Human epididymis protein 4 (HE4) levels inversely correlate with lung function improvement (delta FEV


Journal

Journal of cystic fibrosis : official journal of the European Cystic Fibrosis Society
ISSN: 1873-5010
Titre abrégé: J Cyst Fibros
Pays: Netherlands
ID NLM: 101128966

Informations de publication

Date de publication:
03 2019
Historique:
received: 22 05 2018
revised: 28 08 2018
accepted: 28 08 2018
pubmed: 1 10 2018
medline: 17 7 2020
entrez: 1 10 2018
Statut: ppublish

Résumé

We have recently shown that human epididymis protein 4 (HE4) levels correlate with the severity of cystic fibrosis (CF) lung disease. However, there are no data on how HE4 levels alter in patients receiving CFTR modulating therapy. In this retrospective clinical study, 3 independent CF patient cohorts (US-American: 29, Australian: 12 and Irish: 19 cases) were enrolled carrying at least one Class III CFTR CF-causing mutation (p.Gly551Asp) and being treated with CFTR potentiator ivacaftor. Plasma HE4 was measured by immunoassay before treatment (baseline) and 1-6 months after commencement of ivacaftor, and were correlated with FEV After 1 month of therapy, HE4 levels were significantly lower than at baseline and remained decreased up to 6 months. A significant inverse correlation between absolute and delta values of HE4 and FEV This study shows that plasma HE4 levels inversely correlate with lung function improvement in CF patients receiving ivacaftor. Overall, this potential biomarker may be of value for routine clinical and laboratory follow-up of CFTR modulating therapy.

Sections du résumé

BACKGROUND
We have recently shown that human epididymis protein 4 (HE4) levels correlate with the severity of cystic fibrosis (CF) lung disease. However, there are no data on how HE4 levels alter in patients receiving CFTR modulating therapy.
METHODS
In this retrospective clinical study, 3 independent CF patient cohorts (US-American: 29, Australian: 12 and Irish: 19 cases) were enrolled carrying at least one Class III CFTR CF-causing mutation (p.Gly551Asp) and being treated with CFTR potentiator ivacaftor. Plasma HE4 was measured by immunoassay before treatment (baseline) and 1-6 months after commencement of ivacaftor, and were correlated with FEV
RESULTS
After 1 month of therapy, HE4 levels were significantly lower than at baseline and remained decreased up to 6 months. A significant inverse correlation between absolute and delta values of HE4 and FEV
CONCLUSIONS
This study shows that plasma HE4 levels inversely correlate with lung function improvement in CF patients receiving ivacaftor. Overall, this potential biomarker may be of value for routine clinical and laboratory follow-up of CFTR modulating therapy.

Identifiants

pubmed: 30268371
pii: S1569-1993(18)30795-1
doi: 10.1016/j.jcf.2018.08.013
pii:
doi:

Substances chimiques

Aminophenols 0
Biomarkers 0
Chloride Channel Agonists 0
Quinolones 0
WAP Four-Disulfide Core Domain Protein 2 0
WFDC2 protein, human 0
Cystic Fibrosis Transmembrane Conductance Regulator 126880-72-6
ivacaftor 1Y740ILL1Z

Types de publication

Clinical Study Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

271-277

Informations de copyright

Copyright © 2018 European Cystic Fibrosis Society. Published by Elsevier B.V. All rights reserved.

Auteurs

Béla Nagy (B)

Department of Laboratory Medicine, Faculty of Medicine, University of Debrecen, Debrecen, Hungary. Electronic address: nagy.bela@med.unideb.hu.

Zsolt Bene (Z)

Department of Pediatrics, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.

Zsolt Fejes (Z)

Department of Laboratory Medicine, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.

Sonya L Heltshe (SL)

Department of Pediatrics, University of Washington School of Medicine, Seattle, WA, USA.

David Reid (D)

QIMR Berghofer Medical Research Institute and The Prince Charles Hospital, Brisbane, Australia.

Nicola J Ronan (NJ)

Cork Adult Cystic Fibrosis Centre, Cork University Hospital, Cork, Ireland.

Yvonne McCarthy (Y)

Cork Adult Cystic Fibrosis Centre, Cork University Hospital, Cork, Ireland.

Daniel Smith (D)

QIMR Berghofer Medical Research Institute and The Prince Charles Hospital, Brisbane, Australia.

Attila Nagy (A)

Department of Preventive Medicine, Faculty of Public Health, University of Debrecen, Debrecen, Hungary.

Elizabeth Joseloff (E)

Cystic Fibrosis Foundation, Bethesda, MD, USA.

György Balla (G)

Department of Pediatrics, Faculty of Medicine, University of Debrecen, Debrecen, Hungary; MTA-DE Vascular Biology, Thrombosis and Hemostasis Research Group, Hungarian Academy of Sciences, Debrecen, Hungary.

János Kappelmayer (J)

Department of Laboratory Medicine, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.

Milan Macek (M)

Department of Biology and Medical Genetics, Charles University and Motol University Hospital, Prague, Czech Republic.

Scott C Bell (SC)

QIMR Berghofer Medical Research Institute and The Prince Charles Hospital, Brisbane, Australia.

Barry J Plant (BJ)

Cork Adult Cystic Fibrosis Centre, Cork University Hospital, Cork, Ireland.

Margarida D Amaral (MD)

University of Lisboa, Faculty of Sciences, BioISI-Biosystems & Integrative Sciences Institute, Lisboa, Portugal.

István Balogh (I)

Department of Laboratory Medicine, Faculty of Medicine, University of Debrecen, Debrecen, Hungary; Division of Clinical Genetics, Department of Laboratory Medicine, Faculty of Medicine, University of Debrecen, Debrecen, Hungary.

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