Differentiation between glioblastoma and solitary brain metastasis using neurite orientation dispersion and density imaging.


Journal

Journal of neuroradiology = Journal de neuroradiologie
ISSN: 0150-9861
Titre abrégé: J Neuroradiol
Pays: France
ID NLM: 7705086

Informations de publication

Date de publication:
May 2020
Historique:
received: 02 06 2018
revised: 20 09 2018
accepted: 27 10 2018
pubmed: 16 11 2018
medline: 17 3 2021
entrez: 16 11 2018
Statut: ppublish

Résumé

Neurite orientation dispersion and density imaging (NODDI) is a new technique that applies a three-diffusion-compartment biophysical model. We assessed the usefulness of NODDI for the differentiation of glioblastoma from solitary brain metastasis. NODDI data were prospectively obtained on a 3T magnetic resonance imaging (MRI) scanner from patients with previously untreated, histopathologically confirmed glioblastoma (n = 9) or solitary brain metastasis (n = 6). Using the NODDI Matlab Toolbox, we generated maps of the intra-cellular, extra-cellular, and isotropic volume (VIC, VEC, VISO) fraction. Apparent diffusion coefficient - and fraction anisotropy maps were created from the diffusion data. On each map we manually drew a region of interest around the peritumoral signal-change (PSC) - and the enhancing solid area of the lesion. Differences between glioblastoma and metastatic lesions were assessed and the area under the receiver operating characteristic curve (AUC) was determined. On VEC maps the mean value of the PSC area was significantly higher for glioblastoma than metastasis (P < 0.05); on VISO maps it tended to be higher for metastasis than glioblastoma. There was no significant difference on the other maps. Among the 5 parameters, the VEC fraction in the PSC area showed the highest diagnostic performance. The VEC threshold value of ≥ 0.48 yielded 100% sensitivity, 83.3% specificity, and an AUC of 0.87 for differentiating between the two tumor types. NODDI compartment maps of the PSC area may help to differentiate between glioblastoma and solitary brain metastasis.

Sections du résumé

BACKGROUND AND PURPOSE OBJECTIVE
Neurite orientation dispersion and density imaging (NODDI) is a new technique that applies a three-diffusion-compartment biophysical model. We assessed the usefulness of NODDI for the differentiation of glioblastoma from solitary brain metastasis.
METHODS METHODS
NODDI data were prospectively obtained on a 3T magnetic resonance imaging (MRI) scanner from patients with previously untreated, histopathologically confirmed glioblastoma (n = 9) or solitary brain metastasis (n = 6). Using the NODDI Matlab Toolbox, we generated maps of the intra-cellular, extra-cellular, and isotropic volume (VIC, VEC, VISO) fraction. Apparent diffusion coefficient - and fraction anisotropy maps were created from the diffusion data. On each map we manually drew a region of interest around the peritumoral signal-change (PSC) - and the enhancing solid area of the lesion. Differences between glioblastoma and metastatic lesions were assessed and the area under the receiver operating characteristic curve (AUC) was determined.
RESULTS RESULTS
On VEC maps the mean value of the PSC area was significantly higher for glioblastoma than metastasis (P < 0.05); on VISO maps it tended to be higher for metastasis than glioblastoma. There was no significant difference on the other maps. Among the 5 parameters, the VEC fraction in the PSC area showed the highest diagnostic performance. The VEC threshold value of ≥ 0.48 yielded 100% sensitivity, 83.3% specificity, and an AUC of 0.87 for differentiating between the two tumor types.
CONCLUSIONS CONCLUSIONS
NODDI compartment maps of the PSC area may help to differentiate between glioblastoma and solitary brain metastasis.

Identifiants

pubmed: 30439396
pii: S0150-9861(18)30218-9
doi: 10.1016/j.neurad.2018.10.005
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

197-202

Informations de copyright

Copyright © 2018 The Authors. Published by Elsevier Masson SAS.. All rights reserved.

Auteurs

Yoshihito Kadota (Y)

Departments of Radiology, Faculty of Medicine, University of Miyazaki, 5200 Kihara, Kiyotake, Miyazaki 8891692, Japan. Electronic address: toshinorh@med.miyazaki-u.ac.jp.

Toshinori Hirai (T)

Departments of Radiology, Faculty of Medicine, University of Miyazaki, 5200 Kihara, Kiyotake, Miyazaki 8891692, Japan.

Minako Azuma (M)

Departments of Radiology, Faculty of Medicine, University of Miyazaki, 5200 Kihara, Kiyotake, Miyazaki 8891692, Japan.

Yohei Hattori (Y)

Departments of Radiology, Faculty of Medicine, University of Miyazaki, 5200 Kihara, Kiyotake, Miyazaki 8891692, Japan.

Zaw Aung Khant (ZA)

Departments of Radiology, Faculty of Medicine, University of Miyazaki, 5200 Kihara, Kiyotake, Miyazaki 8891692, Japan.

Masaaki Hori (M)

Department of Radiology, School of Medicine, Juntendo University, Tokyo, Japan.

Kiyotaka Saito (K)

Departments of Neurosurgery, Faculty of Medicine, University of Miyazaki, Miyazaki, Japan.

Kiyotaka Yokogami (K)

Departments of Neurosurgery, Faculty of Medicine, University of Miyazaki, Miyazaki, Japan.

Hideo Takeshima (H)

Departments of Neurosurgery, Faculty of Medicine, University of Miyazaki, Miyazaki, Japan.

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