GLP-1 Receptor in Pancreatic α-Cells Regulates Glucagon Secretion in a Glucose-Dependent Bidirectional Manner.
Journal
Diabetes
ISSN: 1939-327X
Titre abrégé: Diabetes
Pays: United States
ID NLM: 0372763
Informations de publication
Date de publication:
01 2019
01 2019
Historique:
received:
15
03
2018
accepted:
15
10
2018
pubmed:
6
11
2018
medline:
26
3
2019
entrez:
4
11
2018
Statut:
ppublish
Résumé
Glucagon-like peptide 1 (GLP-1) is known to suppress glucagon secretion, but the mechanism by which GLP-1 exerts this effect is unclear. In this study, we demonstrated GLP-1 receptor (GLP-1R) expression in α-cells using both antibody-dependent and antibody-independent strategies. A novel α-cell-specific GLP-1R knockout (αGLP-1R
Identifiants
pubmed: 30389749
pii: db18-0317
doi: 10.2337/db18-0317
pmc: PMC6302540
doi:
Substances chimiques
Glucagon-Like Peptide-1 Receptor
0
Glucagon
9007-92-5
Glucose
IY9XDZ35W2
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Research Support, U.S. Gov't, Non-P.H.S.
Langues
eng
Sous-ensembles de citation
IM
Pagination
34-44Subventions
Organisme : BLRD VA
ID : I01 BX003725
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK074970
Pays : United States
Organisme : NIGMS NIH HHS
ID : U54 GM104940
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK107412
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK081463
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK107444
Pays : United States
Commentaires et corrections
Type : ErratumIn
Informations de copyright
© 2018 by the American Diabetes Association.
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