Parvimonas micra forms a distinct bacterial network with oral pathobionts in colorectal cancer patients.


Journal

Journal of translational medicine
ISSN: 1479-5876
Titre abrégé: J Transl Med
Pays: England
ID NLM: 101190741

Informations de publication

Date de publication:
17 Oct 2024
Historique:
received: 02 07 2024
accepted: 01 10 2024
medline: 18 10 2024
pubmed: 18 10 2024
entrez: 17 10 2024
Statut: epublish

Résumé

Mounting evidence suggests a significant role of the gut microbiota in the development and progression of colorectal cancer (CRC). In particular, an over-representation of oral pathogens has been linked to CRC. The aim of this study was to further investigate the faecal microbial landscape of CRC patients, with a focus on the oral pathogens Parvimonas micra and Fusobacterium nucleatum. In this study, 16S rRNA sequencing was conducted using faecal samples from CRC patients (n = 275) and controls without pathological findings (n = 95). We discovered a significant difference in microbial composition depending on tumour location and microsatellite instability (MSI) status, with P. micra, F. nucleatum, and Peptostreptococcus stomatis found to be more abundant in patients with MSI tumours. Moreover, P. micra and F. nucleatum were associated with a cluster of CRC-related bacteria including Bacteroides fragilis as well as with other oral pathogens such as P. stomatis and various Porphyromonas species. This cluster was distinctly different in the control group, suggesting its potential linkage with CRC. Our results suggest a similar distribution of several CRC-associated bacteria within CRC patients, underscoring the importance of considering the concomitant presence of bacterial species in studies investigating the mechanisms of CRC development and progression.

Sections du résumé

BACKGROUND BACKGROUND
Mounting evidence suggests a significant role of the gut microbiota in the development and progression of colorectal cancer (CRC). In particular, an over-representation of oral pathogens has been linked to CRC. The aim of this study was to further investigate the faecal microbial landscape of CRC patients, with a focus on the oral pathogens Parvimonas micra and Fusobacterium nucleatum.
METHODS METHODS
In this study, 16S rRNA sequencing was conducted using faecal samples from CRC patients (n = 275) and controls without pathological findings (n = 95).
RESULTS RESULTS
We discovered a significant difference in microbial composition depending on tumour location and microsatellite instability (MSI) status, with P. micra, F. nucleatum, and Peptostreptococcus stomatis found to be more abundant in patients with MSI tumours. Moreover, P. micra and F. nucleatum were associated with a cluster of CRC-related bacteria including Bacteroides fragilis as well as with other oral pathogens such as P. stomatis and various Porphyromonas species. This cluster was distinctly different in the control group, suggesting its potential linkage with CRC.
CONCLUSIONS CONCLUSIONS
Our results suggest a similar distribution of several CRC-associated bacteria within CRC patients, underscoring the importance of considering the concomitant presence of bacterial species in studies investigating the mechanisms of CRC development and progression.

Identifiants

pubmed: 39420333
doi: 10.1186/s12967-024-05720-8
pii: 10.1186/s12967-024-05720-8
doi:

Substances chimiques

RNA, Ribosomal, 16S 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

947

Informations de copyright

© 2024. The Author(s).

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Auteurs

Thyra Löwenmark (T)

Department of Medical Biosciences, Pathology, Umeå University, Umeå, Sweden.

Linda Köhn (L)

Department of Medical Biosciences, Pathology, Umeå University, Umeå, Sweden.

Therese Kellgren (T)

Department of Mathematics and Mathematical Statistics, Umeå University, Umeå, Sweden.

William Rosenbaum (W)

Department of Medical Biosciences, Pathology, Umeå University, Umeå, Sweden.

Vicky Bronnec (V)

Department of Medical Biosciences, Pathology, Umeå University, Umeå, Sweden.

Anna Löfgren-Burström (A)

Department of Medical Biosciences, Pathology, Umeå University, Umeå, Sweden.

Carl Zingmark (C)

Department of Medical Biosciences, Pathology, Umeå University, Umeå, Sweden.

Pär Larsson (P)

Department of Medical Biosciences, Pathology, Umeå University, Umeå, Sweden.

Michael Dahlberg (M)

Department of Diagnostics and Intervention, Umeå University, Umeå, Sweden.

Bjoern O Schroeder (BO)

Department of Molecular Biology, Umeå University, Umeå, Sweden.

Sun Nyunt Wai (SN)

Department of Molecular Biology, Umeå University, Umeå, Sweden.

Ingrid Ljuslinder (I)

Department of Diagnostics and Intervention, Umeå University, Umeå, Sweden.

Sofia Edin (S)

Department of Medical Biosciences, Pathology, Umeå University, Umeå, Sweden.

Richard Palmqvist (R)

Department of Medical Biosciences, Pathology, Umeå University, Umeå, Sweden. richard.palmqvist@umu.se.

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