RNA Polymerase II CTD Tyrosine 1 Is Required for Efficient Termination by the Nrd1-Nab3-Sen1 Pathway.


Journal

Molecular cell
ISSN: 1097-4164
Titre abrégé: Mol Cell
Pays: United States
ID NLM: 9802571

Informations de publication

Date de publication:
21 02 2019
Historique:
received: 31 05 2018
revised: 09 09 2018
accepted: 29 11 2018
pubmed: 15 1 2019
medline: 25 6 2019
entrez: 15 1 2019
Statut: ppublish

Résumé

In Saccharomyces cerevisiae, transcription termination at protein-coding genes is coupled to the cleavage of the nascent transcript, whereas most non-coding RNA transcription relies on a cleavage-independent termination pathway involving Nrd1, Nab3, and Sen1 (NNS). Termination involves RNA polymerase II CTD phosphorylation, but a systematic analysis of the contribution of individual residues would improve our understanding of the role of the CTD in this process. Here we investigated the effect of mutating phosphorylation sites in the CTD on termination. We observed widespread termination defects at protein-coding genes in mutants for Ser2 or Thr4 but rare defects in Tyr1 mutants for this genes class. Instead, mutating Tyr1 led to widespread termination defects at non-coding genes terminating via NNS. Finally, we showed that Tyr1 is important for pausing in the 5' end of genes and that slowing down transcription suppresses termination defects. Our work highlights the importance of Tyr1-mediated pausing in NNS-dependent termination.

Identifiants

pubmed: 30639244
pii: S1097-2765(18)31032-3
doi: 10.1016/j.molcel.2018.12.002
pii:
doi:

Substances chimiques

NAB3 protein, S cerevisiae 0
NRD1 protein, S cerevisiae 0
Nuclear Proteins 0
RNA-Binding Proteins 0
Saccharomyces cerevisiae Proteins 0
Tyrosine 42HK56048U
RNA Polymerase II EC 2.7.7.-
SEN1 protein, S cerevisiae EC 3.6.1.-
DNA Helicases EC 3.6.4.-
RNA Helicases EC 3.6.4.13

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

655-669.e7

Subventions

Organisme : CIHR
ID : MOP-133 648
Pays : Canada

Informations de copyright

Copyright © 2018 Elsevier Inc. All rights reserved.

Auteurs

Pierre Collin (P)

Institut de recherches cliniques de Montréal, 110 Avenue des Pins Ouest, Montréal, QC, H2W 1R7, Canada.

Célia Jeronimo (C)

Institut de recherches cliniques de Montréal, 110 Avenue des Pins Ouest, Montréal, QC, H2W 1R7, Canada.

Christian Poitras (C)

Institut de recherches cliniques de Montréal, 110 Avenue des Pins Ouest, Montréal, QC, H2W 1R7, Canada.

François Robert (F)

Institut de recherches cliniques de Montréal, 110 Avenue des Pins Ouest, Montréal, QC, H2W 1R7, Canada; Département de Médecine, Faculté de Médecine, Université de Montréal, 2900 Boulevard Edouard-Montpetit, Montréal, QC H3T 1J4, Canada. Electronic address: francois.robert@ircm.qc.ca.

Articles similaires

Humans Meals Time Factors Female Adult

Vancomycin-associated DRESS demonstrates delay in AST abnormalities.

Ahmed Hussein, Kateri L Schoettinger, Jourdan Hydol-Smith et al.
1.00
Humans Drug Hypersensitivity Syndrome Vancomycin Female Male
Animals TOR Serine-Threonine Kinases Colorectal Neoplasms Colitis Mice
T-Lymphocytes, Regulatory Lung Neoplasms Proto-Oncogene Proteins p21(ras) Animals Humans

Classifications MeSH