Whole-genome sequencing in the investigation of recurrent invasive group A streptococcus outbreaks in a maternity unit.


Journal

The Journal of hospital infection
ISSN: 1532-2939
Titre abrégé: J Hosp Infect
Pays: England
ID NLM: 8007166

Informations de publication

Date de publication:
Mar 2019
Historique:
received: 05 06 2017
accepted: 16 03 2018
pubmed: 27 3 2018
medline: 19 3 2019
entrez: 27 3 2018
Statut: ppublish

Résumé

The clinical manifestations of group A streptococcus (GAS) (Streptococcus pyogenes) are diverse, ranging from asymptomatic colonization to devastating invasive disease. Maternity-related clusters of invasive GAS (iGAS) infection are complex to investigate and control, especially if recurrent. To investigate three episodes of emm 75 GAS/iGAS infection in maternity patients at one hospital site over a four-year period (two with monophyletic ancestry). The episodes are described, together with whole-genome sequence (WGS) isolate analyses. Single nucleotide polymorphism differences were compared with contemporaneous emm 75 genomes. Over the four-year study period, seven mothers had emm 75 GAS/iGAS and one mother had emm 3 iGAS (in year 4) (subsequently discounted as linked). Three (clinical/screening samples) of the seven babies of emm-75-positive mothers and three screened healthcare workers were positive for emm 75 GAS. WGS similarity suggested a shared ancestral lineage and a common source transmission, but directionality of transmission cannot be inferred. However, the findings indicate that persistence of a particular clone in a given setting may be long term. Occupational health procedures were enhanced, staff were screened, and antibiotic therapy was provided to GAS-positive staff and patients. The definitive source of infection could not be identified, although staff-patient transmission was the most likely route. The pattern of clonal GAS transmission over the four-year study period suggests that long-term persistence of GAS may have occurred.

Sections du résumé

BACKGROUND BACKGROUND
The clinical manifestations of group A streptococcus (GAS) (Streptococcus pyogenes) are diverse, ranging from asymptomatic colonization to devastating invasive disease. Maternity-related clusters of invasive GAS (iGAS) infection are complex to investigate and control, especially if recurrent.
AIM OBJECTIVE
To investigate three episodes of emm 75 GAS/iGAS infection in maternity patients at one hospital site over a four-year period (two with monophyletic ancestry).
METHODS METHODS
The episodes are described, together with whole-genome sequence (WGS) isolate analyses. Single nucleotide polymorphism differences were compared with contemporaneous emm 75 genomes.
FINDINGS RESULTS
Over the four-year study period, seven mothers had emm 75 GAS/iGAS and one mother had emm 3 iGAS (in year 4) (subsequently discounted as linked). Three (clinical/screening samples) of the seven babies of emm-75-positive mothers and three screened healthcare workers were positive for emm 75 GAS. WGS similarity suggested a shared ancestral lineage and a common source transmission, but directionality of transmission cannot be inferred. However, the findings indicate that persistence of a particular clone in a given setting may be long term.
CONCLUSIONS CONCLUSIONS
Occupational health procedures were enhanced, staff were screened, and antibiotic therapy was provided to GAS-positive staff and patients. The definitive source of infection could not be identified, although staff-patient transmission was the most likely route. The pattern of clonal GAS transmission over the four-year study period suggests that long-term persistence of GAS may have occurred.

Identifiants

pubmed: 29577990
pii: S0195-6701(18)30169-5
doi: 10.1016/j.jhin.2018.03.018
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

320-326

Informations de copyright

Copyright © 2018. Published by Elsevier Ltd.

Auteurs

H Dickinson (H)

Eastern Field Epidemiology Unit, National Infection Service, Cambridge, UK.

M Reacher (M)

Eastern Field Epidemiology Unit, National Infection Service, Cambridge, UK.

B Nazareth (B)

Anglia Health Protection Team, Thetford, UK.

H Eagle (H)

North West Anglia NHS Foundation Trust, Executive and Infection Control Teams, Peterborough, UK.

D Fowler (D)

North West Anglia NHS Foundation Trust, Executive and Infection Control Teams, Peterborough, UK.

A Underwood (A)

PHE Microbiology Reference Services, National Infection Service, London, UK.

M Chand (M)

PHE Microbiology Reference Services, National Infection Service, London, UK; Guy's & St Thomas Hospitals NHS Foundation Trust, London, UK; NIHR Health Protection Research Unit in Respiratory Infections at Imperial College London, London, UK.

V Chalker (V)

PHE Microbiology Reference Services, National Infection Service, London, UK.

J Coelho (J)

PHE Microbiology Reference Services, National Infection Service, London, UK.

R Daniel (R)

PHE Microbiology Reference Services, National Infection Service, London, UK.

G Kapatai (G)

Eastern Field Epidemiology Unit, National Infection Service, Cambridge, UK.

A Al-Shabib (A)

PHE Microbiology Reference Services, National Infection Service, London, UK.

R Puleston (R)

Eastern Field Epidemiology Unit, National Infection Service, Cambridge, UK; University of Nottingham, School of Medicine, Division of Epidemiology and Public Health, Nottingham, UK. Electronic address: richard.puleston@phe.gov.uk.

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