Therapeutic potential of A2 adenosine receptor pharmacological regulators in the treatment of cardiovascular diseases, recent progress, and prospective.


Journal

Journal of cellular physiology
ISSN: 1097-4652
Titre abrégé: J Cell Physiol
Pays: United States
ID NLM: 0050222

Informations de publication

Date de publication:
02 2019
Historique:
received: 11 11 2017
accepted: 10 07 2018
pubmed: 28 8 2018
medline: 18 12 2019
entrez: 28 8 2018
Statut: ppublish

Résumé

Adenosine and its analogs are of particular interest as potential therapeutic agents for treatment of cardiovascular diseases (CVDs). A2 adenosine receptor subtypes (A2a and A2b) are extensively expressed in cardiovascular system, and modulation of these receptors using A2 adenosine receptor agonists or antagonists regulates heart rate, blood pressure, heart rate variability, and cardiovascular toxicity during both normoxia and hypoxia conditions. Regulation of A2 adenosine receptor signaling via specific and novel pharmacological regulators is a potentially novel therapeutic approach for a better understanding and hence a better management of CVDs. This review summarizes the role of pharmacological A2 adenosine receptor regulators in the pathogenesis of CVDs.

Identifiants

pubmed: 30146778
doi: 10.1002/jcp.27161
doi:

Substances chimiques

Adenosine A2 Receptor Agonists 0
Adenosine A2 Receptor Antagonists 0
Receptors, Adenosine A2 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

1295-1299

Informations de copyright

© 2018 Wiley Periodicals, Inc.

Auteurs

Amirhossein Bahreyni (A)

Department of Clinical Biochemistry and Immunogenetic Research Center, Faculty of Medicine, Mazandaran University of Medical Sciences, Sari, Iran.
Student Research Committee, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Amir Avan (A)

Metabolic Syndrome Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.
Department of Modern Sciences and Technologies, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Mohammad Shabani (M)

Department of Medical Biochemistry, School of Medicine, Iran University of Medical Sciences, Tehran, Iran.

Mikhail Ryzhikov (M)

Division of Pulmonary and Critical Care Medicine, School of Medicine, Washington University, St. Louis, Missouri.

Hamid Fiuji (H)

Department of Biochemistry, Payam-e-Noor University, Mashhad, Iran.

Saman Soleimanpour (S)

Department of Microbiology and Virology, School of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Majid Khazaei (M)

Metabolic Syndrome Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.
Department of Medical Physiology, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Seyed Mahdi Hassanian (SM)

Metabolic Syndrome Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.
Department of Medical Biochemistry, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.
Microanatomy Research Center, Mashhad University of Medical Sciences, Mashhad, Iran.

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Classifications MeSH