DNA methylation defines regional identity of human intestinal epithelial organoids and undergoes dynamic changes during development.


Journal

Gut
ISSN: 1468-3288
Titre abrégé: Gut
Pays: England
ID NLM: 2985108R

Informations de publication

Date de publication:
01 2019
Historique:
received: 10 07 2017
revised: 06 10 2017
accepted: 15 10 2017
pubmed: 17 11 2017
medline: 1 1 2019
entrez: 17 11 2017
Statut: ppublish

Résumé

Human intestinal epithelial organoids (IEOs) are increasingly being recognised as a highly promising translational research tool. However, our understanding of their epigenetic molecular characteristics and behaviour in culture remains limited. We performed genome-wide DNA methylation and transcriptomic profiling of human IEOs derived from paediatric/adult and fetal small and large bowel as well as matching purified human gut epithelium. Furthermore, organoids were subjected to in vitro differentiation and genome editing using CRISPR/Cas9 technology. We discovered stable epigenetic signatures which define regional differences in gut epithelial function, including induction of segment-specific genes during cellular differentiation. Established DNA methylation profiles were independent of cellular environment since organoids retained their regional DNA methylation over prolonged culture periods. In contrast to paediatric and adult organoids, fetal gut-derived organoids showed distinct dynamic changes of DNA methylation and gene expression in culture, indicative of an in vitro maturation. By applying CRISPR/Cas9 genome editing to fetal organoids, we demonstrate that this process is partly regulated by TET1, an enzyme involved in the DNA demethylation process. Lastly, generating IEOs from a child diagnosed with gastric heterotopia revealed persistent and distinct disease-associated DNA methylation differences, highlighting the use of organoids as disease-specific research models. Our study demonstrates striking similarities of epigenetic signatures in mucosa-derived IEOs with matching primary epithelium. Moreover, these results suggest that intestinal stem cell-intrinsic DNA methylation patterns establish and maintain regional gut specification and are involved in early epithelial development and disease.

Identifiants

pubmed: 29141958
pii: gutjnl-2017-314817
doi: 10.1136/gutjnl-2017-314817
pmc: PMC6839835
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

49-61

Subventions

Organisme : Wellcome Trust
Pays : United Kingdom
Organisme : Medical Research Council
ID : MC_PC_12009
Pays : United Kingdom
Organisme : Wellcome Trust
ID : 101241/Z/13/Z
Pays : United Kingdom

Commentaires et corrections

Type : CommentIn

Informations de copyright

© Article author(s) (or their employer(s) unless otherwise stated in the text of the article) 2019. All rights reserved. No commercial use is permitted unless otherwise expressly granted.

Déclaration de conflit d'intérêts

Competing interests: None declared.

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Auteurs

Judith Kraiczy (J)

Department of Paediatrics, University of Cambridge, Addenbrooke's Hospital, Cambridge, UK.

Komal M Nayak (KM)

Department of Paediatrics, University of Cambridge, Addenbrooke's Hospital, Cambridge, UK.

Kate J Howell (KJ)

Department of Paediatrics, University of Cambridge, Addenbrooke's Hospital, Cambridge, UK.
European Molecular Biology Laboratory, European Bioinformatics Institute, Wellcome Trust Genome Campus, Cambridge, UK.

Alexander Ross (A)

Department of Paediatrics, University of Cambridge, Addenbrooke's Hospital, Cambridge, UK.
Anne McLaren Laboratory & Department of Surgery, Wellcome Trust-Medical Research Council Stem Cell Institute, University of Cambridge, Cambridge, UK.

Jessica Forbester (J)

Wellcome Trust Sanger Institute, Wellcome Trust Genome Campus, Cambridge, UK.

Camilla Salvestrini (C)

Department of Paediatric Gastroenterology, Hepatology and Nutrition, Cambridge University Hospitals, Addenbrooke's, Cambridge, UK.

Roxana Mustata (R)

Wellcome Trust-Medical Research Council Stem Cell Institute, University of Cambridge, Cambridge, UK.
Department of Genetics, University of Cambridge, Cambridge, UK.

Sally Perkins (S)

Department of Paediatric Gastroenterology, Hepatology and Nutrition, Cambridge University Hospitals, Addenbrooke's, Cambridge, UK.

Amanda Andersson-Rolf (A)

Wellcome Trust-Medical Research Council Stem Cell Institute, University of Cambridge, Cambridge, UK.
Department of Genetics, University of Cambridge, Cambridge, UK.

Esther Leenen (E)

Department of Paediatrics, University of Cambridge, Addenbrooke's Hospital, Cambridge, UK.

Anke Liebert (A)

Department of Paediatrics, University of Cambridge, Addenbrooke's Hospital, Cambridge, UK.

Ludovic Vallier (L)

Anne McLaren Laboratory & Department of Surgery, Wellcome Trust-Medical Research Council Stem Cell Institute, University of Cambridge, Cambridge, UK.
Wellcome Trust Sanger Institute, Wellcome Trust Genome Campus, Cambridge, UK.

Philip C Rosenstiel (PC)

Institute of Clinical Molecular Biology, Christian-Albrechts-University of Kiel, Kiel, Germany.

Oliver Stegle (O)

European Molecular Biology Laboratory, European Bioinformatics Institute, Wellcome Trust Genome Campus, Cambridge, UK.

Gordon Dougan (G)

Wellcome Trust Sanger Institute, Wellcome Trust Genome Campus, Cambridge, UK.

Robert Heuschkel (R)

Department of Paediatric Gastroenterology, Hepatology and Nutrition, Cambridge University Hospitals, Addenbrooke's, Cambridge, UK.

Bon-Kyoung Koo (BK)

Wellcome Trust-Medical Research Council Stem Cell Institute, University of Cambridge, Cambridge, UK.
Department of Genetics, University of Cambridge, Cambridge, UK.

Matthias Zilbauer (M)

Department of Paediatrics, University of Cambridge, Addenbrooke's Hospital, Cambridge, UK.
Department of Paediatric Gastroenterology, Hepatology and Nutrition, Cambridge University Hospitals, Addenbrooke's, Cambridge, UK.
Wellcome Trust-Medical Research Council Stem Cell Institute, University of Cambridge, Cambridge, UK.

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