Right and Left Heart Function in Lowlanders with COPD at Altitude: Data from a Randomized Study.


Journal

Respiration; international review of thoracic diseases
ISSN: 1423-0356
Titre abrégé: Respiration
Pays: Switzerland
ID NLM: 0137356

Informations de publication

Date de publication:
Historique:
received: 15 05 2018
accepted: 13 08 2018
pubmed: 1 10 2018
medline: 2 4 2020
entrez: 1 10 2018
Statut: ppublish

Résumé

Changes in pulmonary hemodynamics and cardiac function in patients with chronic obstructive pulmonary disease (COPD) traveling to altitude have not been assessed despite an increasing prevalence of the disease. We hypothesized that pulmonary artery pressure (PAP) significantly increases and cardiac function deteriorates during exposure to hypobaric hypoxia as encountered by traveling to moderate altitude or air flight. A total of 37 patients (17 female; median age [quartiles] 66 years [60; 69] with COPD GOLD grade 2-3 [FEV1 57% predicted (49; 71)]) living < 800 m underwent echocardiography in Zurich (490 m) and after 1 night at Davos Jakobshorn (2,590 m) in a randomized order of allocation. The transtricuspid pressure gradient increased from 23 mm Hg (18; 29) to 32 mm Hg (25; 41) (p < 0.0001; Δmedian [95% CI] 7.5 [2.0; 13.0]), the right ventricular fractional area change decreased from 45% (39; 49) to 38% (33; 43) (p = 0.002), while the heart rate and systolic blood pressure increased from 70 bpm (64; 78) to 82 bpm (70; 86) (p < 0.0001) and from 133 mm Hg (123; 141) to 136 mm Hg (126; 148) (p = 0.002), respectively, and left ventricular diastolic dysfunction was more prevalent (24-54%, p = 0.02). This is a first study assessing changes in pulmonary hemodynamics and cardiac function in patients with COPD during a short altitude sojourn. Despite the increase in PAP and indications of change in cardiac function, the exposure was well tolerated. None of the patients had to descend to lower altitude for symptomatic altitude-related disease.

Sections du résumé

BACKGROUND BACKGROUND
Changes in pulmonary hemodynamics and cardiac function in patients with chronic obstructive pulmonary disease (COPD) traveling to altitude have not been assessed despite an increasing prevalence of the disease.
OBJECTIVES OBJECTIVE
We hypothesized that pulmonary artery pressure (PAP) significantly increases and cardiac function deteriorates during exposure to hypobaric hypoxia as encountered by traveling to moderate altitude or air flight.
METHODS METHODS
A total of 37 patients (17 female; median age [quartiles] 66 years [60; 69] with COPD GOLD grade 2-3 [FEV1 57% predicted (49; 71)]) living < 800 m underwent echocardiography in Zurich (490 m) and after 1 night at Davos Jakobshorn (2,590 m) in a randomized order of allocation.
RESULTS RESULTS
The transtricuspid pressure gradient increased from 23 mm Hg (18; 29) to 32 mm Hg (25; 41) (p < 0.0001; Δmedian [95% CI] 7.5 [2.0; 13.0]), the right ventricular fractional area change decreased from 45% (39; 49) to 38% (33; 43) (p = 0.002), while the heart rate and systolic blood pressure increased from 70 bpm (64; 78) to 82 bpm (70; 86) (p < 0.0001) and from 133 mm Hg (123; 141) to 136 mm Hg (126; 148) (p = 0.002), respectively, and left ventricular diastolic dysfunction was more prevalent (24-54%, p = 0.02).
CONCLUSIONS CONCLUSIONS
This is a first study assessing changes in pulmonary hemodynamics and cardiac function in patients with COPD during a short altitude sojourn. Despite the increase in PAP and indications of change in cardiac function, the exposure was well tolerated. None of the patients had to descend to lower altitude for symptomatic altitude-related disease.

Identifiants

pubmed: 30269143
pii: 000492898
doi: 10.1159/000492898
doi:

Types de publication

Journal Article Randomized Controlled Trial

Langues

eng

Sous-ensembles de citation

IM

Pagination

125-134

Commentaires et corrections

Type : CommentIn

Informations de copyright

© 2018 S. Karger AG, Basel.

Auteurs

Mona Lichtblau (M)

Department of Pulmonology, University Hospital Zurich, Zurich, Switzerland.

Tsogyal D Latshang (TD)

Department of Pulmonology, University Hospital Zurich, Zurich, Switzerland.

Michael Furian (M)

Department of Pulmonology, University Hospital Zurich, Zurich, Switzerland.

Séverine Müller-Mottet (S)

Department of Pulmonology, University Hospital Zurich, Zurich, Switzerland.

Silke Küest (S)

Department of Cardiology, University Hospital Zurich, Zurich, Switzerland.

Felix Tanner (F)

Department of Cardiology, University Hospital Zurich, Zurich, Switzerland.

Ekkehard Grünig (E)

Thoraxklinik, University Hospital Heidelberg, German Center for Lung Research (DZL), Heidelberg, Germany.

Konrad E Bloch (KE)

Department of Pulmonology, University Hospital Zurich, Zurich, Switzerland.

Silvia Ulrich (S)

Department of Pulmonology, University Hospital Zurich, Zurich, Switzerlandsilvia.ulrich@usz.ch.

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Classifications MeSH