Mitoepigenetics pathways and natural compounds: a dual approach to combatting hepatocellular carcinoma.


Journal

Medical oncology (Northwood, London, England)
ISSN: 1559-131X
Titre abrégé: Med Oncol
Pays: United States
ID NLM: 9435512

Informations de publication

Date de publication:
27 Oct 2024
Historique:
received: 27 08 2024
accepted: 07 10 2024
medline: 28 10 2024
pubmed: 28 10 2024
entrez: 28 10 2024
Statut: epublish

Résumé

Hepatocellular carcinoma (HCC) is a leading liver cancer that significantly impacts global life expectancy and remains challenging to treat due to often late diagnoses. Despite advances in treatment, the prognosis is still poor, especially in advanced stages. Studies have pointed out that investigations into the molecular mechanisms underlying HCC, including mitochondrial dysfunction and epigenetic regulators, are potentially important targets for diagnosis and therapy. Mitoepigenetics, or the epigenetic modifications of mitochondrial DNA, have drawn wide attention for their role in HCC progression. Besides, molecular biomarkers such as mitochondrial DNA alterations and non-coding RNAs showed early diagnosis and prognosis potential. Additionally, natural compounds like alkaloids, resveratrol, curcumin, and flavonoids show promise in HCC show promise in modulating mitochondrial and epigenetic pathways involved in cancer-related processes. This review discusses how mitochondrial dysfunction and epigenetic modifications, especially mitoepigenetics, influence HCC and delves into the potential of natural products as new adjuvant treatments against HCC.

Identifiants

pubmed: 39465473
doi: 10.1007/s12032-024-02538-8
pii: 10.1007/s12032-024-02538-8
doi:

Substances chimiques

Biological Products 0
DNA, Mitochondrial 0

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

302

Informations de copyright

© 2024. The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature.

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Auteurs

Abdulrahman Hatawsh (A)

Biotechnology School, Nile University, 26th of July Corridor, Sheikh Zayed City, Giza, 12588, Egypt.

Roya Hadi Al-Haddad (RH)

Research and Technology Center of Environment, Water and Renewable Energy, Scientific Research Commission, Baghdad, Iraq.

Ukamaka Gladys Okafor (UG)

Euclid University, Bangui, Central African Republic.

Lamis M Diab (LM)

Department of Medical Biochemistry, Medical Research Institute, Alexandria University, Alexandria, Egypt.

Nino Dekanoidze (N)

David Tvildiani Medical University, Tbilisi, Georgia.

Adeniyi Ayinde Abdulwahab (AA)

Faculty of Pharmaceutical Sciences, Bayero University, Kano, Nigeria.

Osama A Mohammed (OA)

Department of Pharmacology, College of Medicine, University of Bisha, 61922, Bisha, Saudi Arabia.

Ahmed S Doghish (AS)

Department of Biochemistry, Faculty of Pharmacy, Badr University in Cairo (BUC), Badr City, Cairo, 11829, Egypt. ahmed_doghish@azhar.edu.eg.
Biochemistry and Molecular Biology Department, Faculty of Pharmacy (Boys), Al-Azhar University, Nasr City, Cairo, 11231, Egypt. ahmed_doghish@azhar.edu.eg.

Rewan Moussa (R)

Faculty of Medicine, Helwan University, Helwan, Cairo, 11795, Egypt.

Hanan Elimam (H)

Department of Biochemistry, Faculty of Pharmacy, University of Sadat City, Sādāt, 32897, Egypt. Hanan.Elimam@fop.usc.edu.eg.

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