Immunotherapy response and resistance in patients with advanced uveal melanoma: a retrospective cohort study.


Journal

Clinical and experimental medicine
ISSN: 1591-9528
Titre abrégé: Clin Exp Med
Pays: Italy
ID NLM: 100973405

Informations de publication

Date de publication:
01 Oct 2024
Historique:
received: 03 06 2024
accepted: 24 09 2024
medline: 1 10 2024
pubmed: 1 10 2024
entrez: 1 10 2024
Statut: epublish

Résumé

Metastatic uveal melanoma (mUM) is associated with poor prognosis. Ipilimumab/nivolumab has shown antitumor efficacy in phase II studies. Tebentafusp resulted in longer overall survival (OS) compared to investigator`s choice in a phase III study. We sought to describe the radiological response patterns of mUM patients treated with immunotherapy. Patients with mUM treated with ipilimumab/nivolumab and tebentafusp between July 2018 and December 2022, with available radiological assessment per RECISTv1.1 and/or imPERCIST5, were retrospectively identified and included. Progression-free survival (PFS) and OS rates, liver-specific response and pathological assessment in available liver biopsies were evaluated. In the ipilimumab/nivolumab group, median PFS (mPFS) was 2.9 months (95% CI 2.2-28.6) and mOS 28.9 months (95% CI 12.7-NR). Complete (CMR) and partial (PMR) metabolic response per imPERCIST5, and partial response (PR) per RECISTv1.1 were associated with longer PFS and OS by trend, compared to morphologically and metabolically stable or progressive disease. In the tebentafusp group, mPFS was 2.7 months (95% CI 2.2-3) and mOS 18.6 months (95% CI 11.5-NR). PMR and PR were associated with longer PFS by trend. In both treatments, the overall treatment response was associated with the radiological response at the liver site. In available liver tumor biopsies, differences in pathological and radiological responses were noted. ImPERCIST5 and RECIST v1.1 are valuable tools in the radiological response assessment, but both methods display limitations. Accurate biomarkers to stratify patients at risk for disease progression and future translational studies to investigate mechanisms of response and resistance are required.

Identifiants

pubmed: 39352553
doi: 10.1007/s10238-024-01497-8
pii: 10.1007/s10238-024-01497-8
doi:

Substances chimiques

Nivolumab 31YO63LBSN
Ipilimumab 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

234

Informations de copyright

© 2024. The Author(s).

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Auteurs

Alexander Maurer (A)

Department of Nuclear Medicine, University Hospital of Zurich, University of Zurich, Zurich, Switzerland.

Giulio Clerici (G)

Department of Dermatology, University Hospital of Zurich, University of Zurich, Raemistrasse 100, 8091, Zurich, Switzerland.

Jan A Schaab (JA)

Department of Nuclear Medicine, University Hospital of Zurich, University of Zurich, Zurich, Switzerland.

Phil F Cheng (PF)

Department of Oncology, Geneva University Hospital, Geneva, Switzerland.

Daniela Mihic-Probst (D)

Institute for Pathology and Molecular Pathology, University Hospital Zurich, University of Zurich, Zurich, Switzerland.

Cäcilia Mader (C)

Department of Nuclear Medicine, University Hospital of Zurich, University of Zurich, Zurich, Switzerland.

Michael Messerli (M)

Department of Nuclear Medicine, University Hospital of Zurich, University of Zurich, Zurich, Switzerland.

Martin W Huellner (MW)

Department of Nuclear Medicine, University Hospital of Zurich, University of Zurich, Zurich, Switzerland.

Reinhard Dummer (R)

Department of Dermatology, University Hospital of Zurich, University of Zurich, Raemistrasse 100, 8091, Zurich, Switzerland.

Florentia Dimitriou (F)

Department of Dermatology, University Hospital of Zurich, University of Zurich, Raemistrasse 100, 8091, Zurich, Switzerland. florentia.dimitriou@usz.ch.

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