Sortilin levels are associated with peripheral arterial disease in type 2 diabetic subjects.


Journal

Cardiovascular diabetology
ISSN: 1475-2840
Titre abrégé: Cardiovasc Diabetol
Pays: England
ID NLM: 101147637

Informations de publication

Date de publication:
11 01 2019
Historique:
received: 07 11 2018
accepted: 03 01 2019
entrez: 13 1 2019
pubmed: 13 1 2019
medline: 24 4 2019
Statut: epublish

Résumé

Sortilin is a 95-kDa protein which has recently been linked to circulating cholesterol concentration and lifetime risk of developing significant atherosclerotic disease. Sortilin is found inside different cell types and circulating in blood. Higher circulating sortilin concentration has been found in patients with coronary atherosclerosis compared to control subjects. Sortilin concentration is influenced by statin therapy. We enrolled statin-naïve subjects with type 2 diabetes mellitus and we performed a cross-sectional study to evaluate the association between sortilin levels and the presence of clinically significant lower limb peripheral artery disease (PAD) in a population of statin-free diabetic subjects. Out of the 154 patients enrolled in our study, 80 patients were free from PAD, while 74 had clinically significant PAD. Sortilin concentration was significantly higher in the latter group compared to the former (1.61 ± 0.54 ng/mL versus 0.67 ± 0.30 ng/mL, P < 0.01) and there was a trend toward increased sortilin levels as disease severity increased. The association of sortilin levels with PAD remained after adjusting for major risk factors in a multivariate analysis. We showed that sortilin is significantly and independently associated with the presence of lower limb PAD in a statin-free diabetic population and it may be a promising marker for clinically significant atherosclerosis of the lower limbs. Further studies are needed to confirm this finding and to evaluate its clinical usefulness.

Sections du résumé

BACKGROUND
Sortilin is a 95-kDa protein which has recently been linked to circulating cholesterol concentration and lifetime risk of developing significant atherosclerotic disease. Sortilin is found inside different cell types and circulating in blood. Higher circulating sortilin concentration has been found in patients with coronary atherosclerosis compared to control subjects. Sortilin concentration is influenced by statin therapy.
METHODS
We enrolled statin-naïve subjects with type 2 diabetes mellitus and we performed a cross-sectional study to evaluate the association between sortilin levels and the presence of clinically significant lower limb peripheral artery disease (PAD) in a population of statin-free diabetic subjects.
RESULTS
Out of the 154 patients enrolled in our study, 80 patients were free from PAD, while 74 had clinically significant PAD. Sortilin concentration was significantly higher in the latter group compared to the former (1.61 ± 0.54 ng/mL versus 0.67 ± 0.30 ng/mL, P < 0.01) and there was a trend toward increased sortilin levels as disease severity increased. The association of sortilin levels with PAD remained after adjusting for major risk factors in a multivariate analysis.
CONCLUSIONS
We showed that sortilin is significantly and independently associated with the presence of lower limb PAD in a statin-free diabetic population and it may be a promising marker for clinically significant atherosclerosis of the lower limbs. Further studies are needed to confirm this finding and to evaluate its clinical usefulness.

Identifiants

pubmed: 30634965
doi: 10.1186/s12933-019-0805-5
pii: 10.1186/s12933-019-0805-5
pmc: PMC6329108
doi:

Substances chimiques

Adaptor Proteins, Vesicular Transport 0
Biomarkers 0
sortilin Z020Y8WIJ4

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

5

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Auteurs

Federico Biscetti (F)

U.O.C. Clinica Medica e Malattie Vascolari, Department of Medicine, Fondazione Policlinico Universitario A. Gemelli IRCCS, Largo F. Vito 1, 00168, Rome, Italy. f.biscetti@gmail.com.
Laboratory of Vascular Biology and Genetics, Università Cattolica del Sacro Cuore, Rome, Italy. f.biscetti@gmail.com.

Nicola Bonadia (N)

Laboratory of Vascular Biology and Genetics, Università Cattolica del Sacro Cuore, Rome, Italy.
U.O.C. Medicina d'Urgenza e Pronto Soccorso, Fondazione Policlinico Universitario A. Gemelli IRCCS, Rome, Italy.

Francesco Santini (F)

Laboratory of Vascular Biology and Genetics, Università Cattolica del Sacro Cuore, Rome, Italy.

Flavia Angelini (F)

Laboratory of Vascular Biology and Genetics, Università Cattolica del Sacro Cuore, Rome, Italy.

Elisabetta Nardella (E)

U.O.C. Clinica Medica e Malattie Vascolari, Department of Medicine, Fondazione Policlinico Universitario A. Gemelli IRCCS, Largo F. Vito 1, 00168, Rome, Italy.
Laboratory of Vascular Biology and Genetics, Università Cattolica del Sacro Cuore, Rome, Italy.

Dario Pitocco (D)

U.O.S.A. di Diabetologia, Fondazione Policlinico Universitario A. Gemelli IRCCS, Rome, Italy.
Università Cattolica del Sacro Cuore, Rome, Italy.

Angelo Santoliquido (A)

Università Cattolica del Sacro Cuore, Rome, Italy.
U.O.S. Angiologia Columbus, Fondazione Policlinico Universitario A. Gemelli IRCCS, Rome, Italy.

Marco Filipponi (M)

Ospedale San Giovanni Battista ACISMOM, Rome, Italy.

Raffaele Landolfi (R)

U.O.C. Clinica Medica e Malattie Vascolari, Department of Medicine, Fondazione Policlinico Universitario A. Gemelli IRCCS, Largo F. Vito 1, 00168, Rome, Italy.
Università Cattolica del Sacro Cuore, Rome, Italy.

Andrea Flex (A)

U.O.C. Clinica Medica e Malattie Vascolari, Department of Medicine, Fondazione Policlinico Universitario A. Gemelli IRCCS, Largo F. Vito 1, 00168, Rome, Italy.
Laboratory of Vascular Biology and Genetics, Università Cattolica del Sacro Cuore, Rome, Italy.
Università Cattolica del Sacro Cuore, Rome, Italy.

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