Necrotic, apoptotic and autophagic cell fates triggered by nanoparticles.
Apoptosis
/ drug effects
Autophagy
/ drug effects
Endoplasmic Reticulum Stress
/ drug effects
Lysosomes
/ drug effects
Metal Nanoparticles
/ chemistry
Mitochondria
/ drug effects
Necroptosis
/ drug effects
Necrosis
Particle Size
Quantum Dots
/ chemistry
Reactive Oxygen Species
/ metabolism
Signal Transduction
Apoptosis
autophagy
cell death
lysosome
nanoparticles; nanotoxicity
necrosis
stress
Journal
Autophagy
ISSN: 1554-8635
Titre abrégé: Autophagy
Pays: United States
ID NLM: 101265188
Informations de publication
Date de publication:
01 2019
01 2019
Historique:
pubmed:
31
8
2018
medline:
13
3
2020
entrez:
31
8
2018
Statut:
ppublish
Résumé
Nanomaterials have gained a rapid increase in use in a variety of applications that pertain to many aspects of human life. The majority of these innovations are centered on medical applications and a range of industrial and environmental uses ranging from electronics to environmental remediation. Despite the advantages of NPs, the knowledge of their toxicological behavior and their interactions with the cellular machinery that determines cell fate is extremely limited. This review is an attempt to summarize and increase our understanding of the mechanistic basis of nanomaterial interactions with the cellular machinery that governs cell fate and activity. We review the mechanisms of NP-induced necrosis, apoptosis and autophagy and potential implications of these pathways in nanomaterial-induced outcomes. Abbreviations: Ag, silver; CdTe, cadmium telluride; CNTs, carbon nanotubes; EC, endothelial cell; GFP, green fluorescent protein; GO, graphene oxide; GSH, glutathione; HUVECs, human umbilical vein endothelial cells; NP, nanoparticle; PEI, polyethylenimine; PVP, polyvinylpyrrolidone; QD, quantum dot; ROS, reactive oxygen species; SiO
Identifiants
pubmed: 30160607
doi: 10.1080/15548627.2018.1509171
pmc: PMC6287681
doi:
Substances chimiques
Reactive Oxygen Species
0
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
4-33Subventions
Organisme : NIGMS NIH HHS
ID : R35 GM131919
Pays : United States
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