Clinical and biochemical responses to treatment of uncomplicated severe acute malnutrition: a multicenter observational cohort from the OptiDiag study.


Journal

The American journal of clinical nutrition
ISSN: 1938-3207
Titre abrégé: Am J Clin Nutr
Pays: United States
ID NLM: 0376027

Informations de publication

Date de publication:
Sep 2024
Historique:
received: 21 11 2023
revised: 18 05 2024
accepted: 28 05 2024
medline: 5 9 2024
pubmed: 5 9 2024
entrez: 4 9 2024
Statut: ppublish

Résumé

Severe acute malnutrition (SAM) can be diagnosed using weight-for-height Z-score (WHZ) and/or mid-upper arm circumference (MUAC). Although some favor using MUAC alone, valuing its presumed ability to identify children at greatest need for nutritional care, the functional severity and physiological responses to treatment in children with varying deficits in WHZ and MUAC remain inadequately characterized. We aimed to compare clinical and biochemical responses to treatment in children with 1) both low MUAC and low WHZ, 2) low MUAC-only, and 3) low WHZ-only. A multicenter, observational cohort study was conducted in children aged 6-59 mo with nonedematous, uncomplicated SAM in Bangladesh, Burkina Faso, and Liberia. Anthropometric measurements and critical indicators were collected 3 times during treatment; metrics included clinical status, nutritional status, viability, and serum leptin, a biomarker of mortality risk in SAM. Children with combined MUAC and WHZ deficits had greater increases in leptin levels during treatment than those with low MUAC alone, showing a 34.4% greater increase on the second visit (95% confidence interval [CI]: 7.6%, 43.6%; P = 0.02) and a 34.3% greater increase on the third visit (95% CI: 13.2%, 50.3%; P = 0.01). Similarly, weight gain velocity was higher by 1.56 g/kg/d in the combined deficit group (95% CI: 0.38, 2.75; P = 0.03) compared with children with low MUAC-only. Children with combined deficits had higher rates of iron deficiency and wasting while those with low WHZ alone and combined deficits had higher rates of tachypnea and pneumonia during treatment. Given the comparable treatment responses of children with low WHZ alone and those with low MUAC alone, and the greater vulnerability at admission and during treatment in those with combined deficits, our findings support retaining WHZ as an independent diagnostic and admission criterion of SAM, alongside MUAC. This trial was registered at www. gov/study/NCT03400930 as NCT03400930.

Sections du résumé

BACKGROUND BACKGROUND
Severe acute malnutrition (SAM) can be diagnosed using weight-for-height Z-score (WHZ) and/or mid-upper arm circumference (MUAC). Although some favor using MUAC alone, valuing its presumed ability to identify children at greatest need for nutritional care, the functional severity and physiological responses to treatment in children with varying deficits in WHZ and MUAC remain inadequately characterized.
OBJECTIVE OBJECTIVE
We aimed to compare clinical and biochemical responses to treatment in children with 1) both low MUAC and low WHZ, 2) low MUAC-only, and 3) low WHZ-only.
METHODS METHODS
A multicenter, observational cohort study was conducted in children aged 6-59 mo with nonedematous, uncomplicated SAM in Bangladesh, Burkina Faso, and Liberia. Anthropometric measurements and critical indicators were collected 3 times during treatment; metrics included clinical status, nutritional status, viability, and serum leptin, a biomarker of mortality risk in SAM.
RESULTS RESULTS
Children with combined MUAC and WHZ deficits had greater increases in leptin levels during treatment than those with low MUAC alone, showing a 34.4% greater increase on the second visit (95% confidence interval [CI]: 7.6%, 43.6%; P = 0.02) and a 34.3% greater increase on the third visit (95% CI: 13.2%, 50.3%; P = 0.01). Similarly, weight gain velocity was higher by 1.56 g/kg/d in the combined deficit group (95% CI: 0.38, 2.75; P = 0.03) compared with children with low MUAC-only. Children with combined deficits had higher rates of iron deficiency and wasting while those with low WHZ alone and combined deficits had higher rates of tachypnea and pneumonia during treatment.
CONCLUSIONS CONCLUSIONS
Given the comparable treatment responses of children with low WHZ alone and those with low MUAC alone, and the greater vulnerability at admission and during treatment in those with combined deficits, our findings support retaining WHZ as an independent diagnostic and admission criterion of SAM, alongside MUAC. This trial was registered at www.
CLINICALTRIALS RESULTS
gov/study/NCT03400930 as NCT03400930.

Identifiants

pubmed: 39232601
pii: S0002-9165(24)00526-4
doi: 10.1016/j.ajcnut.2024.05.029
pii:
doi:

Substances chimiques

Leptin 0

Banques de données

ClinicalTrials.gov
['NCT03400930']

Types de publication

Journal Article Multicenter Study Observational Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

570-582

Informations de copyright

Copyright © 2024 American Society for Nutrition. Published by Elsevier Inc. All rights reserved.

Auteurs

Lishi Deng (L)

Department of Food Technology, Safety and Health, Faculty of Bioscience Engineering, Ghent University, Ghent, Belgium.

Alemayehu Argaw (A)

Department of Food Technology, Safety and Health, Faculty of Bioscience Engineering, Ghent University, Ghent, Belgium.

Benjamin Guesdon (B)

Department of Expertise and Advocacy, Action Contre la Faim, Paris, France.

Michael Freemark (M)

Division of Pediatric Endocrinology and Diabetes, Duke University Medical Center, Durham, NC, United States; Duke Molecular Physiology Institute (DMPI), Duke University Medical Center, Durham, NC, United States.

Dominique Roberfroid (D)

Belgian Health Care Knowledge Center, Brussels, Belgium; Medicine Department, Faculty of Medicine, University of Namur, Namur, Belgium.

Issa A Kemokai (IA)

Action Contre la Faim, Monrovia, Liberia.

Md Rayhan Mostak (MR)

Palli Karma-Sahayak Foundation, Dhaka, Bangladesh; Action Against Hunger, Dhaka, Bangladesh.

Md Abdul Alim (MA)

Directorate General of Health Services, Bangladesh; Department of Public Health and Primary Care, Ghent University, Ghent, Belgium.

Md Abdul Hashem Khan (MAH)

Community Based Health Care (CBHC), Dhaka, Bangladesh.

Michael Muehlbauer (M)

Duke Molecular Physiology Institute (DMPI), Duke University Medical Center, Durham, NC, United States.

Murad Md Shamsher Tabris Khan (MMST)

National Nutrition Service, Institute of Public Health Nutrition, Dhaka, Bangladesh.

Luke Bawo (L)

Ministry of Health, Monrovia, Liberia.

Nelson K Dunbar (NK)

Ministry of Health, Monrovia, Liberia.

Curtis H Taylor (CH)

Pacific Institute for Research and Evaluation (UL-PIRE), University of Liberia, Monrovia, Liberia.

Helene Fouillet (H)

Université Paris-Saclay, AgroParisTech, INRAE, UMR PNCA, Palaiseau, France.

Jean-Francois Huneau (JF)

Université Paris-Saclay, AgroParisTech, INRAE, UMR PNCA, Palaiseau, France.

Carl Lachat (C)

Department of Food Technology, Safety and Health, Faculty of Bioscience Engineering, Ghent University, Ghent, Belgium.

Patrick Kolsteren (P)

Department of Food Technology, Safety and Health, Faculty of Bioscience Engineering, Ghent University, Ghent, Belgium.

Trenton Dailey-Chwalibóg (T)

Department of Food Technology, Safety and Health, Faculty of Bioscience Engineering, Ghent University, Ghent, Belgium; Agence de Formation de Recherche et d'Expertise en Santé pour l'Afrique (AFRICSanté), Bobo-Dioulasso, Burkina Faso. Electronic address: trenton@dailey-chwalibog.com.

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Classifications MeSH