ML-enhanced peroxisome capacity enables compartmentalization of multienzyme pathway.


Journal

Nature chemical biology
ISSN: 1552-4469
Titre abrégé: Nat Chem Biol
Pays: United States
ID NLM: 101231976

Informations de publication

Date de publication:
14 Oct 2024
Historique:
received: 23 01 2024
accepted: 20 09 2024
medline: 15 10 2024
pubmed: 15 10 2024
entrez: 14 10 2024
Statut: aheadofprint

Résumé

Repurposing an organelle for specialized metabolism provides an avenue for fermentable, unicellular organisms such as Saccharomyces cerevisiae to mimic compartmentalization of metabolic pathways within different plant tissues. Peroxisomes are attractive organelles for repurposing as they are not required for yeast viability when grown on glucose and can efficiently compartmentalize heterologous enzymes to enable physical separation of cytosolic native metabolism and peroxisomal engineered metabolism. However, when not required, peroxisomes are repressed, leading to low functional capacities for heterologous proteins. Here we engineer peroxisomes with enhanced functional capacities, with the goal of compartmentalizing up to eight metabolic enzymes to enhance titers. We implement a machine learning pipeline that allows the identification of factors to overexpress, culminating in a 137% increase in peroxisome functional capacity compared to a wild-type strain. Improved pathway compartmentalization enables an 80% increase in the biosynthesis titers of the monoterpene geraniol, up to 9.5 g L

Identifiants

pubmed: 39402374
doi: 10.1038/s41589-024-01759-2
pii: 10.1038/s41589-024-01759-2
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : National Science Foundation (NSF)
ID : DBI-1548297
Organisme : National Science Foundation (NSF)
ID : DBI-1548297
Organisme : National Science Foundation (NSF)
ID : DBI-1548297
Organisme : National Science Foundation (NSF)
ID : DBI-1548297
Organisme : National Science Foundation (NSF)
ID : DBI-1548297
Organisme : National Science Foundation (NSF)
ID : DBI-1548297
Organisme : National Science Foundation (NSF)
ID : DBI-1548297

Informations de copyright

© 2024. The Author(s), under exclusive licence to Springer Nature America, Inc.

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Auteurs

Jordan J Baker (JJ)

Department of Bioengineering, University of California, Berkeley, CA, USA.
UC Berkeley and UCSF Joint Graduate Program in Bioengineering, University of California, Berkeley, CA, USA.
NSF Center for Cellular Construction, University of California, San Francisco, CA, USA.

Jie Shi (J)

NSF Center for Cellular Construction, University of California, San Francisco, CA, USA.
Department of Functional Genomics and Cellular Engineering, IBM Almaden Research Center, San Jose, CA, USA.

Shangying Wang (S)

NSF Center for Cellular Construction, University of California, San Francisco, CA, USA.
Department of Functional Genomics and Cellular Engineering, IBM Almaden Research Center, San Jose, CA, USA.
Bay Area Institute of Science, Altos Labs, Redwood City, CA, USA.

Elena M Mujica (EM)

Department of Bioengineering, University of California, Berkeley, CA, USA.

Simone Bianco (S)

NSF Center for Cellular Construction, University of California, San Francisco, CA, USA.
Department of Functional Genomics and Cellular Engineering, IBM Almaden Research Center, San Jose, CA, USA.
Bay Area Institute of Science, Altos Labs, Redwood City, CA, USA.

Sara Capponi (S)

NSF Center for Cellular Construction, University of California, San Francisco, CA, USA. sara.capponi@ibm.com.
Department of Functional Genomics and Cellular Engineering, IBM Almaden Research Center, San Jose, CA, USA. sara.capponi@ibm.com.

John E Dueber (JE)

Department of Bioengineering, University of California, Berkeley, CA, USA. jdueber@berkeley.edu.
NSF Center for Cellular Construction, University of California, San Francisco, CA, USA. jdueber@berkeley.edu.
Biological Systems and Engineering Division, Lawrence Berkeley National Laboratory, Berkeley, CA, USA. jdueber@berkeley.edu.

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