Outcomes of endovascular thrombectomy with and without bridging thrombolysis for acute large vessel occlusion ischaemic stroke.


Journal

Internal medicine journal
ISSN: 1445-5994
Titre abrégé: Intern Med J
Pays: Australia
ID NLM: 101092952

Informations de publication

Date de publication:
Mar 2019
Historique:
received: 01 06 2018
revised: 21 07 2018
accepted: 29 07 2018
pubmed: 10 8 2018
medline: 4 12 2019
entrez: 10 8 2018
Statut: ppublish

Résumé

Endovascular thrombectomy (EVT) for management of large vessel occlusion (LVO) acute ischaemic stroke is now current best practice. To determine if bridging intravenous (i.v.) alteplase therapy confers any clinical benefit. A retrospective study of patients treated with EVT for LVO was performed. Outcomes were compared between patients receiving thrombolysis and EVT with EVT alone. Primary end-points were reperfusion rate, 90-day functional outcome and mortality using the modified Rankin Scale (mRS) and symptomatic intracranial haemorrhage (sICH). A total of 355 patients who underwent EVT was included: 210 with thrombolysis (59%) and 145 without (41%). The reperfusion rate was higher in the group receiving i.v. tissue plasminogen activator (tPA) (unadjusted odds ratio (OR) 2.2, 95% confidence interval (CI): 1.29-3.73, P = 0.004), although this effect was attenuated when all variables were considered (adjusted OR (AOR) 1.22, 95% CI: 0.60-2.5, P = 0.580). The percentage achieving functional independence (mRS 0-2) at 90 days was higher in patients who received bridging i.v. tPA (AOR 2.17, 95% CI: 1.06-4.44, P = 0.033). There was no significant difference in major complications, including sICH (AOR 1.4, 95% CI: 0.51-3.83, P = 0.512). There was lower 90-day mortality in the bridging i.v. tPA group (AOR 0.79, 95% CI: 0.36-1.74, P = 0.551). Fewer thrombectomy passes (2 versus 3, P = 0.012) were required to achieve successful reperfusion in the i.v. tPA group. Successful reperfusion (modified thrombolysis in cerebral infarction ≥2b) was the strongest predictor for 90-day functional independence (AOR 10.4, 95% CI:3.6-29.7, P < 0.001). Our study supports the current practice of administering i.v. alteplase before endovascular therapy.

Sections du résumé

BACKGROUND BACKGROUND
Endovascular thrombectomy (EVT) for management of large vessel occlusion (LVO) acute ischaemic stroke is now current best practice.
AIM OBJECTIVE
To determine if bridging intravenous (i.v.) alteplase therapy confers any clinical benefit.
METHODS METHODS
A retrospective study of patients treated with EVT for LVO was performed. Outcomes were compared between patients receiving thrombolysis and EVT with EVT alone. Primary end-points were reperfusion rate, 90-day functional outcome and mortality using the modified Rankin Scale (mRS) and symptomatic intracranial haemorrhage (sICH).
RESULTS RESULTS
A total of 355 patients who underwent EVT was included: 210 with thrombolysis (59%) and 145 without (41%). The reperfusion rate was higher in the group receiving i.v. tissue plasminogen activator (tPA) (unadjusted odds ratio (OR) 2.2, 95% confidence interval (CI): 1.29-3.73, P = 0.004), although this effect was attenuated when all variables were considered (adjusted OR (AOR) 1.22, 95% CI: 0.60-2.5, P = 0.580). The percentage achieving functional independence (mRS 0-2) at 90 days was higher in patients who received bridging i.v. tPA (AOR 2.17, 95% CI: 1.06-4.44, P = 0.033). There was no significant difference in major complications, including sICH (AOR 1.4, 95% CI: 0.51-3.83, P = 0.512). There was lower 90-day mortality in the bridging i.v. tPA group (AOR 0.79, 95% CI: 0.36-1.74, P = 0.551). Fewer thrombectomy passes (2 versus 3, P = 0.012) were required to achieve successful reperfusion in the i.v. tPA group. Successful reperfusion (modified thrombolysis in cerebral infarction ≥2b) was the strongest predictor for 90-day functional independence (AOR 10.4, 95% CI:3.6-29.7, P < 0.001).
CONCLUSION CONCLUSIONS
Our study supports the current practice of administering i.v. alteplase before endovascular therapy.

Identifiants

pubmed: 30091271
doi: 10.1111/imj.14069
doi:

Substances chimiques

Fibrinolytic Agents 0
Tissue Plasminogen Activator EC 3.4.21.68

Types de publication

Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

345-351

Informations de copyright

© 2018 Royal Australasian College of Physicians.

Auteurs

Julian Maingard (J)

Interventional Neuroradiology Service, Radiology Department, Austin Hospital, Melbourne, Victoria, Australia.
School of Medicine, Faculty of Health, Deakin University, Geelong, Victoria, Australia.

Yasmin Shvarts (Y)

Monash University, Melbourne, Victoria, Australia.

Ronan Motyer (R)

Interventional Neuroradiology Service, Department of Radiology, Beaumont Hospital, Dublin, Ireland.

Vincent Thijs (V)

Stroke Division, Florey Institute of Neuroscience and Mental Health, The University of Melbourne, Melbourne, Victoria, Australia.
Department of Neurology, Austin Health, Melbourne, Victoria, Australia.

Paul Brennan (P)

Interventional Neuroradiology Service, Department of Radiology, Beaumont Hospital, Dublin, Ireland.

Alan O'Hare (A)

Interventional Neuroradiology Service, Department of Radiology, Beaumont Hospital, Dublin, Ireland.

Seamus Looby (S)

Interventional Neuroradiology Service, Department of Radiology, Beaumont Hospital, Dublin, Ireland.

John Thornton (J)

Interventional Neuroradiology Service, Department of Radiology, Beaumont Hospital, Dublin, Ireland.

Joshua A Hirsch (JA)

Neuroendovascular Program, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts, USA.

Christen D Barras (CD)

South Australian Health and Medical Research Institute, Adelaide, South Australia, Australia.
Faculty of Health and Medical Sciences, The University of Adelaide, Adelaide, South Australia, Australia.

Ronil V Chandra (RV)

Interventional Neuroradiology Unit, Monash Imaging, Monash Health, Melbourne, Victoria, Australia.
Department of Imaging, Monash University, Melbourne, Victoria, Australia.

Mark Brooks (M)

Interventional Neuroradiology Service, Radiology Department, Austin Hospital, Melbourne, Victoria, Australia.
School of Medicine, Faculty of Health, Deakin University, Geelong, Victoria, Australia.
Stroke Division, Florey Institute of Neuroscience and Mental Health, The University of Melbourne, Melbourne, Victoria, Australia.

Hamed Asadi (H)

Interventional Neuroradiology Service, Radiology Department, Austin Hospital, Melbourne, Victoria, Australia.
School of Medicine, Faculty of Health, Deakin University, Geelong, Victoria, Australia.
Stroke Division, Florey Institute of Neuroscience and Mental Health, The University of Melbourne, Melbourne, Victoria, Australia.
Interventional Neuroradiology Unit, Monash Imaging, Monash Health, Melbourne, Victoria, Australia.
Department of Imaging, Monash University, Melbourne, Victoria, Australia.

Hong K Kok (HK)

Interventional Radiology, Department of Radiology, Northern Hospital, Melbourne, Victoria, Australia.

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