The role of the oral microbiota in the causal effect of adjunctive antibiotics on clinical outcomes in stage III-IV periodontitis patients.


Journal

Microbiome
ISSN: 2049-2618
Titre abrégé: Microbiome
Pays: England
ID NLM: 101615147

Informations de publication

Date de publication:
26 Oct 2024
Historique:
received: 17 04 2024
accepted: 07 10 2024
medline: 27 10 2024
pubmed: 27 10 2024
entrez: 27 10 2024
Statut: epublish

Résumé

Periodontitis, a prevalent chronic inflammatory disease, offers insights into the broader landscape of chronic inflammatory conditions. The progression and treatment outcomes of periodontitis are closely related to the oral microbiota's composition. Adjunctive systemic Amoxicillin 500 mg and Metronidazole 400 mg, often prescribed thrice daily for 7 days to enhance periodontal therapy's efficacy, have lasting effects on the oral microbiome. However, the precise mechanism through which the oral microbiome influences clinical outcomes in periodontitis patients remains debated. This investigation explores the pivotal role of the oral microbiome's composition in mediating the outcomes of adjunctive systemic antibiotic treatment. Subgingival plaque samples from 10 periodontally healthy and 163 periodontitis patients from a randomized clinical trial on periodontal therapy were analyzed. Patients received either adjunctive amoxicillin/metronidazole or a placebo after mechanical periodontal treatment. Microbial samples were collected at various intervals up to 26 months post-therapy. Using topic models, we identified microbial communities associated with normobiotic and dysbiotic states, validated with 86 external and 40 internal samples. Logistic regression models evaluated the association between these microbial communities and clinical periodontitis parameters. A Directed Acyclic Graph (DAG) determined the mediating role of oral microbiota in the causal path of antibiotic treatment effects on clinical outcomes. We identified clear distinctions between dysbiotic and normobiotic microbial communities, differentiating healthy from periodontitis subjects. Dysbiotic states consistently associated with below median %Pocket Probing Depth ≥ 5 mm (OR = 1.26, 95% CI [1.14-1.42]) and %Bleeding on Probing (OR = 1.09, 95% CI [1.00-1.18]). Factors like microbial response to treatment, smoking, and age were predictors of clinical attachment loss progression, whereas sex and antibiotic treatment were not. Further, we showed that the oral microbial treatment response plays a crucial role in the causal effect of antibiotic treatment on clinical treatment outcomes. The shift towards a normobiotic subgingival microbiome, primarily induced by adjunctive antibiotics, underscores the potential for microbiome-targeted interventions to enhance therapeutic efficacy in chronic inflammatory conditions. This study reaffirms the importance of understanding the oral microbiome's role in periodontal health and paves the way for future research exploring personalized treatment strategies based on individual microbiome profiles. Video Abstract.

Sections du résumé

BACKGROUND BACKGROUND
Periodontitis, a prevalent chronic inflammatory disease, offers insights into the broader landscape of chronic inflammatory conditions. The progression and treatment outcomes of periodontitis are closely related to the oral microbiota's composition. Adjunctive systemic Amoxicillin 500 mg and Metronidazole 400 mg, often prescribed thrice daily for 7 days to enhance periodontal therapy's efficacy, have lasting effects on the oral microbiome. However, the precise mechanism through which the oral microbiome influences clinical outcomes in periodontitis patients remains debated. This investigation explores the pivotal role of the oral microbiome's composition in mediating the outcomes of adjunctive systemic antibiotic treatment.
METHODS METHODS
Subgingival plaque samples from 10 periodontally healthy and 163 periodontitis patients from a randomized clinical trial on periodontal therapy were analyzed. Patients received either adjunctive amoxicillin/metronidazole or a placebo after mechanical periodontal treatment. Microbial samples were collected at various intervals up to 26 months post-therapy. Using topic models, we identified microbial communities associated with normobiotic and dysbiotic states, validated with 86 external and 40 internal samples. Logistic regression models evaluated the association between these microbial communities and clinical periodontitis parameters. A Directed Acyclic Graph (DAG) determined the mediating role of oral microbiota in the causal path of antibiotic treatment effects on clinical outcomes.
RESULTS RESULTS
We identified clear distinctions between dysbiotic and normobiotic microbial communities, differentiating healthy from periodontitis subjects. Dysbiotic states consistently associated with below median %Pocket Probing Depth ≥ 5 mm (OR = 1.26, 95% CI [1.14-1.42]) and %Bleeding on Probing (OR = 1.09, 95% CI [1.00-1.18]). Factors like microbial response to treatment, smoking, and age were predictors of clinical attachment loss progression, whereas sex and antibiotic treatment were not. Further, we showed that the oral microbial treatment response plays a crucial role in the causal effect of antibiotic treatment on clinical treatment outcomes.
CONCLUSIONS CONCLUSIONS
The shift towards a normobiotic subgingival microbiome, primarily induced by adjunctive antibiotics, underscores the potential for microbiome-targeted interventions to enhance therapeutic efficacy in chronic inflammatory conditions. This study reaffirms the importance of understanding the oral microbiome's role in periodontal health and paves the way for future research exploring personalized treatment strategies based on individual microbiome profiles. Video Abstract.

Identifiants

pubmed: 39462428
doi: 10.1186/s40168-024-01945-3
pii: 10.1186/s40168-024-01945-3
doi:

Substances chimiques

Amoxicillin 804826J2HU
Metronidazole 140QMO216E
Anti-Bacterial Agents 0

Types de publication

Journal Article Randomized Controlled Trial

Langues

eng

Sous-ensembles de citation

IM

Pagination

220

Informations de copyright

© 2024. The Author(s).

Références

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Auteurs

Sven Kleine Bardenhorst (S)

Department of Periodontology and Operative Dentistry, Muenster University Hospital, Albert-Schweitzer-Campus 1, Building W30, Münster, 48149, Germany.
Institute of Epidemiology and Social Medicine, University of Münster, Münster, Germany.

Daniel Hagenfeld (D)

Department of Periodontology and Operative Dentistry, Muenster University Hospital, Albert-Schweitzer-Campus 1, Building W30, Münster, 48149, Germany. daniel.hagenfeld@ukmuenster.de.

Johannes Matern (J)

Department of Periodontology and Operative Dentistry, Muenster University Hospital, Albert-Schweitzer-Campus 1, Building W30, Münster, 48149, Germany.

Karola Prior (K)

Department of Periodontology and Operative Dentistry, Muenster University Hospital, Albert-Schweitzer-Campus 1, Building W30, Münster, 48149, Germany.

Inga Harks (I)

Department of Periodontology and Operative Dentistry, Muenster University Hospital, Albert-Schweitzer-Campus 1, Building W30, Münster, 48149, Germany.

Peter Eickholz (P)

Department of Periodontology, Center for Dentistry and Oral Medicine, Goethe University Frankfurt, Frankfurt, Germany.

Katrin Lorenz (K)

Department of Periodontology, TU Dresden, Dresden, Germany.

Ti-Sun Kim (TS)

Department of Conservative Dentistry, Section of Periodontology, Clinic for Oral, Dental and Maxillofacial Diseases, Heidelberg University Hospital, Heidelberg, Germany.

Thomas Kocher (T)

Department of Restorative Dentistry, Periodontology, Endodontology, and Preventive and Pediatric Dentistry, University Medicine Greifswald, Greifswald, Germany.

Jörg Meyle (J)

Department of Periodontology, University of Giessen, Giessen, Germany.

Doğan Kaner (D)

Departments of Periodontology and Synoptic Dentistry, Charite-Universitätsmedizin Berlin, Berlin, Germany.
Department of Periodontology, Dental School, Faculty of Health, University of Witten/Herdecke, Witten, Germany.

Yvonne Jockel-Schneider (Y)

Department of Periodontology, University Hospital Würzburg, Würzburg, Germany.

Dag Harmsen (D)

Department of Periodontology and Operative Dentistry, Muenster University Hospital, Albert-Schweitzer-Campus 1, Building W30, Münster, 48149, Germany.

Benjamin Ehmke (B)

Department of Periodontology and Operative Dentistry, Muenster University Hospital, Albert-Schweitzer-Campus 1, Building W30, Münster, 48149, Germany.

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