Sacral neuromodulation for Organophosphate-induced delayed neuropathy neurogenic lower urinary tract dysfunction: a case report.


Journal

BMC urology
ISSN: 1471-2490
Titre abrégé: BMC Urol
Pays: England
ID NLM: 100968571

Informations de publication

Date de publication:
04 Oct 2024
Historique:
received: 18 05 2024
accepted: 23 09 2024
medline: 5 10 2024
pubmed: 5 10 2024
entrez: 4 10 2024
Statut: epublish

Résumé

Organophosphate-Induced Delayed Neuropathy (OPIDN) is a rare neurological disorder triggered by exposure to organophosphorus compounds. These compounds exert their neurotoxic effects by impacting the nervous system, leading to systemic manifestations. Urinary system symptoms are infrequently observed in clinical settings. Currently, effective therapeutic interventions for OPIDN-related urinary symptoms are lacking. Sacral nerve modulation therapy, an FDA-approved approach for managing lower urinary tract symptoms, presents as a promising option. Herein, we present a case of OPIDN-induced lower urinary tract obstruction successfully treated with sacral nerve modulation therapy, resulting in substantial symptom relief. A 27-year-old male patient presented with severe bilateral hydronephrosis, attributed to low bladder compliance and accompanied by a fever persisting for 6 days. The patient's medical history revealed accidental ingestion of organophosphate pesticide (Dimethoate) with no concomitant underlying diseases. In consideration of the potential for OPIDN, surgical intervention in the form of sacral neuromodulation (phase I) was undertaken. Subsequent evaluation one month post-surgery revealed notable improvements in both bladder compliance and bilateral hydronephrosis, necessitating sacral neuromodulation (phase II). Presently, following a 5-month follow-up period, the patient remains asymptomatic and in favorable health. This patient achieved long-term relief using sacral neuromodulation.

Sections du résumé

BACKGROUND BACKGROUND
Organophosphate-Induced Delayed Neuropathy (OPIDN) is a rare neurological disorder triggered by exposure to organophosphorus compounds. These compounds exert their neurotoxic effects by impacting the nervous system, leading to systemic manifestations. Urinary system symptoms are infrequently observed in clinical settings. Currently, effective therapeutic interventions for OPIDN-related urinary symptoms are lacking. Sacral nerve modulation therapy, an FDA-approved approach for managing lower urinary tract symptoms, presents as a promising option. Herein, we present a case of OPIDN-induced lower urinary tract obstruction successfully treated with sacral nerve modulation therapy, resulting in substantial symptom relief.
CASE REPORT METHODS
A 27-year-old male patient presented with severe bilateral hydronephrosis, attributed to low bladder compliance and accompanied by a fever persisting for 6 days. The patient's medical history revealed accidental ingestion of organophosphate pesticide (Dimethoate) with no concomitant underlying diseases. In consideration of the potential for OPIDN, surgical intervention in the form of sacral neuromodulation (phase I) was undertaken. Subsequent evaluation one month post-surgery revealed notable improvements in both bladder compliance and bilateral hydronephrosis, necessitating sacral neuromodulation (phase II). Presently, following a 5-month follow-up period, the patient remains asymptomatic and in favorable health.
CONCLUSION CONCLUSIONS
This patient achieved long-term relief using sacral neuromodulation.

Identifiants

pubmed: 39367402
doi: 10.1186/s12894-024-01600-x
pii: 10.1186/s12894-024-01600-x
doi:

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

213

Informations de copyright

© 2024. The Author(s).

Références

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Auteurs

Junjie Han (J)

Department of Urology, The Second Hospital of Jilin University, 4026 Yatai Avenue, Changchun, 130041, Jilin, People's Republic of China.

Dingliang Zhao (D)

Department of Urology II, The First Hospital of Jilin University, Changchun, 130021, Jilin, People's Republic of China.

Shuqiang Feng (S)

Department of Urology, The Second Hospital of Jilin University, 4026 Yatai Avenue, Changchun, 130041, Jilin, People's Republic of China.

Xuesong Yang (X)

Department of Urology, The Second Hospital of Jilin University, 4026 Yatai Avenue, Changchun, 130041, Jilin, People's Republic of China.

Yinchun Wang (Y)

Department of Urology, The Second Hospital of Jilin University, 4026 Yatai Avenue, Changchun, 130041, Jilin, People's Republic of China.

Zhenhua Dong (Z)

Department of Gastric and Colorectal Surgery, The First Hospital of Jilin University Changchun, Jilin, 130021, People's Republic of China.

Zhao Sun (Z)

Department of Urology, The Second Hospital of Jilin University, 4026 Yatai Avenue, Changchun, 130041, Jilin, People's Republic of China.

Ziyuan Deng (Z)

Department of Urology, The Second Hospital of Jilin University, 4026 Yatai Avenue, Changchun, 130041, Jilin, People's Republic of China.

Yi Zhang (Y)

Department of Urology, The Second Hospital of Jilin University, 4026 Yatai Avenue, Changchun, 130041, Jilin, People's Republic of China. yizhang0502@jlu.edu.cn.

Ranwei Li (R)

Department of Urology, The Second Hospital of Jilin University, 4026 Yatai Avenue, Changchun, 130041, Jilin, People's Republic of China. ranwei1968@sina.com.

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