Macular exudate in idiopathic intracranial hypertension affects outer retina and visual acuity.


Journal

BMJ open ophthalmology
ISSN: 2397-3269
Titre abrégé: BMJ Open Ophthalmol
Pays: England
ID NLM: 101714806

Informations de publication

Date de publication:
24 Sep 2024
Historique:
received: 06 06 2024
accepted: 03 09 2024
medline: 25 9 2024
pubmed: 25 9 2024
entrez: 24 9 2024
Statut: epublish

Résumé

Optical coherence tomography (OCT) is suggested as a potential tool for retinal biomarkers in idiopathic intracranial hypertension (IIH). We explored how macular exudate (ME) affects retinal structure in IIH and investigated its relationship with their clinical features. Patients diagnosed with IIH and matched controls were enrolled. ME detection was done on fundus photography; swept-source OCT was used to image and measure the retinal sublayer thicknesses, including the retinal nerve fibre layer, ganglion cell-inner plexiform layer (GCIPL), inner nuclear layer (INL) and outer retinal layer (ORL). IIH patients underwent lumbar puncture where intracranial pressure (ICP) was assessed. 195 eyes from 98 IIH patients (42 eyes had ME) and 224 eyes from 112 controls were included. IIH patients had thicker INL and ORL compared with controls (both p<0.001) while IIH eyes with ME had thicker INL and ORL thicknesses compared with eyes without ME (both p<0.05). In IIH patients, the retinal sublayer thicknesses correlated with their ICP levels, and GCIPL thickness correlated with visual acuity (VA). Furthermore, ME was associated with higher ICP, worse papilledema and lower VA (all p<0.001). ME affects retinal thickness in IIH patients and is associated with more severe clinical features in IIH. OCT may provide biomarkers informative of clinical changes in IIH. Further longitudinal studies are needed to explore the evolution of ME and its relationship to VA and retinal structure.

Sections du résumé

BACKGROUND BACKGROUND
Optical coherence tomography (OCT) is suggested as a potential tool for retinal biomarkers in idiopathic intracranial hypertension (IIH). We explored how macular exudate (ME) affects retinal structure in IIH and investigated its relationship with their clinical features.
METHODS METHODS
Patients diagnosed with IIH and matched controls were enrolled. ME detection was done on fundus photography; swept-source OCT was used to image and measure the retinal sublayer thicknesses, including the retinal nerve fibre layer, ganglion cell-inner plexiform layer (GCIPL), inner nuclear layer (INL) and outer retinal layer (ORL). IIH patients underwent lumbar puncture where intracranial pressure (ICP) was assessed.
RESULTS RESULTS
195 eyes from 98 IIH patients (42 eyes had ME) and 224 eyes from 112 controls were included. IIH patients had thicker INL and ORL compared with controls (both p<0.001) while IIH eyes with ME had thicker INL and ORL thicknesses compared with eyes without ME (both p<0.05). In IIH patients, the retinal sublayer thicknesses correlated with their ICP levels, and GCIPL thickness correlated with visual acuity (VA). Furthermore, ME was associated with higher ICP, worse papilledema and lower VA (all p<0.001).
CONCLUSION CONCLUSIONS
ME affects retinal thickness in IIH patients and is associated with more severe clinical features in IIH. OCT may provide biomarkers informative of clinical changes in IIH. Further longitudinal studies are needed to explore the evolution of ME and its relationship to VA and retinal structure.

Identifiants

pubmed: 39317461
pii: bmjophth-2024-001810
doi: 10.1136/bmjophth-2024-001810
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© Author(s) (or their employer(s)) 2024. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.

Déclaration de conflit d'intérêts

Competing interests: None declared.

Auteurs

Le Cao (L)

Department of Neurology, West China Hospital, Sichuan University, Chengdu, China.

Hang Wang (H)

Department of Neurology, West China Hospital, Sichuan University, Chengdu, China.

William Robert Kwapong (WR)

Department of Neurology, West China Hospital, Sichuan University, Chengdu, China.

Jincheng Wan (J)

Department of Respiratory and Critical Care Medicine, Minda Hospital of Hubei Minzu University, Enshi, China.

Yuying Yan (Y)

Department of Neurology, West China Hospital, Sichuan University, Chengdu, China.

Guina Liu (G)

Department of Ophthalmology, West China Hospital, Sichuan University, Chengdu, China.

Rui Liu (R)

Department of Ophthalmology, West China Hospital, Sichuan University, Chengdu, China.

Fayun Hu (F)

Department of Neurology, West China Hospital, Sichuan University, Chengdu, China dr.bowu@hotmail.com hufayun2006@163.com.

Bo Wu (B)

Department of Neurology, West China Hospital, Sichuan University, Chengdu, China dr.bowu@hotmail.com hufayun2006@163.com.

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