Melatonin receptor 1B -1193T>C polymorphism is associated with diurnal preference and sleep habits.


Journal

Sleep medicine
ISSN: 1878-5506
Titre abrégé: Sleep Med
Pays: Netherlands
ID NLM: 100898759

Informations de publication

Date de publication:
01 2019
Historique:
received: 27 03 2018
revised: 26 08 2018
accepted: 03 09 2018
pubmed: 7 12 2018
medline: 10 3 2020
entrez: 4 12 2018
Statut: ppublish

Résumé

Melatonin modulates the master circadian clock through the activation of G-protein-coupled receptors MT1 and MT2. It is presumed, therefore, that genetic variations in melatonin receptors can affect both sleep and circadian phase. We investigated whether the -1193T > C (rs4753426) polymorphism in the promoter of MT2 receptor gene (MTNR1B) is associated with diurnal preference and sleep habits. This polymorphism was previously associated with sunshine duration, suggesting a role in circadian entrainment. A total of 814 subjects who completed the Morningness-Eveningness and the Munich Chronotype questionnaires were genotyped for the selected polymorphism. Linear and multinomial regression were performed to test the interaction between gene variants and diurnal preference/sleep habits. The -1193C variant was associated with the extreme morningness phenotype in a codominant model (C/C vs. T/T), recessive model (C/C + C/T vs. T/T) and alleles (C vs. T). A negative correlation was found between -1193C alleles and social jetlag scores. The frequency of -1193T allele was higher in the group that stay in bed more than 8 h/night compared to the group that stay in bed less than 8 h/night on weekends. To the best of our knowledge, these data provide the first insights into the role of MTNR1B gene in the regulation of sleep, biological rhythms, and entrainment in humans.

Sections du résumé

BACKGROUND
Melatonin modulates the master circadian clock through the activation of G-protein-coupled receptors MT1 and MT2. It is presumed, therefore, that genetic variations in melatonin receptors can affect both sleep and circadian phase. We investigated whether the -1193T > C (rs4753426) polymorphism in the promoter of MT2 receptor gene (MTNR1B) is associated with diurnal preference and sleep habits. This polymorphism was previously associated with sunshine duration, suggesting a role in circadian entrainment.
METHODS
A total of 814 subjects who completed the Morningness-Eveningness and the Munich Chronotype questionnaires were genotyped for the selected polymorphism. Linear and multinomial regression were performed to test the interaction between gene variants and diurnal preference/sleep habits.
RESULTS
The -1193C variant was associated with the extreme morningness phenotype in a codominant model (C/C vs. T/T), recessive model (C/C + C/T vs. T/T) and alleles (C vs. T). A negative correlation was found between -1193C alleles and social jetlag scores. The frequency of -1193T allele was higher in the group that stay in bed more than 8 h/night compared to the group that stay in bed less than 8 h/night on weekends.
CONCLUSION
To the best of our knowledge, these data provide the first insights into the role of MTNR1B gene in the regulation of sleep, biological rhythms, and entrainment in humans.

Identifiants

pubmed: 30508778
pii: S1389-9457(18)30813-X
doi: 10.1016/j.sleep.2018.09.023
pii:
doi:

Substances chimiques

Receptor, Melatonin, MT2 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

106-114

Informations de copyright

Copyright © 2018 Elsevier B.V. All rights reserved.

Auteurs

Aline Cristine Pereira E Silva (ACPE)

Laboratory of Molecular Chronobiology, Federal University of Alagoas (UFAL), Alagoas, Brazil.

Maria José Dos Santos (MJD)

Laboratory of Molecular Chronobiology, Federal University of Alagoas (UFAL), Alagoas, Brazil.

Bruna Del Vechio Koike (BDV)

Laboratory of Molecular Chronobiology, Federal University of Alagoas (UFAL), Alagoas, Brazil.

Magna Suzana Alexandre Moreira (MSA)

Laboratory of Pharmacology and Immunity, Federal University of Alagoas (UFAL), Alagoas, Brazil.

Daniel Leite Goes Gitai (DLG)

Laboratory of Cellular and Molecular Biology, Federal University of Alagoas (UFAL), Alagoas, Brazil.

Jorge Artur Peçanha de Miranda Coelho (JAP)

Faculty of Medicine, Federal University of Alagoas (UFAL), Alagoas, Brazil.

Tiago Gomes de Andrade (TG)

Laboratory of Molecular Chronobiology, Federal University of Alagoas (UFAL), Alagoas, Brazil; Faculty of Medicine, Federal University of Alagoas (UFAL), Alagoas, Brazil. Electronic address: deandrade.tiago@pq.cnpq.br.

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Classifications MeSH