Analysis of angiotensin II type 1 receptor and osteoprotegerin gene polymorphism on the risk of cardiovascular mortality risk and progressivity of chronic kidney disease.


Journal

Minerva medica
ISSN: 1827-1669
Titre abrégé: Minerva Med
Pays: Italy
ID NLM: 0400732

Informations de publication

Date de publication:
Aug 2024
Historique:
medline: 8 10 2024
pubmed: 8 10 2024
entrez: 8 10 2024
Statut: ppublish

Résumé

Chronic kidney disease (CKD) is a significant global public health issue with increased risk of atherosclerotic cardiovascular disease (ASCVD) and cardiovascular mortality. Single nucleotide polymorphisms (SNPs) on angiotensin II type 1 receptor (AT1R) A1166C and osteoprotegerin (OPG) C950T gene have received significant attention as a genetic risk factor for cardiovascular disease and CKD. This was a cross-sectional study involving 75 adults with CKD recruited from Nephrology Outpatient Clinics of Universitas Airlangga Hospital, Surabaya, Indonesia. Demographic data was obtained from interviews and medical records. The "CKD Patch" application was used to asses ASCVD and cardiovascular mortality risk scores. Statistical analysis was performed by using SPSS version 26. We detected four different AT1R gene polymorphisms (A1166C, A1160C, G1170T, and G1181C) and two OPG gene polymorphisms (T950C and G1181C) in Indonesian CKD patients. A1160C and G1181C polymorphisms were novel SNPs, newly discovered in this research. No significant association was found between AT1R SNPs and kidney prognostic markers or ASCVD risk/mortality risk scores. However, for OPG C950T we found that TT genotype had a significantly higher ACR than TC or CC genotype (P=0.032). As for OPG G1181C, we found that GG genotype had a higher serum creatinine and albumin to creatinine ratio compared to GC and CC genotypes (P=0.004 and 0.029, respectively). Genotype GC for OPG G1181C was also shown to be protective for having better kidney markers and lowest cardiovascular mortality risk compared to GG and CC genotypes (P=0.018 and 0.032, respectively). Increased ASCVD risk and mortality risk score was not found on individuals with AT1R gene SNPs. However, for OPG gene polymorphism, C950T and G1181C were associated with kidney progression and cardiovascular mortality.

Sections du résumé

BACKGROUND BACKGROUND
Chronic kidney disease (CKD) is a significant global public health issue with increased risk of atherosclerotic cardiovascular disease (ASCVD) and cardiovascular mortality. Single nucleotide polymorphisms (SNPs) on angiotensin II type 1 receptor (AT1R) A1166C and osteoprotegerin (OPG) C950T gene have received significant attention as a genetic risk factor for cardiovascular disease and CKD.
METHODS METHODS
This was a cross-sectional study involving 75 adults with CKD recruited from Nephrology Outpatient Clinics of Universitas Airlangga Hospital, Surabaya, Indonesia. Demographic data was obtained from interviews and medical records. The "CKD Patch" application was used to asses ASCVD and cardiovascular mortality risk scores. Statistical analysis was performed by using SPSS version 26.
RESULTS RESULTS
We detected four different AT1R gene polymorphisms (A1166C, A1160C, G1170T, and G1181C) and two OPG gene polymorphisms (T950C and G1181C) in Indonesian CKD patients. A1160C and G1181C polymorphisms were novel SNPs, newly discovered in this research. No significant association was found between AT1R SNPs and kidney prognostic markers or ASCVD risk/mortality risk scores. However, for OPG C950T we found that TT genotype had a significantly higher ACR than TC or CC genotype (P=0.032). As for OPG G1181C, we found that GG genotype had a higher serum creatinine and albumin to creatinine ratio compared to GC and CC genotypes (P=0.004 and 0.029, respectively). Genotype GC for OPG G1181C was also shown to be protective for having better kidney markers and lowest cardiovascular mortality risk compared to GG and CC genotypes (P=0.018 and 0.032, respectively).
CONCLUSIONS CONCLUSIONS
Increased ASCVD risk and mortality risk score was not found on individuals with AT1R gene SNPs. However, for OPG gene polymorphism, C950T and G1181C were associated with kidney progression and cardiovascular mortality.

Identifiants

pubmed: 39376100
pii: S0026-4806.24.09435-7
doi: 10.23736/S0026-4806.24.09435-7
doi:

Substances chimiques

Osteoprotegerin 0
Receptor, Angiotensin, Type 1 0
TNFRSF11B protein, human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

447-457

Auteurs

Hendri Susilo (H)

Department of Cardiology and Vascular Medicine, Faculty of Medicine Universitas Airlangga, Universitas Airlangga Hospital, Surabaya, Indonesia.

Budi S Pikir (BS)

Department of Cardiology and Vascular Medicine, Faculty of Medicine Universitas Airlangga, Universitas Airlangga Hospital, Surabaya, Indonesia.

Nathanael J Tjipta (NJ)

Faculty of Medicine, Universitas Airlangga, Surabaya, Indonesia.

Mochamad Y Alsagaff (MY)

Department of Cardiology and Vascular Medicine, Faculty of Medicine Universitas Airlangga, Universitas Airlangga Hospital, Surabaya, Indonesia.

Mochammad Thaha (M)

Department of Internal Medicine, Faculty of Medicine Universitas Airlangga, Universitas Airlangga Hospital, Surabaya, Indonesia.

Satriyo D Suryantoro (SD)

Department of Internal Medicine, Faculty of Medicine Universitas Airlangga, Universitas Airlangga Hospital, Surabaya, Indonesia.

Citrawati Dk Wungu (CD)

Department of Physiology and Medical Biochemistry, Faculty of Medicine, Universitas Airlangga, Surabaya, Indonesia - citrawati.dyah@fk.unair.ac.id.
Institute of Tropical Disease, Universitas Airlangga, Surabaya, Indonesia.

Mochamad Amin (M)

Institute of Tropical Disease, Universitas Airlangga, Surabaya, Indonesia.

Derren D Rampengan (DD)

Faculty of Medicine, Universitas Sam Ratulangi, Manado, Indonesia.

Roy N Ramadhan (RN)

Faculty of Medicine, Universitas Airlangga, Surabaya, Indonesia.

Platon Papageorgiou (P)

Faculty of Medicine, University of Patras, Patras, Greece.

Ryan Gauci (R)

Faculty of Medicine, University of Malta, Msida, Malta.

Rebeka Tóthová (R)

Faculty of Medicine, University of Pavol Jozef Šafárik, Košice, Slovakia.

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Classifications MeSH