Association of Neutrophil Extracellular Traps burden with clinical and angiographic characteristics in patients with ST-elevation myocardial infarction.


Journal

Hellenic journal of cardiology : HJC = Hellenike kardiologike epitheorese
ISSN: 2241-5955
Titre abrégé: Hellenic J Cardiol
Pays: Netherlands
ID NLM: 101257381

Informations de publication

Date de publication:
06 Sep 2024
Historique:
received: 14 06 2024
revised: 23 08 2024
accepted: 02 09 2024
medline: 9 9 2024
pubmed: 9 9 2024
entrez: 8 9 2024
Statut: aheadofprint

Résumé

The precise triggers for atherosclerotic plaque rupture and the underlying pathophysiology of coronary thrombogenesis remain elusive. Polymorphonuclear neutrophils, particularly their formation of Neutrophil Extracellular Traps (NETs), have garnered attention in the context of coronary atherothrombosis. This study sought to explore the association of NETs burden with clinical and angiographic characteristics in ST-elevation myocardial infarction (STEMI) patients undergoing primary percutaneous coronary intervention (pPCI) and thrombus aspiration (TA). For this study 336 consecutive STEMI patients undergoing pPCI were considered for TA. Aspirated thrombi underwent histological analysis and NETs quantification via immunohistochemistry. Potential associations of clinical variables and angiographic outcomes with NETs burden were assessed. Manual TA was selectively performed in 72 cases with increased thrombotic burden and 60 thrombi were suitable for analysis and included in the current study. Most thrombi specimens displayed lytic features (63%) and almost three out of four were identified as white thrombi. Increased NETs burden was significantly associated with prolonged pain-to-balloon time (>300 minutes), OR=10.29 (95% CI 2.11-42.22, p=0.001) and stress-induced hyperglycemia OR=6.58 (95% CI 1.23-52.63, p<0.01) after multivariate regression analysis. Additionally, distal embolization, a predictor of adverse outcomes, was more frequent among patients with an elevated NETs burden OR=16.9 (95% CI 4.23-44.52, p<0.01). Elevated NETs burden in STEMI thrombi may be due to delayed reperfusion, stress-induced hyperglycemia and it is associated with increased risk of distal embolization. Further research is needed to elucidate the role of NETs as a potential therapeutic target in acute atherothrombosis.

Sections du résumé

BACKGROUND BACKGROUND
The precise triggers for atherosclerotic plaque rupture and the underlying pathophysiology of coronary thrombogenesis remain elusive. Polymorphonuclear neutrophils, particularly their formation of Neutrophil Extracellular Traps (NETs), have garnered attention in the context of coronary atherothrombosis. This study sought to explore the association of NETs burden with clinical and angiographic characteristics in ST-elevation myocardial infarction (STEMI) patients undergoing primary percutaneous coronary intervention (pPCI) and thrombus aspiration (TA).
METHODS METHODS
For this study 336 consecutive STEMI patients undergoing pPCI were considered for TA. Aspirated thrombi underwent histological analysis and NETs quantification via immunohistochemistry. Potential associations of clinical variables and angiographic outcomes with NETs burden were assessed.
RESULTS RESULTS
Manual TA was selectively performed in 72 cases with increased thrombotic burden and 60 thrombi were suitable for analysis and included in the current study. Most thrombi specimens displayed lytic features (63%) and almost three out of four were identified as white thrombi. Increased NETs burden was significantly associated with prolonged pain-to-balloon time (>300 minutes), OR=10.29 (95% CI 2.11-42.22, p=0.001) and stress-induced hyperglycemia OR=6.58 (95% CI 1.23-52.63, p<0.01) after multivariate regression analysis. Additionally, distal embolization, a predictor of adverse outcomes, was more frequent among patients with an elevated NETs burden OR=16.9 (95% CI 4.23-44.52, p<0.01).
CONCLUSION CONCLUSIONS
Elevated NETs burden in STEMI thrombi may be due to delayed reperfusion, stress-induced hyperglycemia and it is associated with increased risk of distal embolization. Further research is needed to elucidate the role of NETs as a potential therapeutic target in acute atherothrombosis.

Identifiants

pubmed: 39245435
pii: S1109-9666(24)00180-5
doi: 10.1016/j.hjc.2024.09.001
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright © 2024 Hellenic Society of Cardiology. Published by Elsevier Inc. All rights reserved.

Auteurs

Nikolaos Stalikas (N)

First Department of Cardiology, AHEPA University Hospital, Aristotle University of Thessaloniki.

Sofia-Eleni Tzorakoleftheraki (SE)

Pathology Department, Medical School, Aristotle University of Thessaloniki, Greece.

Efstratios Karagiannidis (E)

Second Department of Cardiology, Hippokration University Hospital, Aristotle University of Thessaloniki.

Matthaios Didagelos (M)

First Department of Cardiology, AHEPA University Hospital, Aristotle University of Thessaloniki.

Antonios Ziakas (A)

First Department of Cardiology, AHEPA University Hospital, Aristotle University of Thessaloniki.

Vasileios Kamperidis (V)

First Department of Cardiology, AHEPA University Hospital, Aristotle University of Thessaloniki.

George Giannakoulas (G)

First Department of Cardiology, AHEPA University Hospital, Aristotle University of Thessaloniki.

Vasileios Vassilikos (V)

Third department of Cardiology, Hippokration University Hospital, Aristotle University of Thessaloniki.

Triantafyllia Koletsa (T)

Pathology Department, Medical School, Aristotle University of Thessaloniki, Greece.

George Giannopoulos (G)

Third department of Cardiology, Hippokration University Hospital, Aristotle University of Thessaloniki. Electronic address: ggiann@auth.gr.

Classifications MeSH