Towards a Dynamic Exploration of Vision, Cognition and Emotion in Alcohol-Use Disorders.
Alcohol-use disorders
bottom-up processes
magnocellular pathway
orbitofrontal cortex
parvocellular pathway
top-down processes
visual prediction
visuoperceptive deficits.
Journal
Current neuropharmacology
ISSN: 1875-6190
Titre abrégé: Curr Neuropharmacol
Pays: United Arab Emirates
ID NLM: 101157239
Informations de publication
Date de publication:
2019
2019
Historique:
received:
15
12
2017
revised:
30
07
2018
accepted:
17
08
2018
pubmed:
29
8
2018
medline:
17
9
2019
entrez:
29
8
2018
Statut:
ppublish
Résumé
Visuoperceptive impairments are among the most frequently reported deficits in alcoholuse disorders, but only very few studies have investigated their origin and interactions with other categories of dysfunctions. Besides, these deficits have generally been interpreted in a linear bottom- up perspective, which appears very restrictive with respect to the new models of vision developed in healthy populations. Indeed, new theories highlight the predictive nature of the visual system and demonstrate that it interacts with higher-level cognitive functions to generate top-down predictions. These models notably posit that a fast but coarse visual analysis involving magnocellular pathways helps to compute heuristic guesses regarding the identity and affective value of inputs, which are used to facilitate conscious visual recognition. Building on these new proposals, the present review stresses the need to reconsider visual deficits in alcohol-use disorders as they might have crucial significance for core features of the pathology, such as attentional bias, loss of inhibitory control and emotion decoding impairments. Centrally, we suggest that individuals with severe alcohol-use disorders could present with magnocellular damage and we defend a dynamic explanation of the deficits. Rather than being restricted to high-level processes, deficits could start at early visual stages and then extend and potentially intensify during following steps due to reduced cerebral connectivity and dysfunctional cognitive/emotional regions. A new research agenda is specifically provided to test these hypotheses.
Identifiants
pubmed: 30152285
pii: CN-EPUB-92621
doi: 10.2174/1570159X16666180828100441
pmc: PMC6712295
doi:
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
492-506Informations de copyright
Copyright© Bentham Science Publishers; For any queries, please email at epub@benthamscience.net.
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