Organic chemicals from diesel exhaust particles affects intracellular calcium, inflammation and β-adrenoceptors in endothelial cells.
Calcium
/ metabolism
Calcium Signaling
/ drug effects
Cell Line
Endothelial Cells
/ drug effects
Gene Expression Regulation
Humans
Inflammation Mediators
/ metabolism
Organic Chemicals
/ toxicity
Receptor, PAR-2
/ drug effects
Receptors, Adrenergic, beta
/ drug effects
Receptors, G-Protein-Coupled
/ drug effects
Vehicle Emissions
/ toxicity
Beta adrenoceptors
Calcium signaling
Diesel exhaust particles
Endothelial dysfunction
Extractable organic material
Protease activated receptor
Journal
Toxicology letters
ISSN: 1879-3169
Titre abrégé: Toxicol Lett
Pays: Netherlands
ID NLM: 7709027
Informations de publication
Date de publication:
01 Mar 2019
01 Mar 2019
Historique:
received:
18
04
2018
revised:
14
09
2018
accepted:
19
11
2018
pubmed:
7
12
2018
medline:
27
2
2019
entrez:
4
12
2018
Statut:
ppublish
Résumé
Exposure to diesel exhaust particles (DEP) may contribute to endothelial dysfunction and cardiovascular disease. DEP, extractable organic material from DEP (DEP-EOM) and certain PAHs seem to trigger [Ca
Identifiants
pubmed: 30503853
pii: S0378-4274(18)32056-3
doi: 10.1016/j.toxlet.2018.11.009
pii:
doi:
Substances chimiques
F2RL1 protein, human
0
Inflammation Mediators
0
Organic Chemicals
0
Receptor, PAR-2
0
Receptors, Adrenergic, beta
0
Receptors, G-Protein-Coupled
0
Vehicle Emissions
0
Calcium
SY7Q814VUP
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
18-27Informations de copyright
Copyright © 2018 Elsevier B.V. All rights reserved.