Protective Effect of Selenium-L-methionine on Radiation-induced Acute Pneumonitis and Lung Fibrosis in Rat.


Journal

Current clinical pharmacology
ISSN: 2212-3938
Titre abrégé: Curr Clin Pharmacol
Pays: United Arab Emirates
ID NLM: 101273158

Informations de publication

Date de publication:
2019
Historique:
received: 05 09 2018
revised: 13 11 2018
accepted: 16 11 2018
pubmed: 18 12 2018
medline: 8 7 2020
entrez: 18 12 2018
Statut: ppublish

Résumé

In this study, we aimed to detect the changes in the level of interleukin (IL)-4 and IL-13 cytokines and their downstream genes including interleukin-13 receptor subunit alpha-2 (IL13Ra2), interleukin-4 receptor subunit alpha-1 (IL4Ra1), dual oxidase 1 (DUOX1) and dual oxidase 2 (DUOX2). The protective effects of Selenium-L-methionine on radiation-induced histopathological damages and changes in the level of these cytokines and genes were detected. Four groups of 20 rats (5 rats in each) namely, control; Selenium-L-methionine, radiation and radiation plus Selenium-L-methionine were used in this study. 4 mg/kg of Selenium-Lmethionine was administered 1 day before irradiation and five consecutive days after irradiation. Irradiation was done using a dose of 15 Gy 60Co gamma rays at 109 cGy/min. All rats were sacrificed 10 weeks after irradiation for detecting changes in IL-4 and IL-13 cytokines, the expressions of IL13Ra2, IL4Ra1, Duox1 and Duox2 and histopathological changes. The level of IL-4 but not IL-13 increased after irradiation. This was associated with increased expression of IL4Ra1, Duox1 and Duox2, in addition to changes in morphological properties. Selenium-L-methionine could attenuate all injury markers following lung irradiation. Selenium-L-methionine can protect lung tissues against toxic effects of ionizing radiation. It is possible that the modulation of immune responses and redox interactions are involved in the radioprotective effect of this agent.

Sections du résumé

BACKGROUND
In this study, we aimed to detect the changes in the level of interleukin (IL)-4 and IL-13 cytokines and their downstream genes including interleukin-13 receptor subunit alpha-2 (IL13Ra2), interleukin-4 receptor subunit alpha-1 (IL4Ra1), dual oxidase 1 (DUOX1) and dual oxidase 2 (DUOX2). The protective effects of Selenium-L-methionine on radiation-induced histopathological damages and changes in the level of these cytokines and genes were detected.
METHODS
Four groups of 20 rats (5 rats in each) namely, control; Selenium-L-methionine, radiation and radiation plus Selenium-L-methionine were used in this study. 4 mg/kg of Selenium-Lmethionine was administered 1 day before irradiation and five consecutive days after irradiation. Irradiation was done using a dose of 15 Gy 60Co gamma rays at 109 cGy/min. All rats were sacrificed 10 weeks after irradiation for detecting changes in IL-4 and IL-13 cytokines, the expressions of IL13Ra2, IL4Ra1, Duox1 and Duox2 and histopathological changes.
RESULTS
The level of IL-4 but not IL-13 increased after irradiation. This was associated with increased expression of IL4Ra1, Duox1 and Duox2, in addition to changes in morphological properties. Selenium-L-methionine could attenuate all injury markers following lung irradiation.
CONCLUSION
Selenium-L-methionine can protect lung tissues against toxic effects of ionizing radiation. It is possible that the modulation of immune responses and redox interactions are involved in the radioprotective effect of this agent.

Identifiants

pubmed: 30556505
pii: CCP-EPUB-95234
doi: 10.2174/1574884714666181214101917
pmc: PMC7040518
doi:

Substances chimiques

Il4r protein, rat 0
Interleukin-13 0
Interleukin-13 Receptor alpha2 Subunit 0
Interleukin-4 Receptor alpha Subunit 0
Radiation-Protective Agents 0
Interleukin-4 207137-56-2
Methionine AE28F7PNPL
Dual Oxidases EC 1.11.1.-
Duox1 protein, rat EC 1.6.3.1
Duox2 protein, rat EC 1.6.3.1
Selenium H6241UJ22B

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

157-164

Informations de copyright

Copyright© Bentham Science Publishers; For any queries, please email at epub@benthamscience.net.

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Auteurs

Peyman Amini (P)

Department of Radiology, Faculty of Paramedical, Tehran University of Medical Sciences, Tehran, Iran.

Sedighe Kolivand (S)

Department of Medical Biotechnology, School of Advanced Technologies in Medicine, Tehran University of Medical Sciences, Tehran, Iran.

Hana Saffar (H)

Department of Clinical and Anatomical Pathologist, Tehran University of Medical Science, Imam Khomeini Hospital Complex, Tehran, Iran.

Saeed Rezapoor (S)

Department of Radiology, Faculty of Paramedical, Tehran University of Medical Sciences, Tehran, Iran.

Elahe Motevaseli (E)

Department of Molecular Medicine, School of Advanced Technologies in Medicine, Tehran University of Medical Sciences, Tehran, Iran.

Masoud Najafi (M)

Radiology and Nuclear Medicine Department, School of Paramedical Sciences, Kermanshah University of Medical Sciences, Kermanshah, Iran.

Farzad Nouruzi (F)

Department of Medical Radiation Engineering, Science and Research Branch, Islamic Azad University, Tehran, Iran.

Dheyauldeen Shabeeb (D)

Department of Medical Physics and Biomedical Engineering, Faculty of Medicine, Tehran University of Medical Sciences (International Campus), Tehran, Iran.
Department of Physiology, College of Medicine, University of Misan, Misan, Iraq.

Ahmed Eleojo Musa (AE)

Department of Medical Physics and Biomedical Engineering, Faculty of Medicine, Tehran University of Medical Sciences (International Campus), Tehran, Iran.
Research Center for Molecular and Cellular Imaging, Tehran University of Medical Sciences (International Campus), Tehran, Iran.

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Classifications MeSH