Upper respiratory microbial communities of healthy populations are shaped by niche and age.


Journal

Microbiome
ISSN: 2049-2618
Titre abrégé: Microbiome
Pays: England
ID NLM: 101615147

Informations de publication

Date de publication:
18 Oct 2024
Historique:
received: 11 04 2024
accepted: 23 09 2024
medline: 19 10 2024
pubmed: 19 10 2024
entrez: 18 10 2024
Statut: epublish

Résumé

Alterations in upper respiratory microbiomes have been implicated in shaping host health trajectories, including by limiting mucosal pathogen colonization. However, limited comparative studies of respiratory microbiome development and functioning across age groups have been performed. Herein, we perform shotgun metagenomic sequencing paired with pathogen inhibition assays to elucidate differences in nasal and oral microbiome composition and intermicrobial interactions across healthy 24-month-old infant (n = 229) and adult (n = 100) populations. We find that beta diversity of nasal and oral microbiomes varies with age, with nasal microbiomes showing greater population-level variation compared to oral microbiomes. Infant microbiome alpha diversity was significantly lower across nasal samples and higher in oral samples, relative to adults. Accordingly, we demonstrate significant differences in genus- and species-level composition of microbiomes between sites and age groups. Antimicrobial resistome patterns likewise varied across body sites, with oral microbiomes showing higher resistance gene abundance compared to nasal microbiomes. Biosynthetic gene clusters encoding specialized metabolite production were found in higher abundance across infant oral microbiomes, relative to adults. Investigation of pathogen inhibition revealed greater inhibition of gram-negative and gram-positive bacteria by oral commensals, while nasal isolates had higher antifungal activity. In summary, we identify significant differences in the microbial communities inhabiting nasal and oral cavities of healthy infants relative to adults. These findings inform our understanding of the interactions impacting respiratory microbiome composition and functions related to colonization resistance, with important implications for host health across the lifespan. Video Abstract.

Sections du résumé

BACKGROUND BACKGROUND
Alterations in upper respiratory microbiomes have been implicated in shaping host health trajectories, including by limiting mucosal pathogen colonization. However, limited comparative studies of respiratory microbiome development and functioning across age groups have been performed. Herein, we perform shotgun metagenomic sequencing paired with pathogen inhibition assays to elucidate differences in nasal and oral microbiome composition and intermicrobial interactions across healthy 24-month-old infant (n = 229) and adult (n = 100) populations.
RESULTS RESULTS
We find that beta diversity of nasal and oral microbiomes varies with age, with nasal microbiomes showing greater population-level variation compared to oral microbiomes. Infant microbiome alpha diversity was significantly lower across nasal samples and higher in oral samples, relative to adults. Accordingly, we demonstrate significant differences in genus- and species-level composition of microbiomes between sites and age groups. Antimicrobial resistome patterns likewise varied across body sites, with oral microbiomes showing higher resistance gene abundance compared to nasal microbiomes. Biosynthetic gene clusters encoding specialized metabolite production were found in higher abundance across infant oral microbiomes, relative to adults. Investigation of pathogen inhibition revealed greater inhibition of gram-negative and gram-positive bacteria by oral commensals, while nasal isolates had higher antifungal activity.
CONCLUSIONS CONCLUSIONS
In summary, we identify significant differences in the microbial communities inhabiting nasal and oral cavities of healthy infants relative to adults. These findings inform our understanding of the interactions impacting respiratory microbiome composition and functions related to colonization resistance, with important implications for host health across the lifespan. Video Abstract.

Identifiants

pubmed: 39425237
doi: 10.1186/s40168-024-01940-8
pii: 10.1186/s40168-024-01940-8
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

206

Subventions

Organisme : NIH HHS
ID : F30AI169759
Pays : United States
Organisme : NIH HHS
ID : T32AI055397
Pays : United States
Organisme : NIH HHS
ID : U19AI104317
Pays : United States
Organisme : NIH HHS
ID : U19AI104317
Pays : United States
Organisme : NIH HHS
ID : U19AI142720
Pays : United States
Organisme : NIH HHS
ID : U19AI142720
Pays : United States

Informations de copyright

© 2024. The Author(s).

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Auteurs

Susan Zelasko (S)

Department of Bacteriology, University of Wisconsin-Madison, Madison, WI, USA. szelasko@wisc.edu.
Microbiology Doctoral Training Program, University of Wisconsin-Madison, Madison, WI, USA. szelasko@wisc.edu.

Mary Hannah Swaney (MH)

Microbiology Doctoral Training Program, University of Wisconsin-Madison, Madison, WI, USA.
Department of Medical Microbiology and Immunology, University of Wisconsin-Madison, Madison, WI, USA.

Shelby Sandstrom (S)

Department of Medical Microbiology and Immunology, University of Wisconsin-Madison, Madison, WI, USA.

Timothy C Davenport (TC)

Department of Bacteriology, University of Wisconsin-Madison, Madison, WI, USA.

Christine M Seroogy (CM)

Department of Pediatrics, University of Wisconsin School of Medicine and Public Health, Madison, WI, USA.

James E Gern (JE)

Department of Pediatrics, University of Wisconsin School of Medicine and Public Health, Madison, WI, USA.
Department of Medicine, University of Wisconsin School of Medicine and Public Health, Madison, WI, USA.

Lindsay R Kalan (LR)

Department of Medical Microbiology and Immunology, University of Wisconsin-Madison, Madison, WI, USA.
Department of Medicine, University of Wisconsin School of Medicine and Public Health, Madison, WI, USA.
Department of Biochemistry and Biomedical Sciences, M.G. DeGroote Institute for Infectious Disease Research, David Braley Centre for Antibiotic Discovery, McMaster University, Hamilton, ON, Canada.

Cameron R Currie (CR)

Department of Bacteriology, University of Wisconsin-Madison, Madison, WI, USA. ccurrie@mcmaster.ca.
Department of Biochemistry and Biomedical Sciences, M.G. DeGroote Institute for Infectious Disease Research, David Braley Centre for Antibiotic Discovery, McMaster University, Hamilton, ON, Canada. ccurrie@mcmaster.ca.

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