Asymptomatic carriage of Plasmodium falciparum in children living in a hyperendemic area occurs independently of IgG responses but is associated with a balanced inflammatory cytokine ratio.


Journal

Malaria journal
ISSN: 1475-2875
Titre abrégé: Malar J
Pays: England
ID NLM: 101139802

Informations de publication

Date de publication:
04 Sep 2024
Historique:
received: 06 06 2024
accepted: 17 08 2024
medline: 5 9 2024
pubmed: 5 9 2024
entrez: 5 9 2024
Statut: epublish

Résumé

Asymptomatic carriage of infected red blood cells (iRBCs) can be prevalent in communities regardless of transmission patterns and can occur with infection of different Plasmodium species. Clinical immunity dampens the inflammatory responses leading to disease symptoms in malaria. The aim of this study was to define the immunological correlates of asymptomatic carriage of Plasmodium falciparum in a highly exposed population. 142 asymptomatic Plasmodium-infected individuals greater than 2 years of age without fever (body temperature <37.5 ℃) were followed weekly for 10 weeks before being treated with artemisinin-based combination therapy (ACT). Plasma levels of 38 cytokines were measured at baseline by Luminex and the quantity and growth inhibitory activities of circulating parasite-reactive antibodies measured. The Plasmodium antigen tested included P. falciparum merozoite extract (ME) and schizont extract (SE), and the recombinant proteins erythrocyte binding antigen 175 (EBA-175) and merozoite surface protein 1 (MSP-1 Median levels of IgG against P. falciparum EBA-175 and MSP-1 The above findings indicate that asymptomatic carriage of P. falciparum in children living in a hyperendemic area occurs independently of IgG but is associated with a balanced inflammatory cytokine ratio.

Sections du résumé

BACKGROUND BACKGROUND
Asymptomatic carriage of infected red blood cells (iRBCs) can be prevalent in communities regardless of transmission patterns and can occur with infection of different Plasmodium species. Clinical immunity dampens the inflammatory responses leading to disease symptoms in malaria. The aim of this study was to define the immunological correlates of asymptomatic carriage of Plasmodium falciparum in a highly exposed population.
METHODS METHODS
142 asymptomatic Plasmodium-infected individuals greater than 2 years of age without fever (body temperature <37.5 ℃) were followed weekly for 10 weeks before being treated with artemisinin-based combination therapy (ACT). Plasma levels of 38 cytokines were measured at baseline by Luminex and the quantity and growth inhibitory activities of circulating parasite-reactive antibodies measured. The Plasmodium antigen tested included P. falciparum merozoite extract (ME) and schizont extract (SE), and the recombinant proteins erythrocyte binding antigen 175 (EBA-175) and merozoite surface protein 1 (MSP-1
RESULTS RESULTS
Median levels of IgG against P. falciparum EBA-175 and MSP-1
CONCLUSION CONCLUSIONS
The above findings indicate that asymptomatic carriage of P. falciparum in children living in a hyperendemic area occurs independently of IgG but is associated with a balanced inflammatory cytokine ratio.

Identifiants

pubmed: 39232787
doi: 10.1186/s12936-024-05086-8
pii: 10.1186/s12936-024-05086-8
doi:

Substances chimiques

Immunoglobulin G 0
Cytokines 0
Antibodies, Protozoan 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

268

Informations de copyright

© 2024. The Author(s).

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Auteurs

Balotin Fogang (B)

Molecular Parasitology Laboratory, Centre Pasteur du Cameroun, BP 1274, Yaounde, Cameroon.
Department of Animal Biology and Physiology of the University of Yaoundé I, BP 812, Yaounde, Cameroon.

Matthieu Schoenhals (M)

Molecular Parasitology Laboratory, Centre Pasteur du Cameroun, BP 1274, Yaounde, Cameroon.

Franklin M Maloba (FM)

Molecular Parasitology Laboratory, Centre Pasteur du Cameroun, BP 1274, Yaounde, Cameroon.

Marie Florence Biabi (MF)

Molecular Parasitology Laboratory, Centre Pasteur du Cameroun, BP 1274, Yaounde, Cameroon.
Department of Biochemistry, University of Douala, BP 24157, Douala, Cameroon.

Estelle Essangui (E)

Molecular Parasitology Laboratory, Centre Pasteur du Cameroun, BP 1274, Yaounde, Cameroon.

Christiane Donkeu (C)

Molecular Parasitology Laboratory, Centre Pasteur du Cameroun, BP 1274, Yaounde, Cameroon.
Department of Animal Biology and Physiology of the University of Yaoundé I, BP 812, Yaounde, Cameroon.

Glwadys Cheteug (G)

Molecular Parasitology Laboratory, Centre Pasteur du Cameroun, BP 1274, Yaounde, Cameroon.
Department of Medical Laboratory Sciences, University of Buea, BP 63, Buea, Cameroon.

Marie Kapen (M)

Molecular Parasitology Laboratory, Centre Pasteur du Cameroun, BP 1274, Yaounde, Cameroon.

Rodrigue Keumoe (R)

Molecular Parasitology Laboratory, Centre Pasteur du Cameroun, BP 1274, Yaounde, Cameroon.

Sylvie Kemleu (S)

Molecular Parasitology Laboratory, Centre Pasteur du Cameroun, BP 1274, Yaounde, Cameroon.

Sandrine Nsango (S)

Molecular Parasitology Laboratory, Centre Pasteur du Cameroun, BP 1274, Yaounde, Cameroon.
Faculty of Medicine and Pharmaceutical Sciences, University of Douala, BP 2701, Douala, Cameroon.

Douglas H Cornwall (DH)

Department of Pathology, University of Utah, 15 N Medical Drive, Salt Lake City, 84112, USA.

Carole Eboumbou (C)

Molecular Parasitology Laboratory, Centre Pasteur du Cameroun, BP 1274, Yaounde, Cameroon.
Faculty of Medicine and Pharmaceutical Sciences, University of Douala, BP 2701, Douala, Cameroon.

Ronald Perraut (R)

Molecular Parasitology Laboratory, Centre Pasteur du Cameroun, BP 1274, Yaounde, Cameroon.

Rosette Megnekou (R)

Department of Animal Biology and Physiology of the University of Yaoundé I, BP 812, Yaounde, Cameroon.

Tracey J Lamb (TJ)

Department of Pathology, University of Utah, 15 N Medical Drive, Salt Lake City, 84112, USA. tracey.lamb@path.utah.edu.

Lawrence S Ayong (LS)

Molecular Parasitology Laboratory, Centre Pasteur du Cameroun, BP 1274, Yaounde, Cameroon. layong05@yahoo.co.uk.

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