Efficacy and Pharmacokinetics of Glecaprevir and Pibrentasvir With Concurrent Use of Acid-Reducing Agents in Patients With Chronic HCV Infection.


Journal

Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association
ISSN: 1542-7714
Titre abrégé: Clin Gastroenterol Hepatol
Pays: United States
ID NLM: 101160775

Informations de publication

Date de publication:
02 2019
Historique:
received: 04 04 2018
revised: 21 06 2018
accepted: 04 07 2018
pubmed: 18 7 2018
medline: 1 4 2020
entrez: 18 7 2018
Statut: ppublish

Résumé

Proton pump inhibitors (PPIs) are commonly prescribed to treat acid-related disorders. Some direct-acting antiviral regimens for chronic hepatitis C virus (HCV) infection have reduced efficacy in patients taking concomitant acid-reducing agents, including PPIs, due to interactions between drugs. We analyzed data from 9 multicenter, phase 2 and 3 trials to determine the efficacy and pharmacokinetics of an HCV therapeutic regimen comprising glecaprevir and pibrentasvir (glecaprevir/pibrentasvir) in patients taking concomitant acid-reducing agents. We analyzed data from 2369 patients infected with HCV genotypes 1-6 and compensated liver disease treated with an all-oral regimen of glecaprevir/pibrentasvir for 8-16 weeks. We compared efficacy and pharmacokinetics among patients receiving at least 1 dose of an acid-reducing agent (a PPI, an H2 blocker, or antacid). High-dose PPI was defined as daily dose greater than 20 mg omeprazole dose equivalent. The objectives were to evaluate rate of sustained virologic response 12 weeks post-treatment (SVR12) and to assess steady-state glecaprevir and pibrentasvir exposures in patients on acid-reducing agents. Of the 401 patients (17%) who reported use of acid-reducing agents, 263 took PPIs (11%; 109 patients took a high-dose PPI and 154 patients took a low-dose PPI). Rates of SVR12 were 97.0% among patients who used acid-reducing agents and 97.5% among those not using acid-reducing agents (P = .6). An SVR12 was achieved in 96.3% taking a high-dose PPI and 97.4% taking a low-dose PPI, with no virologic failures in those receiving a high-dose PPI (P = .7). Glecaprevir, but not pibrentasvir, bioavailability was affected; its exposure decreased by 41% in patients taking a high-dose PPI. In an analysis of data from 9 clinical trials, we observed a high rate of SVR12 (approximately 97%) among patients treated with glecaprevir/pibrentasvir for HCV infection-even among patients taking concomitant ARA or high-dose PPI. This was despite decreased glecaprevir exposures in patients when on high-dose PPIs. ClinicalTrials.gov numbers, NCT02243280 (SURVEYOR-I), NCT02243293 (SURVEYOR-II), NCT02604017 (ENDURANCE-1), NCT02640482 (ENDURANCE-2), NCT02640157 (ENDURANCE-3), NCT02636595 (ENDURANCE-4), NCT02642432 (EXPEDITION-1), NCT02651194 (EXPEDITION-4), NCT02446717 (MAGELLAN-I).

Sections du résumé

BACKGROUND & AIMS
Proton pump inhibitors (PPIs) are commonly prescribed to treat acid-related disorders. Some direct-acting antiviral regimens for chronic hepatitis C virus (HCV) infection have reduced efficacy in patients taking concomitant acid-reducing agents, including PPIs, due to interactions between drugs. We analyzed data from 9 multicenter, phase 2 and 3 trials to determine the efficacy and pharmacokinetics of an HCV therapeutic regimen comprising glecaprevir and pibrentasvir (glecaprevir/pibrentasvir) in patients taking concomitant acid-reducing agents.
METHODS
We analyzed data from 2369 patients infected with HCV genotypes 1-6 and compensated liver disease treated with an all-oral regimen of glecaprevir/pibrentasvir for 8-16 weeks. We compared efficacy and pharmacokinetics among patients receiving at least 1 dose of an acid-reducing agent (a PPI, an H2 blocker, or antacid). High-dose PPI was defined as daily dose greater than 20 mg omeprazole dose equivalent. The objectives were to evaluate rate of sustained virologic response 12 weeks post-treatment (SVR12) and to assess steady-state glecaprevir and pibrentasvir exposures in patients on acid-reducing agents.
RESULTS
Of the 401 patients (17%) who reported use of acid-reducing agents, 263 took PPIs (11%; 109 patients took a high-dose PPI and 154 patients took a low-dose PPI). Rates of SVR12 were 97.0% among patients who used acid-reducing agents and 97.5% among those not using acid-reducing agents (P = .6). An SVR12 was achieved in 96.3% taking a high-dose PPI and 97.4% taking a low-dose PPI, with no virologic failures in those receiving a high-dose PPI (P = .7). Glecaprevir, but not pibrentasvir, bioavailability was affected; its exposure decreased by 41% in patients taking a high-dose PPI.
CONCLUSIONS
In an analysis of data from 9 clinical trials, we observed a high rate of SVR12 (approximately 97%) among patients treated with glecaprevir/pibrentasvir for HCV infection-even among patients taking concomitant ARA or high-dose PPI. This was despite decreased glecaprevir exposures in patients when on high-dose PPIs. ClinicalTrials.gov numbers, NCT02243280 (SURVEYOR-I), NCT02243293 (SURVEYOR-II), NCT02604017 (ENDURANCE-1), NCT02640482 (ENDURANCE-2), NCT02640157 (ENDURANCE-3), NCT02636595 (ENDURANCE-4), NCT02642432 (EXPEDITION-1), NCT02651194 (EXPEDITION-4), NCT02446717 (MAGELLAN-I).

Identifiants

pubmed: 30012435
pii: S1542-3565(18)30702-X
doi: 10.1016/j.cgh.2018.07.003
pii:
doi:

Substances chimiques

Antiviral Agents 0
Benzimidazoles 0
Drug Combinations 0
Proton Pump Inhibitors 0
Pyrrolidines 0
Quinoxalines 0
Sulfonamides 0
glecaprevir and pibrentasvir 0

Banques de données

ClinicalTrials.gov
['NCT02243280', 'NCT02243293', 'NCT02604017', 'NCT02640482', 'NCT02640157', 'NCT02636595', 'NCT02642432', 'NCT02651194', 'NCT02446717']

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

527-535.e6

Informations de copyright

Copyright © 2019 AGA Institute. Published by Elsevier Inc. All rights reserved.

Auteurs

Steven Flamm (S)

Northwestern Feinberg School of Medicine, Chicago, Illinois. Electronic address: s-flamm@northwestern.edu.

K Rajender Reddy (KR)

University of Pennsylvania, Philadelphia, Pennsylvania.

Neddie Zadeikis (N)

AbbVie Inc, North Chicago, Illinois.

Tarek Hassanein (T)

Southern California GI and Liver Centers and Southern California Research Center, Coronado, California.

Bruce R Bacon (BR)

Saint Louis University Liver Center, St. Louis, Missouri.

Andreas Maieron (A)

Elisabethinen Hospital, Linz, Austria; University Clinics St. Pölten, Karl Landsteiner University, St. Pölten, Austria.

Stefan Zeuzem (S)

J.W. Goethe University, Frankfurt, Germany.

Marc Bourliere (M)

Hôpital Saint Joseph, Marseilles, France.

Jose L Calleja (JL)

Hospital Universitario Puerta de Hierro, Universidad Autonoma de Madrid, Madrid, Spain.

Matthew P Kosloski (MP)

AbbVie Inc, North Chicago, Illinois.

Rajneet K Oberoi (RK)

AbbVie Inc, North Chicago, Illinois.

Chih-Wei Lin (CW)

AbbVie Inc, North Chicago, Illinois.

Yao Yu (Y)

AbbVie Inc, North Chicago, Illinois.

Sandra Lovell (S)

AbbVie Inc, North Chicago, Illinois.

Dimitri Semizarov (D)

AbbVie Inc, North Chicago, Illinois.

Federico J Mensa (FJ)

AbbVie Inc, North Chicago, Illinois.

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Classifications MeSH