The combined use of EFS, GPX2, and SPRR1A expression could distinguish favorable from poor clinical outcome among epithelial-like head and neck carcinoma subtypes.


Journal

Head & neck
ISSN: 1097-0347
Titre abrégé: Head Neck
Pays: United States
ID NLM: 8902541

Informations de publication

Date de publication:
06 2019
Historique:
received: 05 05 2017
revised: 28 11 2018
accepted: 12 12 2018
pubmed: 18 1 2019
medline: 11 11 2020
entrez: 18 1 2019
Statut: ppublish

Résumé

We aimed at identifying molecular markers predictive of clinical outcome in patients with head and neck cancer based on the expression profile of cells showing epithelial-like (EL) or mesenchymal-like (ML) phenotypes. We analyzed the association between EL and ML cells and migration, drug resistance, or tumor growth. The differential gene expression profile between cell types was used to build a model to stratify patients according to survival. EL cells were sensitive to cisplatin and cetuximab, showed low migration, and generated squamous differentiated tumors in mouse. A differential 93-gene expression signature between ML and EL cells was used to build a three-gene (EFS, GPX2, and SPRR1A) survival model by analyzing the RNA-seq data of the TCGA-HNSC project. Its prognostic value was confirmed in two independent cohorts. EFS, GPX2, and SPRR1A are prognostic markers able to distinguish clinical outcome among subtypes sharing an EL phenotype.

Sections du résumé

BACKGROUND
We aimed at identifying molecular markers predictive of clinical outcome in patients with head and neck cancer based on the expression profile of cells showing epithelial-like (EL) or mesenchymal-like (ML) phenotypes.
MATERIALS AND METHODS
We analyzed the association between EL and ML cells and migration, drug resistance, or tumor growth. The differential gene expression profile between cell types was used to build a model to stratify patients according to survival.
RESULTS
EL cells were sensitive to cisplatin and cetuximab, showed low migration, and generated squamous differentiated tumors in mouse. A differential 93-gene expression signature between ML and EL cells was used to build a three-gene (EFS, GPX2, and SPRR1A) survival model by analyzing the RNA-seq data of the TCGA-HNSC project. Its prognostic value was confirmed in two independent cohorts.
CONCLUSION
EFS, GPX2, and SPRR1A are prognostic markers able to distinguish clinical outcome among subtypes sharing an EL phenotype.

Identifiants

pubmed: 30652380
doi: 10.1002/hed.25623
doi:

Substances chimiques

Adaptor Proteins, Signal Transducing 0
Antineoplastic Agents 0
Cornified Envelope Proline-Rich Proteins 0
EFS protein, human 0
RNA, Messenger 0
SPRR1A protein, human 0
GPX2 protein, human EC 1.11.1.-
Glutathione Peroxidase EC 1.11.1.9
Cetuximab PQX0D8J21J
Cisplatin Q20Q21Q62J

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1830-1845

Subventions

Organisme : Instituto de Salud Carlos III
Pays : International
Organisme : CIBER-BBN
Pays : International

Informations de copyright

© 2019 Wiley Periodicals, Inc.

Auteurs

Miguel Angel Pavón (MA)

Infections and Cancer Laboratory/Cancer Epidemiology Research Program. Catalan Institute of Oncology (ICO) and Bellvitge Institute of Biomedical Research (IDIBELL), Barcelona, Spain.
Centro de Investigación Biomédica en Red en Cáncer (CIBER-ONC), Madrid, Spain.

Irene Arroyo-Solera (I)

Oncogenesis and Antitumor Drug Group, lnstitut d'Investigacions Biomèdiques Sant Pau (IIB-Sant Pau), Barcelona, Spain.
Centro de Investigación Biomédica en Red en Bioingeniería, Biomateriales y Nanomecidicina (CIBER-BBN), Madrid, Spain.

Xavier León (X)

Centro de Investigación Biomédica en Red en Bioingeniería, Biomateriales y Nanomecidicina (CIBER-BBN), Madrid, Spain.
Department of Otorrinolaryngology, Hospital de la Santa Creu i Sant Pau, Barcelona, Spain.

Marta Téllez-Gabriel (M)

Laboratorio Hematología Oncológica y de Transplantes, Institut Investigacions Biomèdiques (IBB) Sant Pau, Hospital de la Santa Creu i Sant Pau, Barcelona, Spain.

David Virós (D)

Department of Otorrinolaryngology, Hospital Germans Tries y Pujol (Can Ruti), Barcelona, Spain.

Alberto Gallardo (A)

Department of Pathology, Hospital de la Santa Creu i Sant Pau, Barcelona, Spain.

Maria Virtudes Céspedes (MV)

Oncogenesis and Antitumor Drug Group, lnstitut d'Investigacions Biomèdiques Sant Pau (IIB-Sant Pau), Barcelona, Spain.
Centro de Investigación Biomédica en Red en Bioingeniería, Biomateriales y Nanomecidicina (CIBER-BBN), Madrid, Spain.

Isolda Casanova (I)

Oncogenesis and Antitumor Drug Group, lnstitut d'Investigacions Biomèdiques Sant Pau (IIB-Sant Pau), Barcelona, Spain.
Centro de Investigación Biomédica en Red en Bioingeniería, Biomateriales y Nanomecidicina (CIBER-BBN), Madrid, Spain.

Antonio Lopez-Pousa (A)

Centro de Investigación Biomédica en Red en Bioingeniería, Biomateriales y Nanomecidicina (CIBER-BBN), Madrid, Spain.
Department of Medical Oncology, Hospital de la Santa Creu i Sant Pau, Barcelona, Spain.

Agustí Barnadas (A)

Department of Medical Oncology, Hospital de la Santa Creu i Sant Pau, Barcelona, Spain.

Miquel Quer (M)

Department of Otorrinolaryngology, Hospital de la Santa Creu i Sant Pau, Barcelona, Spain.

Ramón Mangues (R)

Oncogenesis and Antitumor Drug Group, lnstitut d'Investigacions Biomèdiques Sant Pau (IIB-Sant Pau), Barcelona, Spain.
Centro de Investigación Biomédica en Red en Bioingeniería, Biomateriales y Nanomecidicina (CIBER-BBN), Madrid, Spain.

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Classifications MeSH