Increased peripheral levels of TARC/CCL17 in first episode psychosis patients.


Journal

Schizophrenia research
ISSN: 1573-2509
Titre abrégé: Schizophr Res
Pays: Netherlands
ID NLM: 8804207

Informations de publication

Date de publication:
08 2019
Historique:
received: 13 08 2018
revised: 19 12 2018
accepted: 20 12 2018
pubmed: 8 1 2019
medline: 29 9 2020
entrez: 8 1 2019
Statut: ppublish

Résumé

Evidence for a link between the pathophysiology of schizophrenia and the immune system is mounting. Altered levels of chemokines in plasma have previously been reported in patients with schizophrenia under antipsychotic medication. Here we aimed to study both peripheral and central chemokine levels in drug-naïve or short-time medicated first episode psychosis (FEP) patients. We analyzed nine chemokines in plasma and CSF from 41 FEP patients and 22 healthy controls using electrochemiluminescence assay. In plasma four chemokines; TARC/CCL17, eotaxin/CCL11, MDC/CCL22, IP-10/CXCL10 and in CSF one chemokine; IP-10/CXCL10 showed reliable detection in >50% of the cases. FEP patients displayed increased levels of TARC/CCL17 in plasma compared to healthy controls, 89.6 (IQR 66.2-125.8) pg/mL compared to 48.6 (IQR 28.0-71.7) pg/mL (p = 0.001). The difference was not attributed to confounding factors. Plasma TARC/CCL17 was not associated with PANSS, CGI or GAF scores, neither with cognitive functions. The chemokines eotaxin/CCL11, MDC/CCL22, IP-10/CXCL10 in plasma and IP-10/CXCL10 in CSF did not differ between FEP patients and controls. In line with a previous study showing that chronic patients with schizophrenia display increased plasma TARC/CCL17 levels, we here found an elevation in FEP patients suggesting a role of TARC/CCL17 in early stages of schizophrenia. The exact mechanism of this involvement is still unknown and future longitudinal studies as well as studies of central and peripheral chemokine levels would be of great interest.

Sections du résumé

BACKGROUND
Evidence for a link between the pathophysiology of schizophrenia and the immune system is mounting. Altered levels of chemokines in plasma have previously been reported in patients with schizophrenia under antipsychotic medication. Here we aimed to study both peripheral and central chemokine levels in drug-naïve or short-time medicated first episode psychosis (FEP) patients.
METHOD
We analyzed nine chemokines in plasma and CSF from 41 FEP patients and 22 healthy controls using electrochemiluminescence assay.
RESULTS
In plasma four chemokines; TARC/CCL17, eotaxin/CCL11, MDC/CCL22, IP-10/CXCL10 and in CSF one chemokine; IP-10/CXCL10 showed reliable detection in >50% of the cases. FEP patients displayed increased levels of TARC/CCL17 in plasma compared to healthy controls, 89.6 (IQR 66.2-125.8) pg/mL compared to 48.6 (IQR 28.0-71.7) pg/mL (p = 0.001). The difference was not attributed to confounding factors. Plasma TARC/CCL17 was not associated with PANSS, CGI or GAF scores, neither with cognitive functions. The chemokines eotaxin/CCL11, MDC/CCL22, IP-10/CXCL10 in plasma and IP-10/CXCL10 in CSF did not differ between FEP patients and controls.
CONCLUSION
In line with a previous study showing that chronic patients with schizophrenia display increased plasma TARC/CCL17 levels, we here found an elevation in FEP patients suggesting a role of TARC/CCL17 in early stages of schizophrenia. The exact mechanism of this involvement is still unknown and future longitudinal studies as well as studies of central and peripheral chemokine levels would be of great interest.

Identifiants

pubmed: 30612841
pii: S0920-9964(18)30729-1
doi: 10.1016/j.schres.2018.12.033
pii:
doi:

Substances chimiques

CCL11 protein, human 0
CCL17 protein, human 0
CCL22 protein, human 0
CXCL10 protein, human 0
Chemokine CCL11 0
Chemokine CCL17 0
Chemokine CCL22 0
Chemokine CXCL10 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

221-227

Informations de copyright

Copyright © 2018 Elsevier B.V. All rights reserved.

Auteurs

Anna Malmqvist (A)

Department of Physiology and Pharmacology, Karolinska Institutet, Stockholm, Sweden.

Lilly Schwieler (L)

Department of Physiology and Pharmacology, Karolinska Institutet, Stockholm, Sweden.

Funda Orhan (F)

Department of Physiology and Pharmacology, Karolinska Institutet, Stockholm, Sweden.

Helena Fatouros-Bergman (H)

Centre for Psychiatry Research, Department of Clinical Neuroscience, Karolinska Institutet, Stockholm & Stockholm Health Care Services, Stockholm County Council, Sweden.

Markus Bauer (M)

Department of Physiology and Pharmacology, Karolinska Institutet, Stockholm, Sweden.

Lena Flyckt (L)

Centre for Psychiatry Research, Department of Clinical Neuroscience, Karolinska Institutet, Stockholm & Stockholm Health Care Services, Stockholm County Council, Sweden.

Simon Cervenka (S)

Centre for Psychiatry Research, Department of Clinical Neuroscience, Karolinska Institutet, Stockholm & Stockholm Health Care Services, Stockholm County Council, Sweden.

Göran Engberg (G)

Department of Physiology and Pharmacology, Karolinska Institutet, Stockholm, Sweden.

Fredrik Piehl (F)

Neuroimmunology Unit, Department of Clinical Neuroscience, Karolinska Institutet, Stockholm, Sweden.

Sophie Erhardt (S)

Department of Physiology and Pharmacology, Karolinska Institutet, Stockholm, Sweden. Electronic address: sophie.erhardt@ki.se.

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Classifications MeSH