The Ethanolic Extract of Polygala paniculata L. Blocks Panx1 Channels and Reduces Ischemic Brain Infarct in a Dose- and Sex-Dependent Way.

Polygala paniculata L Neuroprotection Pannexin-1 Photothrombotic ischemia

Journal

Molecular neurobiology
ISSN: 1559-1182
Titre abrégé: Mol Neurobiol
Pays: United States
ID NLM: 8900963

Informations de publication

Date de publication:
13 Sep 2024
Historique:
received: 27 05 2024
accepted: 27 08 2024
medline: 14 9 2024
pubmed: 14 9 2024
entrez: 13 9 2024
Statut: aheadofprint

Résumé

Polygala paniculata L. is a native plant from tropical America. The therapeutic potential of the hydroalcoholic extract of P. paniculata (HEPp) has been scientifically explored due to folk medicine reports on its action against several afflictions. HEPp contains several bioactive molecules with neuroprotective activities, making it a promising candidate for stroke treatment. This study used electrophysiological, biochemical, and in vivo experiments to evaluate the molecular mechanisms underlying HEPp as a neuroprotective therapy for stroke targeting Pannexin-1 (Panx1). Panx1 is a non-selective channel that opens during ischemia and contributes to neuronal death. HEPp was not toxic to cortical neurons and pre-treatment with the extract reduced neuronal death promoted by oxygen and glucose deprivation in a dose-dependent manner. Additionally, HEPp blocked Panx1 currents in a dose-dependent manner and the effect, which was shown to be partially due to rutin. Animals submitted to photothrombosis and post-treated with HEPp had reduced infarct volume, and the effective dose was lower in males (1 mg/kg) than in females (10 mg/kg). On the other hand, in Panx1 KD mice (50% Panx1 levels), the acute treatment reduced the infarct volume only in males. Upon chronic treatment with HEPp, a reduction in Panx1 protein levels was observed. The current study provides reliable evidence of the neuroprotective properties of HEPp in both in vitro and in vivo models of stroke. The underlying mechanism involves, at least in part, the inhibition of Panx1 channel function and possibly downregulation of protein levels, suppressing the secondary events that lead to apoptosis and inflammation.

Identifiants

pubmed: 39271622
doi: 10.1007/s12035-024-04453-5
pii: 10.1007/s12035-024-04453-5
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© 2024. The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature.

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Auteurs

Cristina Martins-Silva (C)

Department of Physiological Sciences, Health Sciences Center, Laboratory of Neurochemistry and Behaviour (LabNeC), Graduate Program in Biochemistry, Federal University of Espirito Santo, Vitoria, ES, 29043910, Brazil. cristina.silva@ufes.br.
Department of Cell Biology and Anatomy, Hotchkiss Brain Institute, Cumming School of Medicine, University of Calgary, 3330 Hospital Dr. NW, Calgary, AB, T2N 1N4, Canada. cristina.silva@ufes.br.

Connor L Anderson (CL)

Department of Cell Biology and Anatomy, Hotchkiss Brain Institute, Cumming School of Medicine, University of Calgary, 3330 Hospital Dr. NW, Calgary, AB, T2N 1N4, Canada.

Andrew K J Boyce (AKJ)

Department of Cell Biology and Anatomy, Hotchkiss Brain Institute, Cumming School of Medicine, University of Calgary, 3330 Hospital Dr. NW, Calgary, AB, T2N 1N4, Canada.

Tassiane E S Andrade (TES)

Laboratory of Neurobiology of Pain and Inflammation, Department of Physiological Sciences, Biological Sciences Center, Federal University of Santa Catarina, Florianópolis, SC, 88040-900, Brazil.
Neuroscience Program, Department of Physiology and Pharmacology, University of Western Ontario, Robarts Research Institute, University of Western Ontario, 100 Perth Dr, London, ON, N6A 5K8, Canada.

Tiago Tizziani (T)

Department of Chemistry, Federal University of Santa Catarina, Florianópolis, SC, 88040-900, Brazil.

Kheytiany H S Lopes (KHS)

Department of Chemistry, Federal University of Santa Catarina, Florianópolis, SC, 88040-900, Brazil.

Gustavo A Micke (GA)

Department of Chemistry, Federal University of Santa Catarina, Florianópolis, SC, 88040-900, Brazil.

Sean P Cregan (SP)

Neuroscience Program, Department of Physiology and Pharmacology, University of Western Ontario, Robarts Research Institute, University of Western Ontario, 100 Perth Dr, London, ON, N6A 5K8, Canada.

Adair Roberto Soares Dos Santos (ARS)

Laboratory of Neurobiology of Pain and Inflammation, Department of Physiological Sciences, Biological Sciences Center, Federal University of Santa Catarina, Florianópolis, SC, 88040-900, Brazil.

Roger J Thompson (RJ)

Department of Cell Biology and Anatomy, Hotchkiss Brain Institute, Cumming School of Medicine, University of Calgary, 3330 Hospital Dr. NW, Calgary, AB, T2N 1N4, Canada.

Classifications MeSH