Carbapenem-resistant Enterobacteriaceae dispersal from sinks is linked to drain position and drainage rates in a laboratory model system.


Journal

The Journal of hospital infection
ISSN: 1532-2939
Titre abrégé: J Hosp Infect
Pays: England
ID NLM: 8007166

Informations de publication

Date de publication:
May 2019
Historique:
received: 12 11 2018
accepted: 10 12 2018
pubmed: 21 12 2018
medline: 14 6 2019
entrez: 21 12 2018
Statut: ppublish

Résumé

Hospital sinks, waste traps and drains can harbour carbapenem-resistant Enterobacteriaceae (CRE). To investigate the dispersal of CRE from sinks in which water delivered from the tap flows directly into the drain and from clinical handwash basins with the drain at the rear. The effect of fast and slow drainage rates was also assessed. Waste traps, known to be colonized with CRE, were taken from a hospital and installed within a model laboratory system. New waste traps were also installed and artificially inoculated with CRE. The potential for bacteria to be dispersed from sinks was assessed using cyclone air samplers and/or settle plates. When the waste traps were artificially contaminated and CRE colonization was confined to the waste trap water, significantly fewer bacteria were dispersed from sinks that drained quickly (P = 0.004) and/or from rear-draining sinks (P = 0.002). When the waste traps were naturally contaminated and CRE colonized the trap, pipework and drain, there was significant interaction between sink drainage and position of the drain (P < 0.001). When drainage was slow, dispersal from rear-draining sinks was almost 30-fold less than from sinks with the drain underneath the tap (P < 0.001). When drainage was fast, rear-draining sinks again released comparatively fewer CRE, although, in this case, the difference was not statistically significant (P = 0.7). Contaminated splashes travelled up to 1 m from the sink. Slow drainage rates and sink designs with the drain directly underneath the tap increase the risk of CRE present in waste traps and drains contaminating the ward environment.

Sections du résumé

BACKGROUND BACKGROUND
Hospital sinks, waste traps and drains can harbour carbapenem-resistant Enterobacteriaceae (CRE).
AIM OBJECTIVE
To investigate the dispersal of CRE from sinks in which water delivered from the tap flows directly into the drain and from clinical handwash basins with the drain at the rear. The effect of fast and slow drainage rates was also assessed.
METHODS METHODS
Waste traps, known to be colonized with CRE, were taken from a hospital and installed within a model laboratory system. New waste traps were also installed and artificially inoculated with CRE. The potential for bacteria to be dispersed from sinks was assessed using cyclone air samplers and/or settle plates.
FINDINGS RESULTS
When the waste traps were artificially contaminated and CRE colonization was confined to the waste trap water, significantly fewer bacteria were dispersed from sinks that drained quickly (P = 0.004) and/or from rear-draining sinks (P = 0.002). When the waste traps were naturally contaminated and CRE colonized the trap, pipework and drain, there was significant interaction between sink drainage and position of the drain (P < 0.001). When drainage was slow, dispersal from rear-draining sinks was almost 30-fold less than from sinks with the drain underneath the tap (P < 0.001). When drainage was fast, rear-draining sinks again released comparatively fewer CRE, although, in this case, the difference was not statistically significant (P = 0.7). Contaminated splashes travelled up to 1 m from the sink.
CONCLUSION CONCLUSIONS
Slow drainage rates and sink designs with the drain directly underneath the tap increase the risk of CRE present in waste traps and drains contaminating the ward environment.

Identifiants

pubmed: 30571992
pii: S0195-6701(18)30711-4
doi: 10.1016/j.jhin.2018.12.007
pmc: PMC6504032
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

63-69

Investigateurs

Zoie Aiken (Z)
Oluwafemi Akinremi (O)
Aiysha Ali (A)
Julie Cawthorne (J)
Paul Cleary (P)
Derrick W Crook (DW)
Valerie Decraene (V)
Andrew Dodgson (A)
Michel Doumith (M)
Matthew Ellington (M)
David W Eyre (DW)
Ryan P George (RP)
John Grimshaw (J)
Malcolm Guiver (M)
Robert Hill (R)
Katie Hopkins (K)
Rachel Jones (R)
Cheryl Lenney (C)
Amy J Mathers (AJ)
Ashley McEwan (A)
Ginny Moore (G)
Mark Neilson (M)
Sarah Neilson (S)
Tim E A Peto (TEA)
Hang T T Phan (HTT)
Mark Regan (M)
Anna C Seale (AC)
Nicole Stoesser (N)
Jay Turner-Gardner (J)
Vicky Watts (V)
Jimmy Walker (J)
A Sarah Walker (A)
David Wyllie (D)
William Welfare (W)
Neil Woodford (N)

Commentaires et corrections

Type : CommentIn

Informations de copyright

Crown Copyright © 2019. Published by Elsevier Ltd. All rights reserved.

Références

Infect Control Hosp Epidemiol. 2009 Jan;30(1):25-33
pubmed: 19046054
J Clin Microbiol. 2012 May;50(5):1558-63
pubmed: 22403422
Emerg Infect Dis. 2012 Aug;18(8):1242-7
pubmed: 22841005
J Hosp Infect. 2012 Sep;82(1):19-24
pubmed: 22841682
Microb Drug Resist. 2013 Apr;19(2):104-9
pubmed: 23067199
Med J Aust. 2013 Mar 18;198(5):267-9
pubmed: 23496403
PLoS One. 2013;8(3):e59015
pubmed: 23536849
Clin Microbiol Infect. 2013 Nov;19(11):E490-8
pubmed: 23829434
Antimicrob Resist Infect Control. 2013 Dec 18;2(1):35
pubmed: 24345195
J Hosp Infect. 2014 Jun;87(2):126-30
pubmed: 24831607
Antimicrob Agents Chemother. 2015 Jan;59(1):714-6
pubmed: 25348541
Int J Antimicrob Agents. 2015 Aug;46(2):169-73
pubmed: 25982912
Euro Surveill. 2016 Apr 28;21(17):
pubmed: 27168586
Clin Infect Dis. 2017 May 15;64(10):1435-1444
pubmed: 28200000
Appl Environ Microbiol. 2017 Mar 31;83(8):
pubmed: 28235877
Antimicrob Resist Infect Control. 2017 Feb 16;6:24
pubmed: 28239453
J Infect Dis. 2017 Feb 15;215(suppl_1):S28-S36
pubmed: 28375512
Antimicrob Resist Infect Control. 2017 Jun 10;6:59
pubmed: 28616203
Pathogens. 2017 Aug 09;6(3):null
pubmed: 28792484
Infect Control Hosp Epidemiol. 2017 Nov;38(11):1329-1334
pubmed: 29061201
J Hosp Infect. 2018 Mar;98(3):275-281
pubmed: 29104124
Clin Infect Dis. 2018 Jul 2;67(2):171-178
pubmed: 29409044
J Hosp Infect. 2018 Nov;100(3):e115-e122
pubmed: 29738784
Antimicrob Agents Chemother. 2018 Nov 26;62(12):
pubmed: 30249685
Appl Environ Microbiol. 2019 Jan 9;85(2):null
pubmed: 30367005
J Clin Microbiol. 1996 Feb;34(2):358-63
pubmed: 8789016
J Hosp Infect. 1996 Aug;33(4):249-62
pubmed: 8864938

Auteurs

P Aranega-Bou (P)

Biosafety, Air and Water Microbiology Group, National Infection Service, Public Health England, Salisbury, UK. Electronic address: Paz.AranegaBou@phe.gov.uk.

R P George (RP)

Manchester University NHS Foundation Trust, Manchester, UK.

N Q Verlander (NQ)

Statistics Unit, Statistics, Modelling and Economics Department, National Infection Service, Public Health England, Colindale, UK.

S Paton (S)

Biosafety, Air and Water Microbiology Group, National Infection Service, Public Health England, Salisbury, UK.

A Bennett (A)

Biosafety, Air and Water Microbiology Group, National Infection Service, Public Health England, Salisbury, UK.

G Moore (G)

Biosafety, Air and Water Microbiology Group, National Infection Service, Public Health England, Salisbury, UK.

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