In utero effects of maternal phthalate exposure on male genital development.


Journal

Prenatal diagnosis
ISSN: 1097-0223
Titre abrégé: Prenat Diagn
Pays: England
ID NLM: 8106540

Informations de publication

Date de publication:
02 2019
Historique:
received: 31 08 2018
revised: 21 11 2018
accepted: 22 11 2018
pubmed: 27 11 2018
medline: 18 12 2019
entrez: 27 11 2018
Statut: ppublish

Résumé

Phthalates are used extensively in commercial and personal care products and maternal exposure is ubiquitous. Phthalates are anti-androgenic, but the potential effects of phthalates on male penile development have not been assessed in utero. The study aims to investigate the association between early pregnancy phthalate exposure and fetal penile development, overall and by race. Prospective cohort study of women with singleton pregnancies presenting for prenatal ultrasound between 18 and 22 weeks' gestation. Maternal urine samples were assayed for eight phthalate monoester metabolites. We used maternal phthalate levels at 18 to 22 weeks' gestation as predictors of fetal size using multiple linear regression models, adjusted for fetal gestational age, maternal age, race, smoking, and education. We incorporated a phthalate by race interaction into a second set of regression models. We detected statistically significant race interactions for continuous phthalates with penile width. Race interactions were also suggested for penile length and volume using tertiles of phthalates with point estimates generally positive for whites and negative for African Americans. Penile development is significantly influenced by race, and the impact of maternal phthalates on penile measurements also varies by race. Maternal phthalate exposure can adversely affect in utero penile growth and development, especially among African Americans.

Sections du résumé

BACKGROUND
Phthalates are used extensively in commercial and personal care products and maternal exposure is ubiquitous. Phthalates are anti-androgenic, but the potential effects of phthalates on male penile development have not been assessed in utero.
OBJECTIVE
The study aims to investigate the association between early pregnancy phthalate exposure and fetal penile development, overall and by race.
METHODS
Prospective cohort study of women with singleton pregnancies presenting for prenatal ultrasound between 18 and 22 weeks' gestation. Maternal urine samples were assayed for eight phthalate monoester metabolites. We used maternal phthalate levels at 18 to 22 weeks' gestation as predictors of fetal size using multiple linear regression models, adjusted for fetal gestational age, maternal age, race, smoking, and education. We incorporated a phthalate by race interaction into a second set of regression models.
RESULTS
We detected statistically significant race interactions for continuous phthalates with penile width. Race interactions were also suggested for penile length and volume using tertiles of phthalates with point estimates generally positive for whites and negative for African Americans.
CONCLUSION
Penile development is significantly influenced by race, and the impact of maternal phthalates on penile measurements also varies by race. Maternal phthalate exposure can adversely affect in utero penile growth and development, especially among African Americans.

Identifiants

pubmed: 30476355
doi: 10.1002/pd.5398
doi:

Substances chimiques

Phthalic Acids 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

209-218

Informations de copyright

© 2018 John Wiley & Sons, Ltd.

Auteurs

Rebecca J Wineland (RJ)

Department of Obstetrics and Gynecology, Medical University of South Carolina, Charleston, South Carolina.

Michael S Bloom (MS)

Departments of Environmental Health Sciences and Epidemiology and Biostatistics, University at Albany, State University of New York, Rensselaer, New York.

Lori Cruze (L)

Wofford College, Spartanburg, South Carolina.

Celeste D Butts (CD)

Department of Environmental Health Sciences, University at Albany, State University of New York, Rensselaer, New York.

Abby G Wenzel (AG)

Marine Biomedicine and Environmental Science, Medical University of South Carolina, Charleston, South Carolina.

E Ramsey Unal (ER)

Department of Obstetrics and Gynecology, Southern Illinois University, Springfield, Illinois.

Satomi Kohno (S)

Department of Biology, St. Cloud State University, Saint Cloud, Minnesota.

Keith B Willan (KB)

Department of Obstetrics and Gynecology, Medical University of South Carolina, Charleston, South Carolina.

John W Brock (JW)

Department of Chemistry, University of North Carolina Asheville, Asheville, North Carolina.

Roger B Newman (RB)

Department of Obstetrics and Gynecology, Medical University of South Carolina, Charleston, South Carolina.

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Classifications MeSH