Left atrial volume assessed by echocardiography identifies patients with high risk of adverse outcome after acute myocardial infarction.

Acute myocardial infarction Echocardiography Indexed left atrial volume New treatment strategies Primary outcomes

Journal

Echo research and practice
ISSN: 2055-0464
Titre abrégé: Echo Res Pract
Pays: England
ID NLM: 101664713

Informations de publication

Date de publication:
21 Oct 2024
Historique:
received: 13 05 2024
accepted: 19 08 2024
medline: 21 10 2024
pubmed: 21 10 2024
entrez: 20 10 2024
Statut: epublish

Résumé

The left atrial (LA) volume has been demonstrated to be an important predictor of adverse outcome in patients with various cardiac conditions, including acute myocardial infarction (AMI). However, new treatment strategies in patients with AMI have led to better patient outcomes. We hypothesised that increased LA size could still predict mortality in patients with AMI despite improved treatment strategies. We included patients with AMI in a prospective multicenter cohort study and the study patients were enrolled from 2014 to 2022. We recorded echocardiographic and clinical data during their index hospitalisation. Indexed LA volume (LAVi) was assessed in all patients and was used as a continuous variable in the univariate and multivariate Cox regression analysis. The study took place over a period of five years and median follow-up time was 3.8 years (range 3.1 to 5.0 years). The primary study outcomes were all-cause mortality and major adverse cardiac events (MACE). MACE was defined as hospital readmission due to myocardial infarction, cardiac arrest, stroke, heart failure, or onset of new atrial fibrillation. We included 487 patients (69 ± 12 years old, 26% female) with AMI. During the follow-up period all-cause mortality was 50 (10.3%) and patients who reached the primary outcomes were 153 (31.4%). The deceased patients had higher LAVi compared to survivors (40.0 ± 12.9 mL/m Our study demonstrated that LA dilatation is an independent echocardiographic predictor of mortality and MACE in patients with AMI despite improved treatment strategies. This finding highlights the potential of using LAVi as a marker for prognostication in these patients.

Sections du résumé

BACKGROUND BACKGROUND
The left atrial (LA) volume has been demonstrated to be an important predictor of adverse outcome in patients with various cardiac conditions, including acute myocardial infarction (AMI). However, new treatment strategies in patients with AMI have led to better patient outcomes. We hypothesised that increased LA size could still predict mortality in patients with AMI despite improved treatment strategies.
METHODS METHODS
We included patients with AMI in a prospective multicenter cohort study and the study patients were enrolled from 2014 to 2022. We recorded echocardiographic and clinical data during their index hospitalisation. Indexed LA volume (LAVi) was assessed in all patients and was used as a continuous variable in the univariate and multivariate Cox regression analysis. The study took place over a period of five years and median follow-up time was 3.8 years (range 3.1 to 5.0 years). The primary study outcomes were all-cause mortality and major adverse cardiac events (MACE). MACE was defined as hospital readmission due to myocardial infarction, cardiac arrest, stroke, heart failure, or onset of new atrial fibrillation.
RESULTS RESULTS
We included 487 patients (69 ± 12 years old, 26% female) with AMI. During the follow-up period all-cause mortality was 50 (10.3%) and patients who reached the primary outcomes were 153 (31.4%). The deceased patients had higher LAVi compared to survivors (40.0 ± 12.9 mL/m
CONCLUSIONS CONCLUSIONS
Our study demonstrated that LA dilatation is an independent echocardiographic predictor of mortality and MACE in patients with AMI despite improved treatment strategies. This finding highlights the potential of using LAVi as a marker for prognostication in these patients.

Identifiants

pubmed: 39428485
doi: 10.1186/s44156-024-00060-1
pii: 10.1186/s44156-024-00060-1
doi:

Types de publication

Journal Article

Langues

eng

Pagination

24

Informations de copyright

© 2024. The Author(s).

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Auteurs

Jorun Tangen (J)

ProCardio Center for Innovation, Department of Cardiology, Oslo University Hospital, Rikshospitalet Sognsvannsveien 20, Nydalen, PO Box 4950, Oslo, NO-0424, Norway.
Institute for Clinical Medicine, University of Oslo, Sognsvannsveien 9, Oslo, 0373, Norway.

Thuy Mi Nguyen (TM)

ProCardio Center for Innovation, Department of Cardiology, Oslo University Hospital, Rikshospitalet Sognsvannsveien 20, Nydalen, PO Box 4950, Oslo, NO-0424, Norway.
Institute for Clinical Medicine, University of Oslo, Sognsvannsveien 9, Oslo, 0373, Norway.

Daniela Melichova (D)

ProCardio Center for Innovation, Department of Cardiology, Oslo University Hospital, Rikshospitalet Sognsvannsveien 20, Nydalen, PO Box 4950, Oslo, NO-0424, Norway.
Department of Cardiology, Hospital of Southern Norway, Sykehusveien 1, Arendal, 4838, Norway.

Lars Gunnar Klaeboe (LG)

ProCardio Center for Innovation, Department of Cardiology, Oslo University Hospital, Rikshospitalet Sognsvannsveien 20, Nydalen, PO Box 4950, Oslo, NO-0424, Norway.
Institute for Clinical Medicine, University of Oslo, Sognsvannsveien 9, Oslo, 0373, Norway.

Marianne Forsa (M)

ProCardio Center for Innovation, Department of Cardiology, Oslo University Hospital, Rikshospitalet Sognsvannsveien 20, Nydalen, PO Box 4950, Oslo, NO-0424, Norway.
Institute for Clinical Medicine, University of Oslo, Sognsvannsveien 9, Oslo, 0373, Norway.

Kristoffer Andresen (K)

ProCardio Center for Innovation, Department of Cardiology, Oslo University Hospital, Rikshospitalet Sognsvannsveien 20, Nydalen, PO Box 4950, Oslo, NO-0424, Norway.
Institute for Clinical Medicine, University of Oslo, Sognsvannsveien 9, Oslo, 0373, Norway.

Adrien Al Wazzan (AA)

ProCardio Center for Innovation, Department of Cardiology, Oslo University Hospital, Rikshospitalet Sognsvannsveien 20, Nydalen, PO Box 4950, Oslo, NO-0424, Norway.

Oyvind Lie (O)

ProCardio Center for Innovation, Department of Cardiology, Oslo University Hospital, Rikshospitalet Sognsvannsveien 20, Nydalen, PO Box 4950, Oslo, NO-0424, Norway.
Institute for Clinical Medicine, University of Oslo, Sognsvannsveien 9, Oslo, 0373, Norway.

Fatih Kizilaslan (F)

Oslo Centre for Biostatistics and Epidemiology, Department of Biostatistics, University of Oslo, Oslo, Norway.

Kristina Haugaa (K)

ProCardio Center for Innovation, Department of Cardiology, Oslo University Hospital, Rikshospitalet Sognsvannsveien 20, Nydalen, PO Box 4950, Oslo, NO-0424, Norway.
Institute for Clinical Medicine, University of Oslo, Sognsvannsveien 9, Oslo, 0373, Norway.

Helge Skulstad (H)

ProCardio Center for Innovation, Department of Cardiology, Oslo University Hospital, Rikshospitalet Sognsvannsveien 20, Nydalen, PO Box 4950, Oslo, NO-0424, Norway.
Institute for Clinical Medicine, University of Oslo, Sognsvannsveien 9, Oslo, 0373, Norway.

Harald Brunvand (H)

ProCardio Center for Innovation, Department of Cardiology, Oslo University Hospital, Rikshospitalet Sognsvannsveien 20, Nydalen, PO Box 4950, Oslo, NO-0424, Norway.
Department of Cardiology, Hospital of Southern Norway, Sykehusveien 1, Arendal, 4838, Norway.

Thor Edvardsen (T)

ProCardio Center for Innovation, Department of Cardiology, Oslo University Hospital, Rikshospitalet Sognsvannsveien 20, Nydalen, PO Box 4950, Oslo, NO-0424, Norway. thor.edvardsen@medisin.uio.no.
Institute for Clinical Medicine, University of Oslo, Sognsvannsveien 9, Oslo, 0373, Norway. thor.edvardsen@medisin.uio.no.

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