Methylation status of interleukin-6 gene promoter in patients with Behçet's disease.


Journal

Reumatologia clinica
ISSN: 2173-5743
Titre abrégé: Reumatol Clin (Engl Ed)
Pays: Spain
ID NLM: 101717526

Informations de publication

Date de publication:
Historique:
received: 11 12 2017
revised: 07 05 2018
accepted: 22 06 2018
pubmed: 5 8 2018
medline: 6 8 2021
entrez: 5 8 2018
Statut: ppublish

Résumé

IL-6 mRNA expression is significantly high in many autoimmune diseases such as Behçet's disease; this is often related with more aggressive phenotypes. Nevertheless, the essential molecular process for its high expression has not been completely realized. The aim of this study was undertaken to estimate the gene copy number variation and promoter methylation to IL-6's high expression. This study was performed on 51 patients and 61 healthy controls. Initially, DNA and RNA were extracted from all specimens. Promoter methylation levels of IL-6 were evaluated by MeDIP-qPCR technique. Also, IL-6 gene expression was measured by Real-time PCR. After that, we evaluated the relationship between gene expression and methylation, as well as their relationship with clinical specification. As we expected, the expression level of IL-6 gene increased significantly in the patient group compared to the healthy subjects. Also, the relative promoter methylation level of the IL-6 mRNA was significantly lower in patient group compared to healthy group (p<0.001). We disclosed that the promoter hypomethylation may be considered as one of the main defects for IL-6 mRNA high expression in patients with Behçet's disease.

Sections du résumé

BACKGROUND BACKGROUND
IL-6 mRNA expression is significantly high in many autoimmune diseases such as Behçet's disease; this is often related with more aggressive phenotypes. Nevertheless, the essential molecular process for its high expression has not been completely realized. The aim of this study was undertaken to estimate the gene copy number variation and promoter methylation to IL-6's high expression.
METHODS METHODS
This study was performed on 51 patients and 61 healthy controls. Initially, DNA and RNA were extracted from all specimens. Promoter methylation levels of IL-6 were evaluated by MeDIP-qPCR technique. Also, IL-6 gene expression was measured by Real-time PCR. After that, we evaluated the relationship between gene expression and methylation, as well as their relationship with clinical specification.
RESULTS RESULTS
As we expected, the expression level of IL-6 gene increased significantly in the patient group compared to the healthy subjects. Also, the relative promoter methylation level of the IL-6 mRNA was significantly lower in patient group compared to healthy group (p<0.001).
DISCUSSION CONCLUSIONS
We disclosed that the promoter hypomethylation may be considered as one of the main defects for IL-6 mRNA high expression in patients with Behçet's disease.

Identifiants

pubmed: 30076035
pii: S1699-258X(18)30124-4
doi: 10.1016/j.reuma.2018.06.006
pii:
doi:

Substances chimiques

IL6 protein, human 0
Interleukin-6 0

Types de publication

Journal Article

Langues

eng spa

Sous-ensembles de citation

IM

Pagination

229-234

Informations de copyright

Copyright © 2018. Publicado por Elsevier España, S.L.U.

Auteurs

Shahriar Alipour (S)

Molecular Medicine, Faculty of Advanced Medical Sciences, Tabriz University of Medical Sciences, Iran; Connective Tissue Diseases Research Center, Tabriz University of Medical Science, Iran.

Ebrahim Sakhinia (E)

Department of Biochemistry, Faculty of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran.

Alireza Khabbazi (A)

Connective Tissue Diseases Research Center, Tabriz University of Medical Science, Iran.

Nasser Samadi (N)

Cancer Biochemistry, Cancer Biotechnology, Faculty of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran.

Zohreh Babaloo (Z)

Department of Immunology Medicine Faculty, Tabriz University of Medical Sciences, Tabriz, Iran.

Mahdi Azad (M)

Department of Medical Laboratory Sciences, Faculty of Allied Medicine, Qazvin University of Medical Sciences, Qazvin, Iran.

Somayeh Abolhasani (S)

Faculty of Dentistry, Tabriz University of Medical Sciences, Tabriz, Iran.

Jafar Farhadi (J)

Molecular Medicine, Faculty of Advanced Medical Sciences, Tabriz University of Medical Sciences, Iran; Connective Tissue Diseases Research Center, Tabriz University of Medical Science, Iran.

Golamreza Jadideslam (G)

Molecular Medicine, Faculty of Advanced Medical Sciences, Tabriz University of Medical Sciences, Iran; Connective Tissue Diseases Research Center, Tabriz University of Medical Science, Iran.

Neda Roshanravan (N)

Cardiovascular Research Center, Tabriz University of Medical Sciences, Tabriz, Iran.

Mohammad Nouri (M)

Department of Biochemistry, Faculty of Medicine, Tabriz University of Medical Sciences, Tabriz, Iran. Electronic address: nourimd@yahoo.com.

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Classifications MeSH