Comparative pathogenesis of Ethiopia/Habru/2014 Lineage-IV peste des petits ruminants virus in goats and cattle.


Journal

BMC veterinary research
ISSN: 1746-6148
Titre abrégé: BMC Vet Res
Pays: England
ID NLM: 101249759

Informations de publication

Date de publication:
17 Oct 2024
Historique:
received: 03 07 2024
accepted: 01 10 2024
medline: 18 10 2024
pubmed: 18 10 2024
entrez: 17 10 2024
Statut: epublish

Résumé

Peste des Petits Ruminants (PPR) is a highly contagious viral disease primarily affecting goats and sheep, with clinical manifestations ranging from peracute disease to subclinical infection, particularly in atypical hosts such as cattle. The role of atypical hosts such as cattle to the spread of PPR remains controversial, with conflicting reports in the literature. Despite its worldwide significance, considerable knowledge gaps exist regarding the pathogenesis and clinical progression in both primary and atypical hosts. This study aimed to elucidate the tissue tropism, pathogenesis, virus shedding, clinical progression, and pathology associated with experimental PPR virus infection in indigenous goats and cattle. To this end, 32 animals-16 goats and 16 cattle-were intranasally inoculated with the Ethiopia/Habru/2014 Lineage-IV strain of the PPR virus followed by detailed clinical evaluations and systematic sampling at pre-established intervals to assess serological conversion, viral shedding, and the pathogenesis of the infection across both species. The results show that goats exhibited typical clinical signs 4 days post-inoculation, with seroconversion by day 6 and early detection of viral RNA in swabs and tissues by day 3 and virus isolation starting day 4. In contrast, cattle exhibited minimal clinical signs, with seroconversion occurring at day 8 with viral RNA detected in tissue samples at day 4 and virus isolation starting day 6 in tissues and in a single nasal swab at day 8. Clinical scores and tissue positivity rates significantly differed between goats and cattle (P = 0.007 and P < 0.001, respectively). While goats exhibited expected gross and histopathological lesions, cattle showed only nonspecific lesions. Together, our findings highlight the importance of comparative pathology studies for better understanding virus dynamics and transmission pathways that may help inform more effective PPR control programs. Future research should explore the pathogenesis of different PPRV lineages in cattle, assessing variations in disease progression and potential for epidemiological impact.

Sections du résumé

BACKGROUND BACKGROUND
Peste des Petits Ruminants (PPR) is a highly contagious viral disease primarily affecting goats and sheep, with clinical manifestations ranging from peracute disease to subclinical infection, particularly in atypical hosts such as cattle. The role of atypical hosts such as cattle to the spread of PPR remains controversial, with conflicting reports in the literature. Despite its worldwide significance, considerable knowledge gaps exist regarding the pathogenesis and clinical progression in both primary and atypical hosts. This study aimed to elucidate the tissue tropism, pathogenesis, virus shedding, clinical progression, and pathology associated with experimental PPR virus infection in indigenous goats and cattle. To this end, 32 animals-16 goats and 16 cattle-were intranasally inoculated with the Ethiopia/Habru/2014 Lineage-IV strain of the PPR virus followed by detailed clinical evaluations and systematic sampling at pre-established intervals to assess serological conversion, viral shedding, and the pathogenesis of the infection across both species.
RESULTS RESULTS
The results show that goats exhibited typical clinical signs 4 days post-inoculation, with seroconversion by day 6 and early detection of viral RNA in swabs and tissues by day 3 and virus isolation starting day 4. In contrast, cattle exhibited minimal clinical signs, with seroconversion occurring at day 8 with viral RNA detected in tissue samples at day 4 and virus isolation starting day 6 in tissues and in a single nasal swab at day 8. Clinical scores and tissue positivity rates significantly differed between goats and cattle (P = 0.007 and P < 0.001, respectively). While goats exhibited expected gross and histopathological lesions, cattle showed only nonspecific lesions.
CONCLUSIONS CONCLUSIONS
Together, our findings highlight the importance of comparative pathology studies for better understanding virus dynamics and transmission pathways that may help inform more effective PPR control programs. Future research should explore the pathogenesis of different PPRV lineages in cattle, assessing variations in disease progression and potential for epidemiological impact.

Identifiants

pubmed: 39420341
doi: 10.1186/s12917-024-04313-3
pii: 10.1186/s12917-024-04313-3
doi:

Substances chimiques

RNA, Viral 0

Types de publication

Journal Article Comparative Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

473

Informations de copyright

© 2024. The Author(s).

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Auteurs

Fasil Aklilu (F)

Animal Health Institute, Sebeta, Ethiopia. fasil.aklilu@yahoo.com.
Aklilu Lemma Institute of Pathobiology, Addis Ababa University, Addis Ababa, Ethiopia. fasil.aklilu@yahoo.com.

Hagos Ashenafi (H)

Aklilu Lemma Institute of Pathobiology, Addis Ababa University, Addis Ababa, Ethiopia.

Tesfu Kassa (T)

Aklilu Lemma Institute of Pathobiology, Addis Ababa University, Addis Ababa, Ethiopia.

Hassen Chaka (H)

Food and Agricultural Organization (NSAH-CJW), Addis Ababa, Ethiopia.

Demeke Sibhatu (D)

Animal Health Institute, Sebeta, Ethiopia.

Dereje Shegu (D)

Animal Health Institute, Sebeta, Ethiopia.

Abde Aliy Mohammed (AA)

Animal Health Institute, Sebeta, Ethiopia.

Redeat Belaineh (R)

Animal Health Institute, Sebeta, Ethiopia.

Menbere Kidane (M)

Animal Health Institute, Sebeta, Ethiopia.

Hagos Asgedom (H)

Animal Health Institute, Sebeta, Ethiopia.

Tesfaye Chibssa (T)

Animal Health Institute, Sebeta, Ethiopia.

Getnet Mekonnen (G)

Animal Health Institute, Sebeta, Ethiopia.

Asegedetch Sirak (A)

Animal Health Institute, Sebeta, Ethiopia.

Solomon Gebredufe (S)

Animal Health Institute, Sebeta, Ethiopia.

Claudia Schulz (C)

Department of Biological Sciences and Pathology, University of Veterinary Medicine Vienna, Vienna, Austria.

Catherine M Herzog (CM)

Huck Institutes of the Life Sciences, The Pennsylvania State University, University Park, PA, USA. catherine.herzog@gmail.com.

Vivek Kapur (V)

Huck Institutes of the Life Sciences, The Pennsylvania State University, University Park, PA, USA. vkapur@psu.edu.
Department of Animal Science, The Pennsylvania State University, University Park, PA, USA. vkapur@psu.edu.

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