Prospective study evaluating immune-mediated mechanisms and predisposing factors underlying persistent postinfectious abdominal complaints.


Journal

Neurogastroenterology and motility
ISSN: 1365-2982
Titre abrégé: Neurogastroenterol Motil
Pays: England
ID NLM: 9432572

Informations de publication

Date de publication:
04 2019
Historique:
received: 19 09 2018
revised: 20 11 2018
accepted: 10 12 2018
pubmed: 19 1 2019
medline: 6 2 2020
entrez: 19 1 2019
Statut: ppublish

Résumé

The role of persistent immune activation in postinfectious irritable bowel syndrome (PI-IBS) remains controversial. Here, we prospectively studied healthy subjects traveling to destinations with a high-risk to develop infectious gastroenteritis (IGE) in order to identify immune-mediated mechanisms and risk factors of PI-IBS. One hundred and one travelers were asked to complete questionnaires on psychological profile and gastrointestinal (GI) symptoms before travel, 2 weeks, 6 months and 1 year after travel. At each visit, blood was collected for PBMC isolation and rectal biopsies were taken. PI-IBS was diagnosed using the Rome III criteria and subjects with persistent postinfectious abdominal complaints (PI-AC) were identified using 3 GSRS symptoms (ie, loose stools, urgency and abdominal pain). Forty-seven of the 101 subjects reported IGE during travel. After 1 year, two subjects were diagnosed with PI-IBS and eight subjects were presented with PI-AC versus two subjects with IBS and two with abdominal complaints in the non-infected group. PBMC analysis showed no differences in T and B cell populations in subjects with PI-AC vs healthy. Additionally, no differences in gene expression were observed in the early postinfectious phase or after 1 year. Regression analysis identified looser stools, higher anxiety and somatization before infection and several postinfectious GI symptoms as risk factors for PI-AC. The incidence of PI-IBS is low following travelers' diarrhea and there is need for larger studies investigating the role of immune activation in PI-IBS. Psychological factors before infection and the severity of symptoms shortly after infection are risk factors for the persistence of PI-AC.

Sections du résumé

BACKGROUND
The role of persistent immune activation in postinfectious irritable bowel syndrome (PI-IBS) remains controversial. Here, we prospectively studied healthy subjects traveling to destinations with a high-risk to develop infectious gastroenteritis (IGE) in order to identify immune-mediated mechanisms and risk factors of PI-IBS.
METHODS
One hundred and one travelers were asked to complete questionnaires on psychological profile and gastrointestinal (GI) symptoms before travel, 2 weeks, 6 months and 1 year after travel. At each visit, blood was collected for PBMC isolation and rectal biopsies were taken. PI-IBS was diagnosed using the Rome III criteria and subjects with persistent postinfectious abdominal complaints (PI-AC) were identified using 3 GSRS symptoms (ie, loose stools, urgency and abdominal pain).
RESULTS
Forty-seven of the 101 subjects reported IGE during travel. After 1 year, two subjects were diagnosed with PI-IBS and eight subjects were presented with PI-AC versus two subjects with IBS and two with abdominal complaints in the non-infected group. PBMC analysis showed no differences in T and B cell populations in subjects with PI-AC vs healthy. Additionally, no differences in gene expression were observed in the early postinfectious phase or after 1 year. Regression analysis identified looser stools, higher anxiety and somatization before infection and several postinfectious GI symptoms as risk factors for PI-AC.
CONCLUSIONS
The incidence of PI-IBS is low following travelers' diarrhea and there is need for larger studies investigating the role of immune activation in PI-IBS. Psychological factors before infection and the severity of symptoms shortly after infection are risk factors for the persistence of PI-AC.

Identifiants

pubmed: 30657233
doi: 10.1111/nmo.13542
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e13542

Subventions

Organisme : Global Opportunities for Associations
ID : GOA 14.011
Pays : International
Organisme : FWO
ID : 1110019 N
Pays : International
Organisme : FWO
ID : 1248513 N
Pays : International
Organisme : Bijzonder Onderzoeksfonds
Pays : International

Informations de copyright

© 2019 John Wiley & Sons Ltd.

Auteurs

Morgane V Florens (MV)

Department of Chronic Diseases, Metabolism and Ageing, KU Leuven, Leuven, Belgium.

Sander Van Wanrooy (S)

Department of Chronic Diseases, Metabolism and Ageing, KU Leuven, Leuven, Belgium.

James Dooley (J)

Department of Microbiology and Immunology, KU Leuven, Leuven, Belgium.
Autoimmune Genetics Laboratory, VIB, Leuven, Belgium.

Javier Aguilera-Lizarraga (J)

Department of Chronic Diseases, Metabolism and Ageing, KU Leuven, Leuven, Belgium.

Winde Vanbrabant (W)

Department of Chronic Diseases, Metabolism and Ageing, KU Leuven, Leuven, Belgium.

Mira M Wouters (MM)

Department of Chronic Diseases, Metabolism and Ageing, KU Leuven, Leuven, Belgium.

Lukas Van Oudenhove (L)

Department of Chronic Diseases, Metabolism and Ageing, KU Leuven, Leuven, Belgium.

Willy E Peetermans (WE)

Department of Microbiology and Immunology, KU Leuven, Leuven, Belgium.
Laboratory for Clinical Infectious and Inflammatory Disorders, KU Leuven, Leuven, Belgium.

Adrian Liston (A)

Department of Microbiology and Immunology, KU Leuven, Leuven, Belgium.
Autoimmune Genetics Laboratory, VIB, Leuven, Belgium.

Guy E Boeckxstaens (GE)

Department of Chronic Diseases, Metabolism and Ageing, KU Leuven, Leuven, Belgium.

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Classifications MeSH