Temporary autonomic modulation with botulinum toxin type A to reduce atrial fibrillation after cardiac surgery.


Journal

Heart rhythm
ISSN: 1556-3871
Titre abrégé: Heart Rhythm
Pays: United States
ID NLM: 101200317

Informations de publication

Date de publication:
02 2019
Historique:
received: 29 05 2018
pubmed: 12 11 2018
medline: 3 10 2020
entrez: 12 11 2018
Statut: ppublish

Résumé

Postoperative atrial fibrillation (POAF) frequently complicates cardiac surgery and is associated with worse outcomes. The cardiac autonomic nervous system is implicated in the pathogenesis of POAF. The purpose of this study was to determine the efficacy and safety of selective cardiac autonomic modulation in preventing POAF. In this randomized, double-blind, placebo-controlled trial, adults undergoing cardiac surgery were randomized 1:1 to intraoperative injection of 250 units onabotulinumtoxinA (botulinum toxin type A [BoNTA]) or placebo into epicardial fat pads. The study was powered to detect a 40% reduction in relative risk of POAF. Time to first episode of in-hospital POAF was the primary outcome, evaluated in patients receiving injection. Additionally, incidence of POAF, length of stay (LOS), and adverse events were examined. The trial assigned 145 patients to injection, 15 of whom were dropped before treatment, leaving 130 patients for analysis. Overall, 36.5% (23/63) of BoNTA-treated patients developed POAF compared with 47.8% (32/67) of placebo-treated patients. The time-to-event analysis revealed a hazard ratio of 0.69 (95% confidence interval 0.41-1.19; P = .18) for the BoNTA vs placebo arm. There were no significant differences in postoperative hospital LOS (median [interquartile range] 6.0 [3.4] vs 6.2 [3.7] days; P = .51) or adverse events prolonging LOS (27/63 [42.9%] vs 30/67 [44.8%]; P = .83) in patients receiving BoNTA vs placebo. Epicardial injection of onabotulinumtoxinA was without discernible adverse effects, but we failed to detect a significant difference in risk of POAF. Future large-scale studies of epicardial onabotulinumtoxinA injection as a potential POAF prevention strategy should be designed to study smaller, but clinically meaningful, treatment effects.

Sections du résumé

BACKGROUND
Postoperative atrial fibrillation (POAF) frequently complicates cardiac surgery and is associated with worse outcomes. The cardiac autonomic nervous system is implicated in the pathogenesis of POAF.
OBJECTIVE
The purpose of this study was to determine the efficacy and safety of selective cardiac autonomic modulation in preventing POAF.
METHODS
In this randomized, double-blind, placebo-controlled trial, adults undergoing cardiac surgery were randomized 1:1 to intraoperative injection of 250 units onabotulinumtoxinA (botulinum toxin type A [BoNTA]) or placebo into epicardial fat pads. The study was powered to detect a 40% reduction in relative risk of POAF. Time to first episode of in-hospital POAF was the primary outcome, evaluated in patients receiving injection. Additionally, incidence of POAF, length of stay (LOS), and adverse events were examined.
RESULTS
The trial assigned 145 patients to injection, 15 of whom were dropped before treatment, leaving 130 patients for analysis. Overall, 36.5% (23/63) of BoNTA-treated patients developed POAF compared with 47.8% (32/67) of placebo-treated patients. The time-to-event analysis revealed a hazard ratio of 0.69 (95% confidence interval 0.41-1.19; P = .18) for the BoNTA vs placebo arm. There were no significant differences in postoperative hospital LOS (median [interquartile range] 6.0 [3.4] vs 6.2 [3.7] days; P = .51) or adverse events prolonging LOS (27/63 [42.9%] vs 30/67 [44.8%]; P = .83) in patients receiving BoNTA vs placebo.
CONCLUSION
Epicardial injection of onabotulinumtoxinA was without discernible adverse effects, but we failed to detect a significant difference in risk of POAF. Future large-scale studies of epicardial onabotulinumtoxinA injection as a potential POAF prevention strategy should be designed to study smaller, but clinically meaningful, treatment effects.

Identifiants

pubmed: 30414840
pii: S1547-5271(18)30822-1
doi: 10.1016/j.hrthm.2018.08.021
pii:
doi:

Substances chimiques

Neurotoxins 0
Botulinum Toxins, Type A EC 3.4.24.69

Types de publication

Journal Article Multicenter Study Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

178-184

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2018 Heart Rhythm Society. Published by Elsevier Inc. All rights reserved.

Auteurs

Nathan H Waldron (NH)

Department of Anesthesiology, Duke University Medical Center, Durham, North Carolina; Duke Clinical Research Institute, Durham, North Carolina. Electronic address: nathan.waldron@dm.duke.edu.

Mary Cooter (M)

Department of Anesthesiology, Duke University Medical Center, Durham, North Carolina.

John C Haney (JC)

Department of Surgery (Cardiothoracic Surgery), Duke University Medical Center, Durham, North Carolina.

Jacob N Schroder (JN)

Department of Surgery (Cardiothoracic Surgery), Duke University Medical Center, Durham, North Carolina.

Jeffrey G Gaca (JG)

Department of Surgery (Cardiothoracic Surgery), Duke University Medical Center, Durham, North Carolina.

Shu S Lin (SS)

Department of Surgery (Cardiothoracic Surgery), Duke University Medical Center, Durham, North Carolina; Department of Immunology, Duke University Medical Center, Durham, North Carolina; Department of Pathology, Duke University Medical Center, Durham, North Carolina.

Martin I Sigurdsson (MI)

Department of Anesthesiology, Duke University Medical Center, Durham, North Carolina.

Marat Fudim (M)

Duke Clinical Research Institute, Durham, North Carolina; Department of Medicine (Cardiology), Duke University Medical Center, Durham, North Carolina.

Mihai V Podgoreanu (MV)

Department of Anesthesiology, Duke University Medical Center, Durham, North Carolina; Duke Clinical Research Institute, Durham, North Carolina.

Mark Stafford-Smith (M)

Department of Anesthesiology, Duke University Medical Center, Durham, North Carolina.

Carmelo A Milano (CA)

Department of Surgery (Cardiothoracic Surgery), Duke University Medical Center, Durham, North Carolina.

Jonathan P Piccini (JP)

Duke Clinical Research Institute, Durham, North Carolina; Department of Medicine (Cardiology), Duke University Medical Center, Durham, North Carolina.

Joseph P Mathew (JP)

Department of Anesthesiology, Duke University Medical Center, Durham, North Carolina.

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