Fibronectin glomerulopathy in an elderly patient with FN1 gene mutation: a case report and literature review.


Journal

BMC nephrology
ISSN: 1471-2369
Titre abrégé: BMC Nephrol
Pays: England
ID NLM: 100967793

Informations de publication

Date de publication:
16 Sep 2024
Historique:
received: 26 07 2024
accepted: 10 09 2024
medline: 17 9 2024
pubmed: 17 9 2024
entrez: 16 9 2024
Statut: epublish

Résumé

Fibronectin glomerulopathy (FNG) is a rare autosomal dominant glomerulopathy that can lead to nephrotic syndrome. Here we report the case of an elderly patient diagnosed with FNG, exhibiting nephrotic-range proteinuria, with a 2-year follow-up. A 75-year-old Korean female visited the nephrology clinic after experiencing generalized edema for 2 months. Her serum creatinine was 1.36 mg/dL, and urine protein-to-creatinine ratio was 3.99 g/g. Kidney biopsy revealed mesangial and subendothelial dense deposits, and immunohistochemistry for fibronectin showed strong positivity in the glomerulus. The patient's family history included non-specific renal disease in her mother and two siblings. Genetic testing of the fibronectin 1 (FN1) gene showed Y973C mutation. She received conservative treatment, including angiotensin II receptor blockers (ARB). Two years after biopsy, the patient has preserved renal function and reduced proteinuria. We report the case of a 75-year-old patient with nephrotic-range proteinuria, who was diagnosed with FNG, and found to harbor a FN1 gene mutation. In this case, conservative treatment including ARB yielded reduction of proteinuria and preservation of renal function.

Sections du résumé

BACKGROUND BACKGROUND
Fibronectin glomerulopathy (FNG) is a rare autosomal dominant glomerulopathy that can lead to nephrotic syndrome. Here we report the case of an elderly patient diagnosed with FNG, exhibiting nephrotic-range proteinuria, with a 2-year follow-up.
CASE PRESENTATION METHODS
A 75-year-old Korean female visited the nephrology clinic after experiencing generalized edema for 2 months. Her serum creatinine was 1.36 mg/dL, and urine protein-to-creatinine ratio was 3.99 g/g. Kidney biopsy revealed mesangial and subendothelial dense deposits, and immunohistochemistry for fibronectin showed strong positivity in the glomerulus. The patient's family history included non-specific renal disease in her mother and two siblings. Genetic testing of the fibronectin 1 (FN1) gene showed Y973C mutation. She received conservative treatment, including angiotensin II receptor blockers (ARB). Two years after biopsy, the patient has preserved renal function and reduced proteinuria.
CONCLUSION CONCLUSIONS
We report the case of a 75-year-old patient with nephrotic-range proteinuria, who was diagnosed with FNG, and found to harbor a FN1 gene mutation. In this case, conservative treatment including ARB yielded reduction of proteinuria and preservation of renal function.

Identifiants

pubmed: 39285372
doi: 10.1186/s12882-024-03752-2
pii: 10.1186/s12882-024-03752-2
doi:

Substances chimiques

Fibronectins 0
FN1 protein, human 0

Types de publication

Case Reports Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

309

Informations de copyright

© 2024. The Author(s).

Références

Gemperle O, Neuweiler J, Reutter FW, Hildebrandt F, Krapf R. Familial glomerulopathy with giant fibrillar (fibronectin-positive) deposits: 15-year follow-up in a large kindred. Am J Kidney Dis. 1996;28(5):668–75.
doi: 10.1016/S0272-6386(96)90247-4 pubmed: 9158203
Strom EH, Banfi G, Krapf R, Abt AB, Mazzucco G, Monga G, Gloor F, Neuweiler J, Riess R, Stosiek P, et al. Glomerulopathy associated with predominant fibronectin deposits: a newly recognized hereditary disease. Kidney Int. 1995;48(1):163–70.
doi: 10.1038/ki.1995.280 pubmed: 7564073
Lusco MA, Chen YP, Cheng H, Dong HR, Najafian B, Alpers CE, Fogo AB. AJKD Atlas of Renal Pathology: Fibronectin Glomerulopathy. Am J Kidney Dis. 2017;70(5):e21–2.
doi: 10.1053/j.ajkd.2017.09.001 pubmed: 29055354
Zhang T, Zhang W, Zuo K, Cheng Z. Clinicopathologic features and outcomes in Fibronectin Glomerulopathy: a Case Series of 19 patients. Front Med (Lausanne). 2020;7:439.
doi: 10.3389/fmed.2020.00439 pubmed: 32923447
Castelletti F, Donadelli R, Banterla F, Hildebrandt F, Zipfel PF, Bresin E, Otto E, Skerka C, Renieri A, Todeschini M, et al. Mutations in FN1 cause glomerulopathy with fibronectin deposits. Proc Natl Acad Sci U S A. 2008;105(7):2538–43.
doi: 10.1073/pnas.0707730105 pubmed: 18268355 pmcid: 2268172
Takii M, Suehiro T, Shima A, Yotsueda H, Hisano S, Katafuchi R. Fibronectin glomerulopathy complicated with persistent cloaca and congenital esophageal atresia: a case report and literature review. BMC Nephrol. 2017;18(1):288.
doi: 10.1186/s12882-017-0704-5 pubmed: 28877681 pmcid: 5588616
Cheng G, Wang Z, Yuan W, Dou Y, Liu D, Xiao J, Zhao Z. Fibronectin glomerulopathy in a 88 year-old male with acute kidney injury on chronic kidney disease: a case report and a review of the literature. Nefrologia. 2017;37(1):93–6.
doi: 10.1016/j.nefro.2016.09.015 pubmed: 27836185
Ishimoto I, Sohara E, Ito E, Okado T, Rai T, Uchida S. Fibronectin glomerulopathy. Clin Kidney J. 2013;6(5):513–5.
doi: 10.1093/ckj/sft097 pubmed: 26064516 pmcid: 4438411
Yoshino M, Miura N, Ohnishi T, Suzuki K, Kitagawa W, Nishikawa K, Imai H. Clinicopathological analysis of glomerulopathy with fibronectin deposits (GFND): a case of sporadic, elderly-onset GFND with codeposition of IgA, C1q, and fibrinogen. Intern Med. 2013;52(15):1715–20.
doi: 10.2169/internalmedicine.52.0046 pubmed: 23903505
Nadamuni M, Piras R, Mazbar S, Higgins JP, Kambham N. Fibronectin glomerulopathy: an unusual cause of adult-onset nephrotic syndrome. Am J Kidney Dis. 2012;60(5):839–42.
doi: 10.1053/j.ajkd.2012.04.029 pubmed: 22721928
Ohtsubo H, Okada T, Nozu K, Takaoka Y, Shono A, Asanuma K, Zhang L, Nakanishi K, Taniguchi-Ikeda M, Kaito H, et al. Identification of mutations in FN1 leading to glomerulopathy with fibronectin deposits. Pediatr Nephrol. 2016;31(9):1459–67.
doi: 10.1007/s00467-016-3368-7 pubmed: 27056061
Ertoy Baydar D, Kutlugun AA, Bresin E, Piras R. A case of familial glomerulopathy with fibronectin deposits caused by the Y973C mutation in fibronectin. Am J Kidney Dis. 2013;61(3):514–8.
doi: 10.1053/j.ajkd.2012.08.050 pubmed: 23219110
Goldman BI, Panner BJ, Welle SL, Gross MD, Gray DA. Prednisone-induced sustained remission in a patient with familial fibronectin glomerulopathy (GFND). CEN Case Rep. 2021;10(4):510–4.
doi: 10.1007/s13730-021-00595-w pubmed: 33837952 pmcid: 8494821
Hata M, Mori T, Hirose Y, Nishida Y, Mandai S, Ando F, Susa K, Iimori S, Naito S, Sohara E, et al. A case of unexpected diagnosis of fibronectin glomerulopathy with histological features of membranoproliferative glomerulonephritis. BMC Nephrol. 2024;25(1):25.
doi: 10.1186/s12882-024-03456-7 pubmed: 38254040 pmcid: 10802068

Auteurs

Ji-Young Choi (JY)

Division of Nephrology, Department of Internal Medicine, School of Medicine, Kyungpook National University, Daegu, Korea.

Mee-Seon Kim (MS)

Department of Pathology, School of Medicine, Kyungpook National University, Daegu, Korea.

Zehwan Kim (Z)

Department of Laboratory Medicine, Yeungnam University College of Medicine, 170 Hyeonchung-ro, Nam-gu, Daegu, 42415, Republic of Korea. ogkim@hotmail.com.

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