarkers associated to BC. A randomized phase II biomarker study will be conducted in 4 Italian centers. Unaffected women aged between 18 and 70 years, carriers of a germline pathogenetic variant (BRCA1, BRCA2, PALB2, or other moderate penetrance genes), or with a >5% BC risk at 10 years (according to the Tyrer-Cuzick or the Breast Cancer Surveillance Consortium Risk models) or with a previous diagnosis of intraepithelial neoplasia will be eligible. A total of 200 participants will be randomized to one of the four arms: LDT; LDT + ICR; LI; LI + ICR. Interventions will span six months, with baseline and follow-up clinic visits and interim phone calls. The aim of the study is to verify whether LDT increases circulating SHBG more than LI with or without ICR after 6 months. Secondary objectives include assessing HOMA-index, inflammatory markers, adiponectin/leptin ratio, quality of life (QoL), safety, toxicity, mammographic density, and changes in microbiome composition across groups. The study's innovation lies in its inclusion of diverse BC risk categories and combination of pharmaceutical and behavioral interventions, potentially enhancing intervention efficacy while balancing tamoxifen's side effects on QoL, especially menopausal symptoms. EuCT number:2023-503994-39-00; Clinical trials.gov NCT06033092.

Sections du résumé

BACKGROUND BACKGROUND
Breast Cancer (BC) prevention strategies range from lifestyle changes such as increasing physical activity and reducing body weight to preventive drugs like tamoxifen, known to reduce BC incidence in high-risk women. Sex Hormone Binding Globulin (SHBG) is related to BC risk due to its ability to bind circulating estradiol at high affinity and to regulate estradiol action. A study protocol is presented based on the assessment of the effect of different interventions such as tamoxifen at 10 mg every other day (LDT), intermittent caloric restriction (ICR) two days per week, lifestyle intervention (LI, step counter use) and their combination on the modulation of SHBG and several other biomarkers associated to BC.
METHODS METHODS
A randomized phase II biomarker study will be conducted in 4 Italian centers. Unaffected women aged between 18 and 70 years, carriers of a germline pathogenetic variant (BRCA1, BRCA2, PALB2, or other moderate penetrance genes), or with a >5% BC risk at 10 years (according to the Tyrer-Cuzick or the Breast Cancer Surveillance Consortium Risk models) or with a previous diagnosis of intraepithelial neoplasia will be eligible. A total of 200 participants will be randomized to one of the four arms: LDT; LDT + ICR; LI; LI + ICR. Interventions will span six months, with baseline and follow-up clinic visits and interim phone calls.
DISCUSSION CONCLUSIONS
The aim of the study is to verify whether LDT increases circulating SHBG more than LI with or without ICR after 6 months. Secondary objectives include assessing HOMA-index, inflammatory markers, adiponectin/leptin ratio, quality of life (QoL), safety, toxicity, mammographic density, and changes in microbiome composition across groups. The study's innovation lies in its inclusion of diverse BC risk categories and combination of pharmaceutical and behavioral interventions, potentially enhancing intervention efficacy while balancing tamoxifen's side effects on QoL, especially menopausal symptoms.
TRIAL REGISTRATION BACKGROUND
EuCT number:2023-503994-39-00; Clinical trials.gov NCT06033092.

Identifiants

pubmed: 39226292
doi: 10.1371/journal.pone.0309511
pii: PONE-D-24-29473
doi:

Substances chimiques

Tamoxifen 094ZI81Y45
Sex Hormone-Binding Globulin 0
Biomarkers, Tumor 0
Antineoplastic Agents, Hormonal 0
SHBG protein, human 0

Banques de données

ClinicalTrials.gov
['NCT06033092']

Types de publication

Journal Article Clinical Trial, Phase II Randomized Controlled Trial Clinical Trial Protocol Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

e0309511

Informations de copyright

Copyright: © 2024 Guerrieri-Gonzaga et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

Déclaration de conflit d'intérêts

The authors have declared that no competing interests exist.

Auteurs

Aliana Guerrieri-Gonzaga (A)

Division of Cancer Prevention and Genetics, European Institute of Oncology IRCCS, Milan, Italy.

Davide Serrano (D)

Division of Cancer Prevention and Genetics, European Institute of Oncology IRCCS, Milan, Italy.

Patrizia Gnagnarella (P)

Division of Epidemiology and Biostatistics, European Institute of Oncology IRCCS, Milan, Italy.

Harriet Johansson (H)

Division of Cancer Prevention and Genetics, European Institute of Oncology IRCCS, Milan, Italy.

Stefania Zovato (S)

Familial Cancer Unit, Veneto Institute of Oncology IOV IRCSS, Padova, Italy.

Mariateresa Nardi (M)

Familial Cancer Unit, Veneto Institute of Oncology IOV IRCSS, Padova, Italy.

Matilde Pensabene (M)

Division of Breast Oncology, Istituto Nazionale Tumori-IRCCS-Fondazione G. Pascale, Naples, Italy.

Simona Buccolo (S)

Division of Breast Oncology, Istituto Nazionale Tumori-IRCCS-Fondazione G. Pascale, Naples, Italy.

Andrea DeCensi (A)

Division of Medical Oncology, E.O. Galliera Hospital, Genoa, Italy.
Wolfson Institute of Preventive Medicine, Queen Mary University of London, London, United Kingdom.

Irene Maria Briata (IM)

Division of Medical Oncology, E.O. Galliera Hospital, Genoa, Italy.

Luigi Pistelli (L)

Division of Breast Oncology, Istituto Nazionale Tumori-IRCCS-Fondazione G. Pascale, Naples, Italy.

Clementina Sansone (C)

Stazione Zoologica Anton Dohrn, Istituto Nazionale di Biologia, Ecologia e Biotecnologie Marine, University of Naples "Federico II", Naples, Italy.

Sara Mannucci (S)

Division of Cancer Prevention and Genetics, European Institute of Oncology IRCCS, Milan, Italy.

Valentina Aristarco (V)

Division of Cancer Prevention and Genetics, European Institute of Oncology IRCCS, Milan, Italy.

Debora Macis (D)

Division of Cancer Prevention and Genetics, European Institute of Oncology IRCCS, Milan, Italy.

Matteo Lazzeroni (M)

Division of Cancer Prevention and Genetics, European Institute of Oncology IRCCS, Milan, Italy.

Gaetano Aurilio (G)

Division of Cancer Prevention and Genetics, European Institute of Oncology IRCCS, Milan, Italy.

Chiara Arianna Accornero (CA)

Division of Cancer Prevention and Genetics, European Institute of Oncology IRCCS, Milan, Italy.

Sara Gandini (S)

Department of Experimental Oncology, IEO, European Institute of Oncology IRCCS, Milan, Italy.

Bernardo Bonanni (B)

Division of Cancer Prevention and Genetics, European Institute of Oncology IRCCS, Milan, Italy.

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Classifications MeSH