8-hydroxy-2'-deoxyguanosine, a biomarker of oxidative DNA injury, in diabetic kidney disease.

8-Hydroxy-2′-deoxyguanosine Diabetic kidney disease Oxidative stress Type-2 diabetes

Journal

Nutrition, metabolism, and cardiovascular diseases : NMCD
ISSN: 1590-3729
Titre abrégé: Nutr Metab Cardiovasc Dis
Pays: Netherlands
ID NLM: 9111474

Informations de publication

Date de publication:
24 Aug 2024
Historique:
received: 30 07 2024
accepted: 22 08 2024
medline: 14 9 2024
pubmed: 14 9 2024
entrez: 13 9 2024
Statut: aheadofprint

Résumé

To gain insight into the extent of oxidative stress and DNA damage in diabetic kidney disease (DKD), a serious complication of diabetes, we compared the levels of the oxidative stress-related metabolite 8-hydroxy-2'-deoxyguanosine (8-OHdG) in a case-control study accurately matching diabetic patients with and without renal complications. We analyzed serum 8-OHdG in relation to clinical indicators of kidney function in a group of type-2 diabetes patients including 33 patients with DKD and 33 without DKD. Circulating levels of 8-OHdG were higher in patients with DKD than in those without (4.6 ± 0.7 ng/mL vs 4.0 ± 0.8 ng/mL, p = 0.002). In a logistic regression analysis adjusting for potential confounders, 8-OHdG was associated with DKD (OR: 2.90, 95%CI:1.15-7.34; p = 0.02) and in a linear regression model, a 1 ng/mL increase of this biomarker entailed a reduction of 11.5 mL/min/1.73 m This study supports the role of oxidative stress in the pathogenesis of diabetic nephropathy and highlights the potential of serum 8-OHdG as a biomarker for assessing oxidative stress and DNA damage in patients with diabetes and renal complications.

Sections du résumé

BACKGROUND AND AIMS OBJECTIVE
To gain insight into the extent of oxidative stress and DNA damage in diabetic kidney disease (DKD), a serious complication of diabetes, we compared the levels of the oxidative stress-related metabolite 8-hydroxy-2'-deoxyguanosine (8-OHdG) in a case-control study accurately matching diabetic patients with and without renal complications.
METHODS AND RESULTS RESULTS
We analyzed serum 8-OHdG in relation to clinical indicators of kidney function in a group of type-2 diabetes patients including 33 patients with DKD and 33 without DKD. Circulating levels of 8-OHdG were higher in patients with DKD than in those without (4.6 ± 0.7 ng/mL vs 4.0 ± 0.8 ng/mL, p = 0.002). In a logistic regression analysis adjusting for potential confounders, 8-OHdG was associated with DKD (OR: 2.90, 95%CI:1.15-7.34; p = 0.02) and in a linear regression model, a 1 ng/mL increase of this biomarker entailed a reduction of 11.5 mL/min/1.73 m
CONCLUSION CONCLUSIONS
This study supports the role of oxidative stress in the pathogenesis of diabetic nephropathy and highlights the potential of serum 8-OHdG as a biomarker for assessing oxidative stress and DNA damage in patients with diabetes and renal complications.

Identifiants

pubmed: 39271385
pii: S0939-4753(24)00325-9
doi: 10.1016/j.numecd.2024.08.015
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright © 2024 The Italian Diabetes Society, the Italian Society for the Study of Atherosclerosis, the Italian Society of Human Nutrition and the Department of Clinical Medicine and Surgery, Federico II University. Published by Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest The authors declare no conflict of interest.

Auteurs

Belinda Spoto (B)

CNR-Institute of Clinical Physiology, Reggio Cal, Italy. Electronic address: belindagilda.spoto@cnr.it.

Cristina Politi (C)

CNR-Institute of Clinical Physiology, Reggio Cal, Italy.

Patrizia Pizzini (P)

CNR-Institute of Clinical Physiology, Reggio Cal, Italy.

Rosa M Parlongo (RM)

CNR-Institute of Clinical Physiology, Reggio Cal, Italy.

Alessandra Testa (A)

CNR-Institute of Clinical Physiology, Reggio Cal, Italy.

Marco Mobrici (M)

CNR-Institute of Clinical Physiology, Reggio Cal, Italy.

Giovanni L Tripepi (GL)

CNR-Institute of Clinical Physiology, Reggio Cal, Italy.

Francesca Mallamaci (F)

CNR-Institute of Clinical Physiology, Reggio Cal, Italy; Nephrology, Dialysis and Transplantation Unit (GOM-BMM), Reggio Calabria, Italy.

Carmine Zoccali (C)

Renal Research Institute NY, New York, USA; BIOGEM, Ariano Irpino, Italy.

Classifications MeSH