Epigenetic Control of the Notch and Eph Signaling Pathways by the Prion Protein: Implications for Prion Diseases.
Cellular prion protein
Eph
HDAC
Notch
Prion infection
Journal
Molecular neurobiology
ISSN: 1559-1182
Titre abrégé: Mol Neurobiol
Pays: United States
ID NLM: 8900963
Informations de publication
Date de publication:
Mar 2019
Mar 2019
Historique:
received:
01
12
2017
accepted:
26
06
2018
pubmed:
13
7
2018
medline:
18
7
2019
entrez:
13
7
2018
Statut:
ppublish
Résumé
Among the ever-growing number of self-replicating proteins involved in neurodegenerative diseases, the prion protein PrP remains the most infamous for its central role in transmissible spongiform encephalopathies (TSEs). In these diseases, pathogenic prions propagate through a seeding mechanism, where normal PrP
Identifiants
pubmed: 29998397
doi: 10.1007/s12035-018-1193-7
pii: 10.1007/s12035-018-1193-7
doi:
Substances chimiques
Prion Proteins
0
Receptors, Notch
0
Receptors, Eph Family
EC 2.7.10.1
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
2159-2173Subventions
Organisme : Fondation pour la Recherche Médicale
ID : DEQ20150331689
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