Prognostic value of soluble programmed death-1 and soluble programmed death ligand-1 in severe traumatic brain injury patients.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
11 Oct 2024
Historique:
received: 20 05 2024
accepted: 26 09 2024
medline: 12 10 2024
pubmed: 12 10 2024
entrez: 11 10 2024
Statut: epublish

Résumé

Patients with traumatic brain injury (TBI) frequently exhibit concomitant immunosuppression. In this study, we evaluated the predictive values of soluble programmed death-1 (sPD-1) and soluble programmed death ligand-1 (sPD-L1) in patients with severe TBI. Peripheral blood sPD-1 and sPD-L1 levels were measured within 48 h of patient admission. A total of 20 healthy volunteers and 82 patients were enrolled in this study. The levels of sPD-1 and sPD-L1 were upregulated in patients with severe TBI (P < 0.001). They were significantly increased in the post-TBI severe pneumonia group and among non-survivors (P < 0.001). The area under the curves (AUCs) for sPD-1 and sPD-L1 levels to predict severe pneumonia were 0.714 and 0.696, respectively, and the AUCs to predict mortality were 0.758 and 0.735. The levels of sPD-1 and sPD-L1 are correlated with the GCS scores at admission, APACHE II scores, length of MV, and time elapsed to mortality. The levels of sPD-1 and sPD-L1 emerged as independent predictive factors for severe pneumonia and mortality. This study demonstrates that upregulation of sPD-1 and sPD-L1 in severe TBI patients is significantly associated with severe pneumonia and mortality, suggesting their potential as predictive biomarkers for these outcomes.

Identifiants

pubmed: 39394380
doi: 10.1038/s41598-024-74520-3
pii: 10.1038/s41598-024-74520-3
doi:

Substances chimiques

B7-H1 Antigen 0
Programmed Cell Death 1 Receptor 0
Biomarkers 0
CD274 protein, human 0
PDCD1 protein, human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

23791

Subventions

Organisme : the Foundation for the School Health Association of Shandong
ID : SDWS2022186
Organisme : Dongying Natural Science Foundation
ID : 2023ZR036

Informations de copyright

© 2024. The Author(s).

Références

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Auteurs

Lei Liu (L)

Department of Internal Medicine, the Affiliated Hospital of China University of Petroleum (East China), Qingdao, 266580, China.

Pengpeng Lan (P)

Neurological Intensive Care Department, Shengli Oilfield Central Hospital, Dongying City, 257000, Shandong Province, China.

Guiping Wu (G)

Neurological Intensive Care Department, Shengli Oilfield Central Hospital, Dongying City, 257000, Shandong Province, China.

Xiaojie Zhu (X)

Department of Respiratory Medicine, Dongying District People's Hospital, Dongying City, 257000, Shandong Province, China.

Hongfeng Shi (H)

Neurological Intensive Care Department, Shengli Oilfield Central Hospital, Dongying City, 257000, Shandong Province, China.

Yan Li (Y)

Neurological Intensive Care Department, Shengli Oilfield Central Hospital, Dongying City, 257000, Shandong Province, China.

Ruili Li (R)

Neurological Intensive Care Department, Shengli Oilfield Central Hospital, Dongying City, 257000, Shandong Province, China.

Ling Zhao (L)

Department of Nursing, Shengli Oilfield Central Hospital, Dongying City, 257000, Shandong Province, China.

Juan Xu (J)

Neurological Intensive Care Department, Shengli Oilfield Central Hospital, Dongying City, 257000, Shandong Province, China. xujuanemail@163.com.

Min Xu (M)

Department of Internal Medicine, the Affiliated Hospital of China University of Petroleum (East China), Qingdao, 266580, China. xumin0224@163.com.
Academy of Medical Engineering and Translational Medicine, Tianjin University, Tianjin, 300072, China. xumin0224@163.com.

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