Triple immunostaining demonstrates the possible existence of segregated-nucleus-containing atypical monocytes in human primary myelofibrosis bone marrow: a case report.

Bone marrow Case report Immunohistochemistry Monocyte Primary myelofibrosis Segregated-nucleus-containing atypical monocyte

Journal

Journal of medical case reports
ISSN: 1752-1947
Titre abrégé: J Med Case Rep
Pays: England
ID NLM: 101293382

Informations de publication

Date de publication:
30 Oct 2024
Historique:
received: 08 03 2024
accepted: 03 09 2024
medline: 30 10 2024
pubmed: 30 10 2024
entrez: 30 10 2024
Statut: epublish

Résumé

Segregated-nucleus-containing atypical monocytes have recently been identified in mice. Segregated-nucleus-containing atypical monocytes are thought to originate from the bone marrow and induce fibrosis in the drug-injured lung. The Lyc6c A 74-year-old Japanese male visited our hospital for clinical follow-up after total prostatectomy for prostatic cancer. Anemia, thrombocytosis, and elevated lactate dehydrogenase were suddenly observed in a periodic examination. CALR mutation type 2 (p.K385fs*47) was observed. The histological features of the patient's bone marrow were consistent with fibrotic primary myelofibrosis. We immunohistochemically studied the bone marrow in an attempt to identify a human counterpart to murine segregated-nucleus-containing atypical monocytes. We detected a few CD16 There is a possibility that human segregated-nucleus-containing atypical monocytes exist in the bone marrow of primary myelofibrosis patients and might be related to marrow fibrosis.

Sections du résumé

BACKGROUND BACKGROUND
Segregated-nucleus-containing atypical monocytes have recently been identified in mice. Segregated-nucleus-containing atypical monocytes are thought to originate from the bone marrow and induce fibrosis in the drug-injured lung. The Lyc6c
CASE PRESENTATION METHODS
A 74-year-old Japanese male visited our hospital for clinical follow-up after total prostatectomy for prostatic cancer. Anemia, thrombocytosis, and elevated lactate dehydrogenase were suddenly observed in a periodic examination. CALR mutation type 2 (p.K385fs*47) was observed. The histological features of the patient's bone marrow were consistent with fibrotic primary myelofibrosis. We immunohistochemically studied the bone marrow in an attempt to identify a human counterpart to murine segregated-nucleus-containing atypical monocytes. We detected a few CD16
CONCLUSION CONCLUSIONS
There is a possibility that human segregated-nucleus-containing atypical monocytes exist in the bone marrow of primary myelofibrosis patients and might be related to marrow fibrosis.

Identifiants

pubmed: 39472997
doi: 10.1186/s13256-024-04844-1
pii: 10.1186/s13256-024-04844-1
doi:

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

510

Informations de copyright

© 2024. The Author(s).

Références

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Auteurs

Shunsuke Homma (S)

Department of Medical Intern Center, Tokyo Women's Medical University Adachi Medical Center, Tokyo, Japan.
Department of Hematology, Nippon Medical School, Tokyo, Japan.

Toshie Ogasawara (T)

Department of Medicine, Tokyo Women's Medical University Adachi Medical Center, Tokyo, Japan.

Michie Suga (M)

Department of Diagnostic Pathology, Tokyo Women's Medical University Adachi Medical Center, 4-33-1, Kohoku, Adachi-ku, 123-8558, Tokyo, Japan.

Yoshiyasu Nakamura (Y)

Molecular Pathology and Genetics Division, Kanagawa Cancer Center Research Institute, 2-3-2 Nakao, Asahi-Ku, Yokohama, 241-8515, Japan.

Katsuya Takenaka (K)

Molecular Pathology and Genetics Division, Kanagawa Cancer Center Research Institute, 2-3-2 Nakao, Asahi-Ku, Yokohama, 241-8515, Japan.
TR Company, Shin Nippon Biomedical Laboratories, Ltd., Kagoshima, Japan.

Shoko Marshall (S)

Department of Medicine, Tokyo Women's Medical University Adachi Medical Center, Tokyo, Japan.

Kiyotaka Kawauchi (K)

Department of Medicine, Tokyo Women's Medical University Adachi Medical Center, Tokyo, Japan.

Naoki Mori (N)

Department of Medicine, Tokyo Women's Medical University Adachi Medical Center, Tokyo, Japan.
Department of Hematology, International University of Health and Welfare Narita Hospital, Narita, Japan.

Hajime Kuroda (H)

Department of Diagnostic Pathology, Tokyo Women's Medical University Adachi Medical Center, 4-33-1, Kohoku, Adachi-ku, 123-8558, Tokyo, Japan.

Naoya Nakamura (N)

Department of Pathology, Tokai University School of Medicine, Isehara, Japan.

Yohei Miyagi (Y)

Molecular Pathology and Genetics Division, Kanagawa Cancer Center Research Institute, 2-3-2 Nakao, Asahi-Ku, Yokohama, 241-8515, Japan.

Atsuko Masunaga (A)

Department of Diagnostic Pathology, Tokyo Women's Medical University Adachi Medical Center, 4-33-1, Kohoku, Adachi-ku, 123-8558, Tokyo, Japan. nonkoamesho0324@yahoo.co.jp.
Molecular Pathology and Genetics Division, Kanagawa Cancer Center Research Institute, 2-3-2 Nakao, Asahi-Ku, Yokohama, 241-8515, Japan. nonkoamesho0324@yahoo.co.jp.

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Classifications MeSH